about
High-dose irradiation induces cell cycle arrest, apoptosis, and developmental defects during Drosophila oogenesisMultiple-pathway analysis of double-strand break repair mutations in DrosophilaMechanisms of recombination between diverged sequences in wild-type and BLM-deficient mouse and human cells.Impaired tissue growth is mediated by checkpoint kinase 1 (CHK1) in the integrated stress response.Effect of Low Doses (5-40 cGy) of Gamma-irradiation on Lifespan and Stress-related Genes Expression Profile in Drosophila melanogaster.Reducing DNA polymerase alpha in the absence of Drosophila ATR leads to P53-dependent apoptosis and developmental defectsMultiple functions of Drosophila BLM helicase in maintenance of genome stability.Direct Binding to Replication Protein A (RPA)-coated Single-stranded DNA Allows Recruitment of the ATR Activator TopBP1 to Sites of DNA Damage.A genetic screen for DNA double-strand break repair mutations in DrosophilaAnnealing of Complementary DNA Sequences During Double-Strand Break Repair in Drosophila Is Mediated by the Ortholog of SMARCAL1.Genomic analysis of adaptive differentiation in Drosophila melanogasterDrosophila PCH2 is required for a pachytene checkpoint that monitors double-strand-break-independent events leading to meiotic crossover formation.Stwl modifies chromatin compaction and is required to maintain DNA integrity in the presence of perturbed DNA replication.HorkaD, a chromosome instability-causing mutation in Drosophila, is a dominant-negative allele of Lodestar.DNA damage responses in Drosophila nbs mutants with reduced or altered NBS function.Double-strand break repair assays determine pathway choice and structure of gene conversion events in Drosophila melanogaster.DNA Repair in Drosophila: Mutagens, Models, and Missing Genes.Somatic cell mutations caused by 365 nm LED-UVA due to DNA double-strand breaks through oxidative damage.A Role for the Twins Protein Phosphatase (PP2A-B55) in the Maintenance of Drosophila Genome Integrity.Loss of Drosophila Mei-41/ATR Alters Meiotic Crossover Patterning.The initiator caspase Dronc plays a non-apoptotic role in promoting DNA damage signalling in D. melanogaster.Overexpression of the ATR homologous checkpoint kinase Mei-41 induces a G2/M checkpoint in imaginal tissue
P2860
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P2860
description
2006 nî lūn-bûn
@nan
2006年の論文
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2006年論文
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2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
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2006年论文
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2006年论文
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2006年论文
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name
Drosophila ATR in double-strand break repair.
@ast
Drosophila ATR in double-strand break repair.
@en
type
label
Drosophila ATR in double-strand break repair.
@ast
Drosophila ATR in double-strand break repair.
@en
prefLabel
Drosophila ATR in double-strand break repair.
@ast
Drosophila ATR in double-strand break repair.
@en
P2093
P2860
P1433
P1476
Drosophila ATR in double-strand break repair.
@en
P2093
Burnley Jaklevic
Jeannine R LaRocque
Jeff Sekelsky
Tin Tin Su
P2860
P304
P356
10.1534/GENETICS.106.067330
P407
P577
2006-12-28T00:00:00Z