Mutually exclusive use of viral promoters in Epstein-Barr virus latently infected lymphocytes
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Identification of alternative transcripts encoding the essential murine gammaherpesvirus lytic transactivator RTAIntron retention may regulate expression of Epstein-Barr virus nuclear antigen 3 family genesEpstein-Barr virus involvement in T-cell malignancy: significance in adult T-cell leukemia.Murine gammaherpesvirus 68 encodes a functional regulator of complement activationCTCF prevents the epigenetic drift of EBV latency promoter QpThe Epstein-Barr virus major latent promoter Qp is constitutively active, hypomethylated, and methylation sensitiveRedefining the Epstein-Barr virus-encoded nuclear antigen EBNA-1 gene promoter and transcription initiation site in group I Burkitt lymphoma cell linesCharacterization of gammaherpesvirus 68 gene 50 transcriptionAlternative splicing dictates translational start in Epstein-Barr virus transcripts.Viral latency and its regulation: lessons from the gamma-herpesviruses.Molecular virology of Epstein-Barr virus.cdc2/cyclin B1-dependent phosphorylation of EBNA2 at Ser243 regulates its function in mitosis.EBNA3A association with RBP-Jkappa down-regulates c-myc and Epstein-Barr virus-transformed lymphoblast growth.The importance of epigenetic alterations in the development of epstein-barr virus-related lymphomas.An ATM/Chk2-mediated DNA damage-responsive signaling pathway suppresses Epstein-Barr virus transformation of primary human B cellsTranscriptional activation by EBV nuclear antigen 1 is essential for the expression of EBV's transforming genes.To be or not IIb: a multi-step process for Epstein-Barr virus latency establishment and consequences for B cell tumorigenesisA subpopulation of normal B cells latently infected with Epstein-Barr virus resembles Burkitt lymphoma cells in expressing EBNA-1 but not EBNA-2 or LMP1.Molecular genetic analysis of Epstein-Barr virus Cp promoter functionTranscription of the Epstein-Barr virus nuclear antigen 1 (EBNA1) gene occurs before induction of the BCR2 (Cp) EBNA gene promoter during the initial stages of infection in B cells.oriP is essential for EBNA gene promoter activity in Epstein-Barr virus-immortalized lymphoblastoid cell lines.Tiled microarray identification of novel viral transcript structures and distinct transcriptional profiles during two modes of productive murine gammaherpesvirus 68 infection.A simple reverse transcriptase PCR assay to distinguish EBNA1 gene transcripts associated with type I and II latency from those arising during induction of the viral lytic cycleLate expression of a beta chemokine homolog by murine cytomegalovirus.Novel downstream elements upregulate transcription initiated from an Epstein-Barr virus latent promoter.Host-cell-determined methylation of specific Epstein-Barr virus promoters regulates the choice between distinct viral latency programsEpstein-Barr virus latency in blood mononuclear cells: analysis of viral gene transcription during primary infection and in the carrier state.DNA-binding-defective mutants of the Epstein-Barr virus lytic switch activator Zta transactivate with altered specificitiesMarked, transient inhibition of expression of the Epstein-Barr virus latent membrane protein gene in Burkitt's lymphoma cell lines by electroporation.Epstein-Barr virus recombinants with specifically mutated BCRF1 genes.Methylation of discrete sites within the enhancer region regulates the activity of the Epstein-Barr virus BamHI W promoter in Burkitt lymphoma lines.Variable expression of latent membrane protein in nasopharyngeal carcinoma can be related to methylation status of the Epstein-Barr virus BNLF-1 5'-flanking region.The Epstein-Barr virus (EBV) nuclear antigen 1 BamHI F promoter is activated on entry of EBV-transformed B cells into the lytic cycle.EBNA-2 transactivates a lymphoid-specific enhancer in the BamHI C promoter of Epstein-Barr virus.Characterization of the Epstein-Barr virus BZLF1 protein transactivation domain.Identification of critical cis elements involved in mediating Epstein-Barr virus nuclear antigen 2-dependent activity of an enhancer located upstream of the viral BamHI C promoter.Epstein-Barr virus latent gene expression in uncultured peripheral blood lymphocytes.The Epstein-Barr virus nuclear protein 1 promoter active in type I latency is autoregulated.Host-cell-phenotype-dependent control of the BCR2/BWR1 promoter complex regulates the expression of Epstein-Barr virus nuclear antigens 2-6Efficient transcription of the Epstein-Barr virus immediate-early BZLF1 and BRLF1 genes requires protein synthesis.
P2860
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P2860
Mutually exclusive use of viral promoters in Epstein-Barr virus latently infected lymphocytes
description
1989 nî lūn-bûn
@nan
1989 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年学术文章
@wuu
1989年学术文章
@zh-cn
1989年学术文章
@zh-hans
1989年学术文章
@zh-my
1989年学术文章
@zh-sg
1989年學術文章
@yue
name
Mutually exclusive use of vira ...... latently infected lymphocytes
@ast
Mutually exclusive use of vira ...... latently infected lymphocytes
@en
Mutually exclusive use of vira ...... latently infected lymphocytes
@nl
type
label
Mutually exclusive use of vira ...... latently infected lymphocytes
@ast
Mutually exclusive use of vira ...... latently infected lymphocytes
@en
Mutually exclusive use of vira ...... latently infected lymphocytes
@nl
prefLabel
Mutually exclusive use of vira ...... latently infected lymphocytes
@ast
Mutually exclusive use of vira ...... latently infected lymphocytes
@en
Mutually exclusive use of vira ...... latently infected lymphocytes
@nl
P2860
P356
P1476
Mutually exclusive use of vira ...... latently infected lymphocytes
@en
P2093
M Woisetschlaeger
P2860
P304
P356
10.1073/PNAS.86.17.6498
P407
P577
1989-09-01T00:00:00Z