about
5-aminoimidazole-4-carboxamide-1-beta-4-ribofuranoside (AICAR) attenuates the expression of LPS- and Abeta peptide-induced inflammatory mediators in astroglia.Neuroprotective effect of novel cognitive enhancer noopept on AD-related cellular model involves the attenuation of apoptosis and tau hyperphosphorylationSpines, plasticity, and cognition in Alzheimer's model miceMechanisms of mononuclear phagocyte recruitment in Alzheimer's diseaseDendritic vulnerability in neurodegenerative disease: insights from analyses of cortical pyramidal neurons in transgenic mouse modelsTransgenic models of Alzheimer's disease: learning from animalsSpine pruning in 5xFAD mice starts on basal dendrites of layer 5 pyramidal neuronsEarly-onset behavioral and synaptic deficits in a mouse model of Alzheimer's disease.Life extension factor klotho prevents mortality and enhances cognition in hAPP transgenic mice.Functional alterations in memory networks in early Alzheimer's disease.Neurodegeneration in glaucoma: progression and calcium-dependent intracellular mechanisms.Multi-target action of the novel anti-Alzheimer compound CHF5074: in vivo study of long term treatment in Tg2576 mice.Synaptotagmins interact with APP and promote Aβ generation.Selective dendritic susceptibility to bioenergetic, excitotoxic and redox perturbations in cortical neurons.Impaired spine stability underlies plaque-related spine loss in an Alzheimer's disease mouse model.Nature, nurture and neurology: gene-environment interactions in neurodegenerative disease. FEBS Anniversary Prize Lecture delivered on 27 June 2004 at the 29th FEBS Congress in Warsaw.Correlation of Alzheimer disease neuropathologic changes with cognitive status: a review of the literatureAmyloid imaging of Alzheimer's disease using Pittsburgh Compound B.Molecular mechanisms mediating pathological plasticity in Huntington's disease and Alzheimer's disease.A Novel Form of Compensation in the Tg2576 Amyloid Mouse Model of Alzheimer's Disease.Smart optical probes for near-infrared fluorescence imaging of Alzheimer's disease pathology.Interaction between amyloid-β pathology and cortical functional columnar organization.Neuronal protein trafficking associated with Alzheimer disease: from APP and BACE1 to glutamate receptorsSignificant structural but not physiological changes in cortical neurons of 12-month-old Tg2576 mice.A single dose of passive immunotherapy has extended benefits on synapses and neurites in an Alzheimer's disease mouse model.Quantitative 3D investigation of Neuronal network in mouse spinal cord model.The upside of APP at synapses.Neuronal hyperactivity--A key defect in Alzheimer's disease?Brain phylogeny, ontogeny and dysfunction: integrating evolutionary, developmental and clinical perspectives in cognitive neuroscience.Calcineurin/NFAT Signaling in Activated Astrocytes Drives Network Hyperexcitability in Aβ-Bearing Mice.Dendritic spine abnormalities in amyloid precursor protein transgenic mice demonstrated by gene transfer and intravital multiphoton microscopy.Non-fibrillar beta-amyloid abates spike-timing-dependent synaptic potentiation at excitatory synapses in layer 2/3 of the neocortex by targeting postsynaptic AMPA receptors.AMP-activated protein kinase: 'a cup of tea' against cholesterol-induced neurotoxicity.Efficacy of cyclin dependent kinase 4 inhibitors as potent neuroprotective agents against insults relevant to Alzheimer's disease.Triflusal reduces dense-core plaque load, associated axonal alterations and inflammatory changes, and rescues cognition in a transgenic mouse model of Alzheimer's disease.Passive immunotherapy rapidly increases structural plasticity in a mouse model of Alzheimer disease.The diphenylpyrazole compound anle138b blocks Aβ channels and rescues disease phenotypes in a mouse model for amyloid pathology.Exenatide upregulates gene expression of glucagon-like peptide-1 receptor and nerve growth factor in streptozotocin/nicotinamide-induced diabetic mice.Effect of active Aβ immunotherapy on neurons in human Alzheimer's disease.Morphological Bases of Neuronal Hyperexcitability in Neurodegeneration.
P2860
Q24815455-B435F9E0-66EB-4EAE-B33C-30CED2446760Q27499804-51CB23F1-78E9-4213-95A8-667015902A87Q27691328-F5FE841D-6B42-4572-95F3-6381837F46E8Q28275105-E26B7C5F-1784-4D71-B908-AB2B26CBAF80Q33533576-60901296-430A-4D44-8439-2AA588371D83Q33844718-2618954F-1FAA-408F-A290-F3F00C1BC5CCQ34147787-B158EDA3-3A82-455D-8B4A-6AD96A7F85ABQ34460984-9DFE2F0F-E268-4B7A-93ED-C4CF5C3ADCEEQ34462326-9A98BC45-DF41-4157-AC91-6EE4B864E620Q34565270-D712D771-949F-41BB-A255-169AE920D0A9Q34579467-1FF28D23-6145-4731-9804-E17B94429E1DQ34656871-9B971A6B-E79D-498A-8CF8-81202863F006Q35879747-AA56DCCE-C817-41E8-B24D-D96FD7105673Q35989360-EC57C3B6-C138-4264-B362-EA9096AD66E9Q36007461-439AF30F-74F9-4731-BEC4-0F34F4DA2663Q36122602-15D47298-6410-43AE-B9AD-5A0B8CD9E794Q36575903-B9A6E8E3-C253-4179-A838-3BC85909B0B3Q36639112-B81BA273-4A32-49C7-8F86-88343EC115E9Q36705304-AB7AAB84-413B-4C40-98A7-7788BD679F1FQ37009427-CA6D62F3-29D2-41F5-A3B2-8EEADB030D4AQ37072475-C1436124-6997-4892-AD74-14987F432E69Q37117343-446513DB-E9AA-4050-9D93-4DD6734EEAE8Q37174236-4BF99D40-C970-4CA0-9269-A4B12F264B37Q37195632-61684FA0-700D-4390-946F-39EF649210C5Q37259695-9A421C30-B59E-42D9-8BB1-CDB3061595AEQ37598458-F9866826-D944-4D37-8A81-DDCC5836ACF9Q37825632-75AE015C-6A5A-4350-9494-F1369B95DC22Q38379006-3E5AFBE2-2BA2-435A-816C-DB68DF13520EQ38764880-E9C31BCD-0D2D-4B3F-9013-9C036694B392Q40179990-9F2E1FD9-137C-4180-89D3-BB724EB62F12Q42087146-9FD5825F-5312-4DD2-A374-636A873E5B19Q42493702-9BD84F4F-C877-4C78-A091-1FD27C02CAA9Q42869845-D49FAF00-1F49-4832-ACA8-3A131DC69CE4Q42912864-98E66977-5E23-47E5-B161-5601BE984D2AQ43098773-7EEC2E22-70EA-4F47-8407-2222F9483799Q43142065-B0589A8E-B621-45EC-9C35-D587ADC37555Q46342780-3E64143F-E346-4218-A1CD-DE1B023D4FA8Q47991877-4CCDF81A-F37B-40A6-94CA-FC811BEFF4A4Q48445250-AB4D9093-E3DE-4704-9EA0-65B28AD88946Q51581195-CCB8AEB8-3A03-4367-8E93-7FE86762ADDB
P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Neuronal structure is altered by amyloid plaques.
@ast
Neuronal structure is altered by amyloid plaques.
@en
type
label
Neuronal structure is altered by amyloid plaques.
@ast
Neuronal structure is altered by amyloid plaques.
@en
prefLabel
Neuronal structure is altered by amyloid plaques.
@ast
Neuronal structure is altered by amyloid plaques.
@en
P2860
P1476
Neuronal structure is altered by amyloid plaques.
@en
P2860
P304
P356
10.1515/REVNEURO.2004.15.4.267
P577
2004-01-01T00:00:00Z