Molecular changes in the polymerase genes (PA and PB1) associated with high pathogenicity of H5N1 influenza virus in mallard ducks.
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Connecting the study of wild influenza with the potential for pandemic diseaseGenetics, receptor binding property, and transmissibility in mammals of naturally isolated H9N2 Avian Influenza virusesHost Determinant Residue Lysine 627 Lies on the Surface of a Discrete, Folded Domain of Influenza Virus Polymerase PB2 SubunitMammalian adaptive mutations of the PA protein of highly pathogenic avian H5N1 influenza virus.Full factorial analysis of mammalian and avian influenza polymerase subunits suggests a role of an efficient polymerase for virus adaptationMolecular mechanisms of interspecies transmission and pathogenicity of influenza viruses: Lessons from the 2009 pandemic.Virulence and genetic compatibility of polymerase reassortant viruses derived from the pandemic (H1N1) 2009 influenza virus and circulating influenza A viruses.Genomic polymorphism of the pandemic A (H1N1) influenza viruses correlates with viral replication, virulence, and pathogenicity in vitro and in vivoA(H5N1) Virus Evolution in South East Asia.Mutations in polymerase genes enhanced the virulence of 2009 pandemic H1N1 influenza virus in miceH5N1 pathogenesis studies in mammalian models.Molecular characterization of highly pathogenic H5N1 avian influenza A viruses isolated from raccoon dogs in ChinaThe N-terminal region of the PA subunit of the RNA polymerase of influenza A/HongKong/156/97 (H5N1) influences promoter binding.Detection of mammalian virulence determinants in highly pathogenic avian influenza H5N1 viruses: multivariate analysis of published data.Generation and Comprehensive Analysis of Host Cell Interactome of the PA Protein of the Highly Pathogenic H5N1 Avian Influenza Virus in Mammalian CellsThe HA and NS genes of human H5N1 influenza A virus contribute to high virulence in ferrets.Association of RIG-I with innate immunity of ducks to influenzaGenotype diversity of H9N2 viruses isolated from wild birds and chickens in Hunan Province, China.Characterization of H5N1 influenza viruses isolated from humans in vitro.Reassortant H9N2 influenza viruses containing H5N1-like PB1 genes isolated from black-billed magpies in Southern ChinaThermostability of subpopulations of H2N3 influenza virus isolates from mallard ducks.Role of position 627 of PB2 and the multibasic cleavage site of the hemagglutinin in the virulence of H5N1 avian influenza virus in chickens and ducks.Virulence determinants of avian H5N1 influenza A virus in mammalian and avian hosts: role of the C-terminal ESEV motif in the viral NS1 protein.Amino acid substitutions in PB1 of avian influenza viruses influence pathogenicity and transmissibility in chickens.NP body domain and PB2 contribute to increased virulence of H5N1 highly pathogenic avian influenza viruses in chickens.Characterization of an H10N8 influenza virus isolated from Dongting lake wetland.Virulence of H5N1 virus in mice attenuates after in vitro serial passages.The PA protein directly contributes to the virulence of H5N1 avian influenza viruses in domestic ducksH5N1 influenza virus pathogenesis in genetically diverse mice is mediated at the level of viral load.Emergence of avian influenza viruses with enhanced transcription activity by a single amino acid substitution in the nucleoprotein during replication in chicken brains.Additional Evidence That the Polymerase Subunits Contribute to the Viral Replication and the Virulence of H5N1 Avian Influenza Virus Isolates in Mice.Emergence of mammalian species-infectious and -pathogenic avian influenza H6N5 virus with no evidence of adaptationPA-X decreases the pathogenicity of highly pathogenic H5N1 influenza A virus in avian species by inhibiting virus replication and host response.Construction of a gE-Deleted Pseudorabies Virus and Its Efficacy to the New-Emerging Variant PRV Challenge in the Form of Killed VaccineComplete genome sequence of an H9N2 avian influenza virus isolated from egret in Lake Dongting wetlandPA from an H5N1 highly pathogenic avian influenza virus activates viral transcription and replication and induces apoptosis and interferon expression at an early stage of infectionThe PB2, PA, HA, NP, and NS genes of a highly pathogenic avian influenza virus A/whooper swan/Mongolia/3/2005 (H5N1) are responsible for pathogenicity in ducksThree amino acid changes in PB1-F2 of highly pathogenic H5N1 avian influenza virus affect pathogenicity in mallard ducks.Complete Genome Sequence of a Novel Reassortant H3N6 Avian Influenza Virus Isolated from Domestic Green-Winged Teal.Wild ducks as long-distance vectors of highly pathogenic avian influenza virus (H5N1)
P2860
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P2860
Molecular changes in the polymerase genes (PA and PB1) associated with high pathogenicity of H5N1 influenza virus in mallard ducks.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Molecular changes in the polym ...... luenza virus in mallard ducks.
@ast
Molecular changes in the polym ...... luenza virus in mallard ducks.
@en
type
label
Molecular changes in the polym ...... luenza virus in mallard ducks.
@ast
Molecular changes in the polym ...... luenza virus in mallard ducks.
@en
prefLabel
Molecular changes in the polym ...... luenza virus in mallard ducks.
@ast
Molecular changes in the polym ...... luenza virus in mallard ducks.
@en
P2093
P2860
P356
P1433
P1476
Molecular changes in the polym ...... luenza virus in mallard ducks.
@en
P2093
D J Hulse-Post
E Hoffmann
T D Nguyen
P2860
P304
P356
10.1128/JVI.00435-07
P407
P577
2007-06-06T00:00:00Z