Sequence-independent RNA cleavages generate the primers for plus strand DNA synthesis in hepatitis B viruses: implications for other reverse transcribing elements.
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Hepatitis B virus replicationEvidence that the RNAseH activity of the duck hepatitis B virus is unable to act on exogenous substratesHepatitis B virus reverse transcriptase: diverse functions as classical and emerging targets for antiviral interventionA bulged region of the hepatitis B virus RNA encapsidation signal contains the replication origin for discontinuous first-strand DNA synthesisHepatitis B virus molecular biology and pathogenesisIncorporation of eukaryotic translation initiation factor eIF4E into viral nucleocapsids via interaction with hepatitis B virus polymerase.Effects of genomic length on translocation of hepatitis B virus polymerase-linked oligomer.Double-stranded linear duck hepatitis B virus (DHBV) stably integrates at a higher frequency than wild-type DHBV in LMH chicken hepatoma cells.Properties of monoclonal antibodies directed against hepatitis B virus polymerase protein.Mapping of the hepatitis B virus reverse transcriptase TP and RT domains by transcomplementation for nucleotide priming and by protein-protein interactionIn vivo effects of mutations in woodchuck hepatitis virus enhancer II.Infection of ducklings with virus particles containing linear double-stranded duck hepatitis B virus DNA: illegitimate replication and reversionIntegration of hepadnavirus DNA in infected liver: evidence for a linear precursor.Identification of an essential molecular contact point on the duck hepatitis B virus reverse transcriptaseA novel cis-acting element facilitates minus-strand DNA synthesis during reverse transcription of the hepatitis B virus genome.Base pairing among three cis-acting sequences contributes to template switching during hepadnavirus reverse transcription.cis-Acting sequences 5E, M, and 3E interact to contribute to primer translocation and circularization during reverse transcription of avian hepadnavirus DNAThree novel cis-acting elements required for efficient plus-strand DNA synthesis of the hepatitis B virus genome.Animal models and the molecular biology of hepadnavirus infectionComplementarity between epsilon and phi sequences in pregenomic RNA influences hepatitis B virus replication efficiency.Hepadnavirus Genome Replication and Persistence.Illegitimate replication of linear hepadnavirus DNA through nonhomologous recombinationNucleotide priming and reverse transcriptase activity of hepatitis B virus polymerase expressed in insect cellsFormation of hepatitis B virus covalently closed circular DNA: removal of genome-linked proteinRelationship between viral DNA synthesis and virion envelopment in hepatitis B viruses.Transcomplementation of nucleotide priming and reverse transcription between independently expressed TP and RT domains of the hepatitis B virus reverse transcriptase.Effects of mutations within and adjacent to the terminal repeats of hepatitis B virus pregenomic RNA on viral DNA synthesisExpression of functional hepatitis B virus polymerase in yeast reveals it to be the sole viral protein required for correct initiation of reverse transcription.When retroviral reverse transcriptases reach the end of their RNA templates.The conformation of the 3' end of the minus-strand DNA makes multiple contributions to template switches during plus-strand DNA synthesis of duck hepatitis B virus.Template switches during plus-strand DNA synthesis of duck hepatitis B virus are influenced by the base composition of the minus-strand terminal redundancy.Genomic DNA double-strand breaks are targets for hepadnaviral DNA integrationResidues Arg703, Asp777, and Arg781 of the RNase H domain of hepatitis B virus polymerase are critical for viral DNA synthesis.Small DNA hairpin negatively regulates in situ priming during duck hepatitis B virus reverse transcription.Molecular biology of the hepatitis B virus for cliniciansChanging the site of initiation of plus-strand DNA synthesis inhibits the subsequent template switch during replication of a hepadnavirus.Mutations that increase in situ priming also decrease circularization for duck hepatitis B virus.Purification and enzymatic characterization of the hepatitis B virus ribonuclease H, a new target for antiviral inhibitors.Analysis of duck hepatitis B virus reverse transcription indicates a common mechanism for the two template switches during plus-strand DNA synthesis.Duck hepatitis B virus integrations in LMH chicken hepatoma cells: identification and characterization of new episomally derived integrations.
P2860
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P2860
Sequence-independent RNA cleavages generate the primers for plus strand DNA synthesis in hepatitis B viruses: implications for other reverse transcribing elements.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Sequence-independent RNA cleav ...... reverse transcribing elements.
@ast
Sequence-independent RNA cleav ...... reverse transcribing elements.
@en
type
label
Sequence-independent RNA cleav ...... reverse transcribing elements.
@ast
Sequence-independent RNA cleav ...... reverse transcribing elements.
@en
prefLabel
Sequence-independent RNA cleav ...... reverse transcribing elements.
@ast
Sequence-independent RNA cleav ...... reverse transcribing elements.
@en
P2860
P1433
P1476
Sequence-independent RNA cleav ...... reverse transcribing elements.
@en
P2093
P2860
P304
P407
P50
P577
1991-11-01T00:00:00Z