Mutations affecting hepadnavirus plus-strand DNA synthesis dissociate primer cleavage from translocation and reveal the origin of linear viral DNA.
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Distinct modes of regulation of transcription of hepatitis B virus by the nuclear receptors HNF4alpha and COUP-TF1Hepatitis B virus replicationEvidence that the RNAseH activity of the duck hepatitis B virus is unable to act on exogenous substratesMechanism of inhibition of duck hepatitis B virus polymerase by (-)-beta-L-2',3'-dideoxy-3'-thiacytidineDNA Polymerase κ Is a Key Cellular Factor for the Formation of Covalently Closed Circular DNA of Hepatitis B VirusHepatitis B virus molecular biology and pathogenesisDetection of HBV Covalently Closed Circular DNAEffects of genomic length on translocation of hepatitis B virus polymerase-linked oligomer.Double-stranded linear duck hepatitis B virus (DHBV) stably integrates at a higher frequency than wild-type DHBV in LMH chicken hepatoma cells.Properties of monoclonal antibodies directed against hepatitis B virus polymerase protein.Mapping of the hepatitis B virus reverse transcriptase TP and RT domains by transcomplementation for nucleotide priming and by protein-protein interactionClonal expansion of hepatocytes during chronic woodchuck hepatitis virus infection.Generation of replication-competent hepatitis B virus nucleocapsids in insect cells.In vivo effects of mutations in woodchuck hepatitis virus enhancer II.Infection of ducklings with virus particles containing linear double-stranded duck hepatitis B virus DNA: illegitimate replication and reversionLow dynamic state of viral competition in a chronic avian hepadnavirus infection.Integration of hepadnavirus DNA in infected liver: evidence for a linear precursor.Serine phosphoacceptor sites within the core protein of hepatitis B virus contribute to genome replication pleiotropically.Critical roles of nuclear receptor response elements in replication of hepatitis B virus.Hepatitis B virus biology.Identification of an essential molecular contact point on the duck hepatitis B virus reverse transcriptaseClonal expansion of normal-appearing human hepatocytes during chronic hepatitis B virus infection.The pre-S domain of the large viral envelope protein determines host range in avian hepatitis B viruses.A novel cis-acting element facilitates minus-strand DNA synthesis during reverse transcription of the hepatitis B virus genome.Antiviral treatment of chronic hepatitis B virus (HBV) infectionsHuman hepatitis B virus production in avian cells is characterized by enhanced RNA splicing and the presence of capsids containing shortened genomesCharacterization of the host factors required for hepadnavirus covalently closed circular (ccc) DNA formationConditional replication of duck hepatitis B virus in hepatoma cells.Targeting Hepatitis B Virus With CRISPR/Cas9.Base pairing among three cis-acting sequences contributes to template switching during hepadnavirus reverse transcription.cis-Acting sequences 5E, M, and 3E interact to contribute to primer translocation and circularization during reverse transcription of avian hepadnavirus DNAThree novel cis-acting elements required for efficient plus-strand DNA synthesis of the hepatitis B virus genome.Generation of duck hepatitis B virus polymerase mutants through site-directed mutagenesis which demonstrate resistance to lamivudine [(--)-beta-L-2', 3'-dideoxy-3'-thiacytidine] in vitroAnimal models and the molecular biology of hepadnavirus infectionResidual integrated viral DNA after hepadnavirus clearance by nucleoside analog therapy.Complementarity between epsilon and phi sequences in pregenomic RNA influences hepatitis B virus replication efficiency.Hepadnavirus Genome Replication and Persistence.Mutations in the epsilon sequences of human hepatitis B virus affect both RNA encapsidation and reverse transcriptionIllegitimate replication of linear hepadnavirus DNA through nonhomologous recombinationNucleotide priming and reverse transcriptase activity of hepatitis B virus polymerase expressed in insect cells
P2860
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P2860
Mutations affecting hepadnavirus plus-strand DNA synthesis dissociate primer cleavage from translocation and reveal the origin of linear viral DNA.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Mutations affecting hepadnavir ...... he origin of linear viral DNA.
@ast
Mutations affecting hepadnavir ...... he origin of linear viral DNA.
@en
type
label
Mutations affecting hepadnavir ...... he origin of linear viral DNA.
@ast
Mutations affecting hepadnavir ...... he origin of linear viral DNA.
@en
prefLabel
Mutations affecting hepadnavir ...... he origin of linear viral DNA.
@ast
Mutations affecting hepadnavir ...... he origin of linear viral DNA.
@en
P2860
P1433
P1476
Mutations affecting hepadnavir ...... he origin of linear viral DNA.
@en
P2093
P2860
P304
P407
P50
P577
1991-03-01T00:00:00Z