Efficiency of human immunodeficiency virus type 1 postentry infection processes: evidence against disproportionate numbers of defective virions.
about
Extensive complement-dependent enhancement of HIV-1 by autologous non-neutralising antibodies at early stages of infectionHIV-1 capsid: the multifaceted key player in HIV-1 infectionCharacterization of the early events in dengue virus cell entry by biochemical assays and single-virus tracking.Optimal cytoplasmic transport in viral infectionsHIV-1 pre-integration complexes selectively target decondensed chromatin in the nuclear periphery.In vitro priming recapitulates in vivo HIV-1 specific T cell responses, revealing rapid loss of virus reactive CD4 T cells in acute HIV-1 infection.Rapid dissociation of HIV-1 from cultured cells severely limits infectivity assays, causes the inactivation ascribed to entry inhibitors, and masks the inherently high level of infectivity of virions.Lens epithelium-derived growth factor fusion proteins redirect HIV-1 DNA integration.The amyloidogenic SEVI precursor, PAP248-286, is highly unfolded in solution despite an underlying helical tendency.Innate sensing of HIV-infected cells.APOBEC3A is a specific inhibitor of the early phases of HIV-1 infection in myeloid cells.Optimal antiviral switching to minimize resistance risk in HIV therapy.The design and validation of a novel phenotypic assay to determine HIV-1 coreceptor usage of clinical isolates.Generation of representative primary virus isolates from blood plasma after isolation of HIV-1 with CD44 MicroBeads.Revisiting HIV-1 uncoating.Quantification of reverse transcriptase activity by real-time PCR as a fast and accurate method for titration of HIV, lenti- and retroviral vectors.Optimized Infectivity of the Cell-Free Single-Cycle Human Immunodeficiency Viruses Type 1 (HIV-1) and Its Restriction by Host Cells.Retroviral integration site selection.Human Cytomegalovirus gH/gL/gO Promotes the Fusion Step of Entry into All Cell Types, whereas gH/gL/UL128-131 Broadens Virus Tropism through a Distinct Mechanism.Epitope targeting and viral inoculum are determinants of Nef-mediated immune evasion of HIV-1 from cytotoxic T lymphocytesMutational analysis of the C-terminal gag cleavage sites in human immunodeficiency virus type 1A Novel Entry/Uncoating Assay Reveals the Presence of at Least Two Species of Viral Capsids During Synchronized HIV-1 Infection.Pol-specific CD8+ T cells recognize simian immunodeficiency virus-infected cells prior to Nef-mediated major histocompatibility complex class I downregulation.Cell factors stimulate human immunodeficiency virus type 1 reverse transcription in vitroMolecular determinants of the ratio of inert to infectious virus particles.TNPO3 protects HIV-1 replication from CPSF6-mediated capsid stabilization in the host cell cytoplasm.Bacterial curli protein promotes the conversion of PAP248-286 into the amyloid SEVI: cross-seeding of dissimilar amyloid sequencesMutations in human immunodeficiency virus type 1 nucleocapsid protein zinc fingers cause premature reverse transcriptionNucleocapsid protein function in early infection processes.Characterization of the behavior of functional viral genomes during the early steps of human immunodeficiency virus type 1 infection.High efficiency of HIV-1 genomic RNA packaging and heterozygote formation revealed by single virion analysis.Helical conformation of the SEVI precursor peptide PAP248-286, a dramatic enhancer of HIV infectivity, promotes lipid aggregation and fusion.Synchronous infection of SIV and HIV in vitro for virology, immunology and vaccine-related studies.Capacity for infectious HIV-1 virion capture differs by envelope antibody specificity.Viral and cellular requirements for the nuclear entry of retroviral preintegration nucleoprotein complexes.Quantitative microscopy of functional HIV post-entry complexes reveals association of replication with the viral capsidQuantification system for the viral dynamics of a highly pathogenic simian/human immunodeficiency virus based on an in vitro experiment and a mathematical modelVirion-associated Vpr of human immunodeficiency virus type 1 triggers activation of apoptotic events and enhances fas-induced apoptosis in human T cells.Cell-free HTLV-1 infects dendritic cells leading to transmission and transformation of CD4(+) T cells.HIV-1 envelope glycoprotein stimulates viral transcription and increases the infectivity of the progeny virus through the manipulation of cellular machinery.
P2860
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P2860
Efficiency of human immunodeficiency virus type 1 postentry infection processes: evidence against disproportionate numbers of defective virions.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Efficiency of human immunodefi ...... numbers of defective virions.
@ast
Efficiency of human immunodefi ...... numbers of defective virions.
@en
type
label
Efficiency of human immunodefi ...... numbers of defective virions.
@ast
Efficiency of human immunodefi ...... numbers of defective virions.
@en
prefLabel
Efficiency of human immunodefi ...... numbers of defective virions.
@ast
Efficiency of human immunodefi ...... numbers of defective virions.
@en
P2860
P356
P1433
P1476
Efficiency of human immunodefi ...... numbers of defective virions.
@en
P2093
David E Ott
James A Thomas
P2860
P304
P356
10.1128/JVI.02357-06
P407
P577
2007-01-31T00:00:00Z