Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination - Wikidata - wikimedia - TriplyDB

Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination

Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination

http://www.wikidata.org/entity/Q35840746

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Molecular biology of hepatitis B virus infection DNA Polymerase κ Is a Key Cellular Factor for the Formation of Covalently Closed Circular DNA of Hepatitis B Virus Detection of HBV Covalently Closed Circular DNA Double-stranded linear duck hepatitis B virus (DHBV) stably integrates at a higher frequency than wild-type DHBV in LMH chicken hepatoma cells.Infection of ducklings with virus particles containing linear double-stranded duck hepatitis B virus DNA: illegitimate replication and reversion Integration of hepadnavirus DNA in infected liver: evidence for a linear precursor.Hepatitis B virus biology.Antiviral treatment of chronic hepatitis B virus (HBV) infections Characterization of the host factors required for hepadnavirus covalently closed circular (ccc) DNA formation Origins and Evolution of Hepatitis B Virus and Hepatitis D Virus.Positive selection of yeast nonhomologous end-joining genes and a retrotransposon conflict hypothesis.Animal models and the molecular biology of hepadnavirus infection Hepadnavirus Genome Replication and Persistence.Formation of hepatitis B virus covalently closed circular DNA: removal of genome-linked protein Relationship between viral DNA synthesis and virion envelopment in hepatitis B viruses.Hepatocyte turnover during resolution of a transient hepadnaviral infection.Single-cell analysis of covalently closed circular DNA copy numbers in a hepadnavirus-infected liver The conformation of the 3' end of the minus-strand DNA makes multiple contributions to template switches during plus-strand DNA synthesis of duck hepatitis B virus.The amount of hepatocyte turnover that occurred during resolution of transient hepadnavirus infections was lower when virus replication was inhibited with entecavir.Immune selection during chronic hepadnavirus infection Genomic DNA double-strand breaks are targets for hepadnaviral DNA integration Small DNA hairpin negatively regulates in situ priming during duck hepatitis B virus reverse transcription.Molecular biology of the hepatitis B virus for clinicians Metabolism and function of hepatitis B virus cccDNA: Implications for the development of cccDNA-targeting antiviral therapeutics.HBV DNA Integration: Molecular Mechanisms and Clinical Implications Use of Current and New Endpoints in the Evaluation of Experimental Hepatitis B Therapeutics.A Role for the Host DNA Damage Response in Hepatitis B Virus cccDNA Formation-and Beyond?Integrated hepatitis B virus DNA preserves the binding sequence of transcription factor Yin and Yang 1 at the virus-cell junction.The binding site of transcription factor YY1 is required for intramolecular recombination between terminally repeated sequences of linear replicative hepatitis B virus DNA.Mutations that increase in situ priming also decrease circularization for duck hepatitis B virus.Duck hepatitis B virus integrations in LMH chicken hepatoma cells: identification and characterization of new episomally derived integrations.Loss and acquisition of duck hepatitis B virus integrations in lineages of LMH-D2 chicken hepatoma cells.Covalently closed circular viral DNA formed from two types of linear DNA in woodchuck hepatitis virus-infected liver.Mutations within DR2 independently reduce the amount of both minus- and plus-strand DNA synthesized during duck hepatitis B virus replication.Base pairing between cis-acting sequences contributes to template switching during plus-strand DNA synthesis in human hepatitis B virus.Inhibition of hepatitis B virus DNA replicative intermediate forms by recombinant interferon-gamma.Characterization of nucleosome positioning in hepadnaviral covalently closed circular DNA minichromosomes.Hepatitis B Virus and DNA Damage Response: Interactions and Consequences for the Infection.Liver sampling: a vital window into HBV pathogenesis on the path to functional cure.Host functions used by hepatitis B virus to complete its life cycle: Implications for developing host-targeting agents to treat chronic hepatitis B

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P2860

Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination

http://www.wikidata.org/entity/Q35840746

description

1995 nî lūn-bûn

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1995年の論文

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1995年論文

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1995年論文

@zh-hant

1995年論文

@zh-hk

1995年論文

@zh-mo

1995年論文

@zh-tw

1995年论文

@wuu

1995年论文

@zh

1995年论文

@zh-cn

name

Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination

@ast

Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination

@en

type

label

Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination

@ast

Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination

@en

prefLabel

Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination

@ast

Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination

@en

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Journal of Virology

P1476

Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombination

@en

P2093

J Summers

http://www.w3.org/2001/XMLSchema#string

W Yang

http://www.w3.org/2001/XMLSchema#string

P2860

Mechanism of translation of the hepadnaviral polymerase (P) gene.

Sequence-independent RNA cleavages generate the primers for plus strand DNA synthesis in hepatitis B viruses: implications for other reverse transcribing elements.

Novel forms of woodchuck hepatitis virus DNA isolated from chronically infected woodchuck liver nuclei

pet, a small sequence distal to the pregenome cap site, is required for expression of the duck hepatitis B virus pregenome.

Identification of factor-binding sites in the duck hepatitis B virus enhancer and in vivo effects of enhancer mutations.

Novel mechanism for reverse transcription in hepatitis B viruses

Mutations affecting hepadnavirus plus-strand DNA synthesis dissociate primer cleavage from translocation and reveal the origin of linear viral DNA.

cis-acting sequences required for encapsidation of duck hepatitis B virus pregenomic RNA.

Infection initiated by the RNA pregenome of a DNA virus

Synthesis of hepadnavirus particles that contain replication-defective duck hepatitis B virus genomes in cultured HuH7 cells.

P304

4029-4036

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scholarly article

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P433

7

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P478

69

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1995-07-01T00:00:00Z

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7769660

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189136

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