Herpes simplex virus type 2 virion host shutoff protein regulates alpha/beta interferon but not adaptive immune responses during primary infection in vivo.
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mRNA decay during herpes simplex virus (HSV) infections: protein-protein interactions involving the HSV virion host shutoff protein and translation factors eIF4H and eIF4A.Shutoff of Host Gene Expression in Influenza A Virus and Herpesviruses: Similar Mechanisms and Common ThemesEncephalomyocarditis virus induces PKR-independent mitogen-activated protein kinase activation in macrophages.Induction of interferon-λ contributes to TLR3 and RIG-I activation-mediated inhibition of herpes simplex virus type 2 replication in human cervical epithelial cells.Herpes simplex virus infection stabilizes cellular IEX-1 mRNA.RNA-dependent protein kinase is required for alpha-1 interferon transgene-induced resistance to genital herpes simplex virus type 2.Global mRNA degradation during lytic gammaherpesvirus infection contributes to establishment of viral latency.The virion host shutoff endonuclease (UL41) of herpes simplex virus interacts with the cellular cap-binding complex eIF4F.Uterine epithelial cells specifically induce interferon-stimulated genes in response to polyinosinic-polycytidylic acid independently of estradiol.Inhibition of γ-secretase cleavage in the notch signaling pathway blocks HSV-2-induced type I and type II interferon production.Immobilization of pseudorabies virus in porcine tracheal respiratory mucus revealed by single particle tracking.Herpes simplex virus serotype and entry receptor availability alter CNS disease in a mouse model of neonatal HSVUs3 kinase encoded by herpes simplex virus 1 mediates downregulation of cell surface major histocompatibility complex class I and evasion of CD8+ T cellsActivation and evasion of innate antiviral immunity by herpes simplex virusInnate and adaptive immune responses to herpes simplex virusThe virion host shutoff protein of herpes simplex virus 1 blocks the replication-independent activation of NF-κB in dendritic cells in the absence of type I interferon signaling.Herpes simplex virus virion host shutoff protein: immune evasion mediated by a viral RNase?Susceptibility of CCR5-deficient mice to genital herpes simplex virus type 2 is linked to NK cell mobilizationHerpes simplex virus type 2-mediated disease is reduced in mice lacking RNase LThe amino terminus of the vaccinia virus E3 protein is necessary to inhibit the interferon responseHSV-2 inhibits type-I interferon signaling via multiple complementary and compensatory STAT2-associated mechanismsHSV ICP0 recruits USP7 to modulate TLR-mediated innate responseResponse of aged mice to primary virus infections.Response of primary human airway epithelial cells to influenza infection: a quantitative proteomic studyTo replicate or not to replicate: achieving selective oncolytic virus replication in cancer cells through translational control.Herpes simplex virus 1 UL41 protein abrogates the antiviral activity of hZAP by degrading its mRNASelective ablation of virion host shutoff protein RNase activity attenuates herpes simplex virus 2 in mice.mRNA decay during herpes simplex virus (HSV) infections: mutations that affect translation of an mRNA influence the sites at which it is cleaved by the HSV virion host shutoff (Vhs) protein.Disruption of the U(L)41 gene in the herpes simplex virus 2 dl5-29 mutant increases its immunogenicity and protective capacity in a murine model of genital herpes.The virion host shutoff RNase plays a key role in blocking the activation of protein kinase R in cells infected with herpes simplex virus 1.Small interfering RNAs that deplete the cellular translation factor eIF4H impede mRNA degradation by the virion host shutoff protein of herpes simplex virusHSV-1 strain McKrae is more neuroinvasive than HSV-1 KOS after corneal or vaginal inoculation in miceUse of transcriptional profiling to delineate the initial response of mice to intravaginal herpes simplex virus type 2 infection.Antiviral activity of trappin-2 and elafin in vitro and in vivo against genital herpes.Host responses to wild-type and attenuated herpes simplex virus infection in the absence of Stat1Type I IFN response to Papiine herpesvirus 2 (Herpesvirus papio 2; HVP2) determines neuropathogenicity in mice.Implication of human herpesviruses in oncogenesis through immune evasion and supression.The D10 decapping enzyme of vaccinia virus contributes to decay of cellular and viral mRNAs and to virulence in miceRNA degradation induced by the herpes simplex virus vhs protein proceeds 5' to 3' in vitro.Recognition of herpes simplex viruses: toll-like receptors and beyond
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P2860
Herpes simplex virus type 2 virion host shutoff protein regulates alpha/beta interferon but not adaptive immune responses during primary infection in vivo.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Herpes simplex virus type 2 vi ...... ing primary infection in vivo.
@ast
Herpes simplex virus type 2 vi ...... ing primary infection in vivo.
@en
type
label
Herpes simplex virus type 2 vi ...... ing primary infection in vivo.
@ast
Herpes simplex virus type 2 vi ...... ing primary infection in vivo.
@en
prefLabel
Herpes simplex virus type 2 vi ...... ing primary infection in vivo.
@ast
Herpes simplex virus type 2 vi ...... ing primary infection in vivo.
@en
P2093
P2860
P1433
P1476
Herpes simplex virus type 2 vi ...... ing primary infection in vivo.
@en
P2093
Jenny A Murphy
Lynda A Morrison
Rebecca J Duerst
Tracy J Smith
P2860
P304
P356
10.1128/JVI.77.17.9337-9345.2003
P407
P577
2003-09-01T00:00:00Z