Induction of Bim and Bid gene expression during accelerated apoptosis in severe sepsis. - Wikidata - wikimedia - TriplyDB

Induction of Bim and Bid gene expression during accelerated apoptosis in severe sepsis.

Induction of Bim and Bid gene expression during accelerated apoptosis in severe sepsis.

http://www.wikidata.org/entity/Q36993180

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The significance and regulatory mechanisms of innate immune cells in the development of sepsis BH3-only proteins: a 20-year stock-take Chemical chaperone TUDCA prevents apoptosis and improves survival during polymicrobial sepsis in mice Sepsis-induced immunosuppression: from cellular dysfunctions to immunotherapy.Deficiency of Bid protein reduces sepsis-induced apoptosis and inflammation, while improving septic survival.Upregulation of the pro-apoptotic genes BID and FAS in septic shock patients Surviving the first hours in sepsis: getting the basics right (an intensivist's perspective).Impact of sepsis on CD4 T cell immunity Cecal ligation and puncture.Insights into the apoptotic death of immune cells in sepsis.Full activation of CD4+ T cells early during sepsis requires specific antigen.Prevention of lymphocyte apoptosis in septic mice with cancer increases mortality.BH3-only proteins contribute to steatotic liver ischemia-reperfusion injury.Post-operative infection and sepsis in humans is associated with deficient gene expression of γc cytokines and their apoptosis mediators Impact of Conditional miRNA126 Overexpression on Apoptosis-Resistant Endothelial Cell Production.Dynamic expression of leukocyte innate immune genes in whole blood from horses with lipopolysaccharide-induced acute systemic inflammation.Uncontrolled sepsis: a systematic review of translational immunology studies in intensive care medicine.Evaluation of mRNA Biomarkers to Identify Risk of Hospital Acquired Infections in Children Admitted to Paediatric Intensive Care Unit.Molecular mechanisms underlying delayed apoptosis in neutrophils from multiple trauma patients with and without sepsis Effect of Regulatory T Cells on Promoting Apoptosis of T Lymphocyte and Its Regulatory Mechanism in Sepsis.A prospective analysis of lymphocyte phenotype and function over the course of acute sepsis.Glucose starvation induces cell death in K-ras-transformed cells by interfering with the hexosamine biosynthesis pathway and activating the unfolded protein response.Mechanical ventilation during experimental sepsis increases deposition of advanced glycation end products and myocardial inflammation Lymphocytes, apoptosis and sepsis: making the jump from mice to humans.Year in review 2008: Critical Care--sepsis Clinical and Experimental Sepsis Impairs CD8 T-Cell-Mediated Immunity.ICU-acquired immunosuppression and the risk for secondary fungal infections.Immunotherapy: A promising approach to reverse sepsis-induced immunosuppression.Apoptosis-induced lymphopenia in sepsis and other severe injuries.Overexpression of HO-1 Contributes to Sepsis-Induced Immunosuppression by Modulating the Th1/Th2 Balance and Regulatory T-Cell Function.Different changes in mitochondrial apoptotic pathway in lymphocytes and granulocytes in cirrhotic patients with sepsis.Skeletal Muscle and Lymphocyte Mitochondrial Dysfunctions in Septic Shock Trigger ICU-Acquired Weakness and Sepsis-Induced Immunoparalysis.Sustained high serum caspase-3 concentrations and mortality in septic patients.Evaluation of BCL2 and TNFα as mRNA biomarkers for monitoring the immune response in critically ill children

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Induction of Bim and Bid gene expression during accelerated apoptosis in severe sepsis.

http://www.wikidata.org/entity/Q36993180

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Induction of Bim and Bid gene expression during accelerated apoptosis in severe sepsis.

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type

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Induction of Bim and Bid gene expression during accelerated apoptosis in severe sepsis.

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Induction of Bim and Bid gene expression during accelerated apoptosis in severe sepsis.

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Induction of Bim and Bid gene expression during accelerated apoptosis in severe sepsis.

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Andreas Hoeft

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Frank Stüber

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Jens-Christian Schewe

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Lutz E Lehmann

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Malte Book

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Stefan Müller

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Stefan U Weber

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Sven Klaschik

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Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine

Apoptosis initiated when BH3 ligands engage multiple Bcl-2 homologs, not Bax or Bak

Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins.

Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity

The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex

CD95's deadly mission in the immune system

Combined loss of proapoptotic genes Bak or Bax with Bim synergizes to cause defects in hematopoiesis and in thymocyte apoptosis

Caspases: enemies within

The biochemistry of apoptosis

The Bcl2 family: regulators of the cellular life-or-death switch

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5

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