Armed CD4+ Th1 effector cells and activated macrophages participate in bile duct injury in murine biliary atresia. - Wikidata - wikimedia - TriplyDB

Armed CD4+ Th1 effector cells and activated macrophages participate in bile duct injury in murine biliary atresia.

Armed CD4+ Th1 effector cells and activated macrophages participate in bile duct injury in murine biliary atresia.

http://www.wikidata.org/entity/Q35944551

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Pathogenesis of biliary atresia: defining biology to understand clinical phenotypes B cell deficient mice are protected from biliary obstruction in the rotavirus-induced mouse model of biliary atresia.Biliary atresia: recent progress.Interactions between Th1 cells and Tregs affect regulation of hepatic fibrosis in biliary atresia through the IFN-γ/STAT1 pathway.High-dose IgG therapy mitigates bile duct-targeted inflammation and obstruction in a mouse model of biliary atresia Nuclear receptors: mediators and modifiers of inflammation-induced cholestasis.Biliary atresia: will blocking inflammation tame the disease?Staging of biliary atresia at diagnosis by molecular profiling of the liver Generation of a cholangiocyte-specific cDNA expression library for the identification of B and T cell autoantigens in murine biliary disease.Transcriptomic and physiological responses to fishmeal substitution with plant proteins in formulated feed in farmed Atlantic salmon (Salmo salar).RRAS: A key regulator and an important prognostic biomarker in biliary atresia.The pathogenesis of biliary atresia: evidence for a virus-induced autoimmune disease.DNA hypomethylation causes bile duct defects in zebrafish and is a distinguishing feature of infantile biliary atresia.HMGB1-promoted and TLR2/4-dependent NK cell maturation and activation take part in rotavirus-induced murine biliary atresia.Osteopontin upregulation in rotavirus-induced murine biliary atresia requires replicating virus but is not necessary for development of biliary atresia.Natural killer cells promote long-term hepatobiliary inflammation in a low-dose rotavirus model of experimental biliary atresia What Causes Biliary Atresia? Unique Aspects of the Neonatal Immune System Provide Clues to Disease Pathogenesis Dendritic Cells Regulate Treg-Th17 Axis in Obstructive Phase of Bile Duct Injury in Murine Biliary Atresia.Cytomegalovirus-specific T-cell reactivity in biliary atresia at the time of diagnosis is associated with deficits in regulatory T cells.Cellular and humoral autoimmunity directed at bile duct epithelia in murine biliary atresia.Methylation Microarray Studies Highlight PDGFA Expression as a Factor in Biliary Atresia Progressive biliary destruction is independent of a functional tumor necrosis factor-alpha pathway in a rhesus rotavirus-induced murine model of biliary atresia Effector role of neonatal hepatic CD8+ lymphocytes in epithelial injury and autoimmunity in experimental biliary atresia Role of myeloid differentiation factor 88 in Rhesus rotavirus-induced biliary atresia.Aetiology of biliary atresia: what is actually known?Cholangiocytes as immune modulators in rotavirus-induced murine biliary atresia α-enolase autoantibodies cross-reactive to viral proteins in a mouse model of biliary atresia.Neonatal NK cells target the mouse duct epithelium via Nkg2d and drive tissue-specific injury in experimental biliary atresia.Clues to the etiology of bile duct injury in biliary atresia Regulatory T cells inhibit Th1 cell-mediated bile duct injury in murine biliary atresia.Prevention of cholestasis in the murine rotavirus-induced biliary atresia model using passive immunization and nonreplicating virus-like particles.Recent Advances in Etiology of Biliary Atresia.Interleukin-8: A critical chemokine in biliary atresia.The Sea Lamprey as an Etiological Model for Biliary Atresia.Reovirus type-2-triggered autoimmune cholangitis in extrahepatic bile ducts of weanling DBA/1J mice.Preferential TNFα signaling via TNFR2 regulates epithelial injury and duct obstruction in experimental biliary atresia Swiss outcomes in biliary atresia: are there lessons to be learned?Corticosteroid treatment in biliary atresia: Tonic or toast?Elevated Th17 cells accompanied by decreased regulatory T cells and cytokine environment in infants with biliary atresia.Development of a guinea pig model of perinatal cytomegalovirus-induced hepatobiliary injury.

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P2860

Armed CD4+ Th1 effector cells and activated macrophages participate in bile duct injury in murine biliary atresia.

http://www.wikidata.org/entity/Q35944551

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Armed CD4+ Th1 effector cells ...... ury in murine biliary atresia.

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Armed CD4+ Th1 effector cells ...... ury in murine biliary atresia.

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J.CLIM.2005.01.012

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Clinical Immunology

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Armed CD4+ Th1 effector cells ...... ury in murine biliary atresia.

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Brian L Kotzin

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Cara L Mack

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Rebecca M Tucker

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Ronald J Sokol

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P2860

Tumor necrosis factor receptor and Fas signaling mechanisms.

Etiopathogenesis of biliary atresia.

Biliary atresia is associated with CD4+ Th1 cell-mediated portal tract inflammation

Reovirus 3 not detected by reverse transcriptase-mediated polymerase chain reaction analysis of preserved tissue from infants with cholestatic liver disease.

Group A rotaviruses produce extrahepatic biliary obstruction in orally inoculated newborn mice.

Obstruction of extrahepatic bile ducts by lymphocytes is regulated by IFN-gamma in experimental biliary atresia.

A histological study of extrahepatic biliary atresia.

Myeloperoxidase-positive inflammatory cells participate in bile duct damage in primary biliary cirrhosis through nitric oxide-mediated reactions.

Mechanisms regulating the level of IL-2 mRNA in T lymphocytes.

Detection of reovirus RNA in hepatobiliary tissues from patients with extrahepatic biliary atresia and choledochal cysts.

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