A subpopulation of normal B cells latently infected with Epstein-Barr virus resembles Burkitt lymphoma cells in expressing EBNA-1 but not EBNA-2 or LMP1.
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Epstein-Barr virus LMP2A-induced B-cell survival in two unique classes of EmuLMP2A transgenic miceExpression of Epstein-Barr virus nuclear antigen-1 induces B cell neoplasia in transgenic miceGamma-herpesvirus latency requires T cell evasion during episome maintenanceThe LMP2A ITAM is essential for providing B cells with development and survival signals in vivoMechanisms of T-cell activation by human T-cell lymphotropic virus type I.Mechanisms that regulate Epstein-Barr virus EBNA-1 gene transcription during restricted latency are conserved among lymphocryptoviruses of Old World primates.CTCF prevents the epigenetic drift of EBV latency promoter QpBiology and disease associations of Epstein-Barr virus.Latent membrane protein 2A-mediated effects on the phosphatidylinositol 3-Kinase/Akt pathway.Interferon regulatory factor 7 is induced by Epstein-Barr virus latent membrane protein 1Functional analysis of the CD4(+) T-cell response to Epstein-Barr virus: T-cell-mediated activation of resting B cells and induction of viral BZLF1 expression.Interferon regulatory factor 7 mediates activation of Tap-2 by Epstein-Barr virus latent membrane protein 1.Interferon regulatory factor 2 represses the Epstein-Barr virus BamHI Q latency promoter in type III latency.Molecular virology of Epstein-Barr virus.Use of quantitative competitive PCR to measure Epstein-Barr virus genome load in the peripheral blood of pediatric transplant patients with lymphoproliferative disorders.Virus and cell RNAs expressed during Epstein-Barr virus replicationEpstein-Barr virus LMP2A interferes with global transcription factor regulation when expressed during B-lymphocyte development.EBV in Hodgkin Lymphoma.Burkitt's lymphoma: the Rosetta Stone deciphering Epstein-Barr virus biology.Susceptibility of human placenta derived mesenchymal stromal/stem cells to human herpesviruses infection.Demonstration of the Burkitt's lymphoma Epstein-Barr virus phenotype in dividing latently infected memory cells in vivo.Reprogramming of EBV-immortalized B-lymphocyte cell lines into induced pluripotent stem cells.Tonsillar memory B cells, latently infected with Epstein-Barr virus, express the restricted pattern of latent genes previously found only in Epstein-Barr virus-associated tumors.Isotypes of Epstein-Barr virus antibodies in rheumatoid arthritis: association with rheumatoid factors and citrulline-dependent antibodiesThe Epstein-Barr virus latency BamHI-Q promoter is positively regulated by STATs and Zta interference with JAK/STAT activation leads to loss of BamHI-Q promoter activityEpstein-barr virus latent membrane protein 2B (LMP2B) modulates LMP2A activity.Exploiting the interplay between innate and adaptive immunity to improve immunotherapeutic strategies for Epstein-Barr-virus-driven disordersEpstein-barr virus nuclear antigen 1: from immunologically invisible to a promising T cell target.Detection of heterogeneous Epstein-Barr virus gene expression patterns within individual post-transplantation lymphoproliferative disorders.Transcription start sites downstream of the Epstein-Barr virus (EBV) Fp promoter in early-passage Burkitt lymphoma cells define a fourth promoter for expression of the EBV EBNA-1 proteinThe Epstein-Barr virus EBNA-1 promoter Qp requires an initiator-like element.Interferon-independent and -induced regulation of Epstein-Barr virus EBNA-1 gene transcription in Burkitt lymphoma.Chronic herpesvirus reactivation occurs in aging.Epstein-Barr virus latent membrane protein 2A is a B-cell receptor mimic and essential for B-cell survivalEBV-encoded latent membrane protein 1 cooperates with BAFF/BLyS and APRIL to induce T cell-independent Ig heavy chain class switchingEndogenous presentation of CD8+ T cell epitopes from Epstein-Barr virus-encoded nuclear antigen 1.Epstein-Barr virus latent membrane protein 2A preferentially signals through the Src family kinase Lync-myc activation renders proliferation of Epstein-Barr virus (EBV)-transformed cells independent of EBV nuclear antigen 2 and latent membrane protein 1.Characterization of the CBF2 binding site within the Epstein-Barr virus latency C promoter and its role in modulating EBNA2-mediated transactivation.EBV Persistence--Introducing the Virus.
P2860
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P2860
A subpopulation of normal B cells latently infected with Epstein-Barr virus resembles Burkitt lymphoma cells in expressing EBNA-1 but not EBNA-2 or LMP1.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
A subpopulation of normal B ce ...... EBNA-1 but not EBNA-2 or LMP1.
@ast
A subpopulation of normal B ce ...... EBNA-1 but not EBNA-2 or LMP1.
@en
type
label
A subpopulation of normal B ce ...... EBNA-1 but not EBNA-2 or LMP1.
@ast
A subpopulation of normal B ce ...... EBNA-1 but not EBNA-2 or LMP1.
@en
prefLabel
A subpopulation of normal B ce ...... EBNA-1 but not EBNA-2 or LMP1.
@ast
A subpopulation of normal B ce ...... EBNA-1 but not EBNA-2 or LMP1.
@en
P2093
P2860
P1433
P1476
A subpopulation of normal B ce ...... EBNA-1 but not EBNA-2 or LMP1
@en
P2093
L di Renzo
P2860
P304
P407
P577
1995-06-01T00:00:00Z