Rorγt+ innate lymphocytes and γδ T cells initiate psoriasiform plaque formation in mice. - Wikidata - wikimedia - TriplyDB

Rorγt+ innate lymphocytes and γδ T cells initiate psoriasiform plaque formation in mice.

Rorγt+ innate lymphocytes and γδ T cells initiate psoriasiform plaque formation in mice.

http://www.wikidata.org/entity/Q36005413

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Recent advances in understanding psoriasis Interleukin 17-Mediated Host Defense against Candida albicans Leukocyte integrins: role in leukocyte recruitment and as therapeutic targets in inflammatory disease Diversity of IL-17-producing T lymphocytes Innate lymphoid cells in the skin IL-22 Protects against Tissue Damage during Cutaneous Leishmaniasis Stromal Fibroblasts in Tertiary Lymphoid Structures: A Novel Target in Chronic Inflammation Group 3 innate lymphoid cells (ILC3s): Origin, differentiation, and plasticity in humans and mice Sterol metabolism controls T(H)17 differentiation by generating endogenous RORγ agonists Aldara activates TLR7-independent immune defence IMQ Induced K14-VEGF Mouse: A Stable and Long-Term Mouse Model of Psoriasis-Like Inflammation Psoriasiform dermatitis is driven by IL-36-mediated DC-keratinocyte crosstalk The role of dendritic cells in autoimmunity IL-21R signaling suppresses IL-17+ gamma delta T cell responses and production of IL-17 related cytokines in the lung at steady state and after Influenza A virus infection.IL-1β-dependent activation of dendritic epidermal T cells in contact hypersensitivity.Activation of the aryl hydrocarbon receptor dampens the severity of inflammatory skin conditions.Differential developmental requirement and peripheral regulation for dermal Vγ4 and Vγ6T17 cells in health and inflammation.Establishment and function of tissue-resident innate lymphoid cells in the skin.Radiation-Induced Dermatitis is Mediated by IL17-Expressing γδ T Cells.Nociceptive sensory neurons drive interleukin-23-mediated psoriasiform skin inflammation.A new player on the psoriasis block: IL-17A- and IL-22-producing innate lymphoid cells.Mannan induces ROS-regulated, IL-17A-dependent psoriasis arthritis-like disease in mice.IL-22 fate reporter reveals origin and control of IL-22 production in homeostasis and infection Mixing Signals: Molecular Turn Ons and Turn Offs for Innate γδ T-Cells.Stroma-derived interleukin-34 controls the development and maintenance of langerhans cells and the maintenance of microglia.Tumor necrosis factor superfamily in innate immunity and inflammation.Commensal bacteria and cutaneous immunity.The thymic cortical epithelium determines the TCR repertoire of IL-17-producing γδT cells.Downregulation of TNIP1 Expression Leads to Increased Proliferation of Human Keratinocytes and Severer Psoriasis-Like Conditions in an Imiquimod-Induced Mouse Model of Dermatitis Dermal γδ T cells--What have we learned?Cross Talk between Proliferative, Angiogenic, and Cellular Mechanisms Orchestred by HIF-1α in Psoriasis.Antibiotics in neonatal life increase murine susceptibility to experimental psoriasis.Cutting Edge: PD-1 Regulates Imiquimod-Induced Psoriasiform Dermatitis through Inhibition of IL-17A Expression by Innate γδ-Low T Cells.Inflammation induces dermal Vγ4+ γδT17 memory-like cells that travel to distant skin and accelerate secondary IL-17-driven responses.IL-15 receptor α signaling constrains the development of IL-17-producing γδ T cells.IL-6 Signaling in Myelomonocytic Cells Is Not Crucial for the Development of IMQ-Induced Psoriasis IL-22 regulates lymphoid chemokine production and assembly of tertiary lymphoid organs.Intestinal Microbiota Promotes Psoriasis-Like Skin Inflammation by Enhancing Th17 Response.Sterile inflammation - do innate lymphoid cell subsets play a role?Dermal γδ T Cells Do Not Freely Re-Circulate Out of Skin and Produce IL-17 to Promote Neutrophil Infiltration during Primary Contact Hypersensitivity

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P2860

Rorγt+ innate lymphocytes and γδ T cells initiate psoriasiform plaque formation in mice.

http://www.wikidata.org/entity/Q36005413

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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name

Rorγt+ innate lymphocytes and ...... form plaque formation in mice.

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Rorγt+ innate lymphocytes and ...... form plaque formation in mice.

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type

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Rorγt+ innate lymphocytes and ...... form plaque formation in mice.

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Rorγt+ innate lymphocytes and ...... form plaque formation in mice.

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Rorγt+ innate lymphocytes and ...... form plaque formation in mice.

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Rorγt+ innate lymphocytes and ...... form plaque formation in mice.

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P1433

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P1433

Journal of Clinical Investigation

P1476

Rorγt+ innate lymphocytes and ...... form plaque formation in mice.

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P2093

Alexander A Navarini

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Barbara Ingold

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Burkhard Becher

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Frank L Heppner

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Paulina Kulig

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Stanislav Pantelyushin

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Stefan Haak

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P2860

Effects of AIN457, a Fully Human Antibody to Interleukin-17A, on Psoriasis, Rheumatoid Arthritis, and Uveitis

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Small anti-viral compounds activate immune cells via the TLR7 MyD88-dependent signaling pathway

Comparison of ustekinumab and etanercept for moderate-to-severe psoriasis

Diversity of murine gamma genes and expression in fetal and adult T lymphocytes

GATA3 expression is decreased in psoriasis and during epidermal regeneration; induction by narrow-band UVB and IL-4

Pivotal role of dermal IL-17-producing γδ T cells in skin inflammation.

RORgammat and commensal microflora are required for the differentiation of mucosal interleukin 22-producing NKp46+ cells

Cutaneous immunosurveillance by self-renewing dermal gammadelta T cells

Cutting edge: Identification of a motile IL-17-producing gammadelta T cell population in the dermis

P304

2252-2256

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10.1172/JCI61862

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6

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122

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224870498

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22546855

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3366412

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