Abnormal glucose homeostasis in skeletal muscle-specific PGC-1alpha knockout mice reveals skeletal muscle-pancreatic beta cell crosstalk. - Wikidata - wikimedia - TriplyDB

Abnormal glucose homeostasis in skeletal muscle-specific PGC-1alpha knockout mice reveals skeletal muscle-pancreatic beta cell crosstalk.

Abnormal glucose homeostasis in skeletal muscle-specific PGC-1alpha knockout mice reveals skeletal muscle-pancreatic beta cell crosstalk.

http://www.wikidata.org/entity/Q36025294

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Skeletal muscle PGC-1α is required for maintaining an acute LPS-induced TNFα response Skeletal muscle PGC-1α controls whole-body lactate homeostasis through estrogen-related receptor α-dependent activation of LDH B and repression of LDH A Potential Role of Omega-3 Fatty Acids on the Myogenic Program of Satellite Cells Complex Coordination of Cell Plasticity by a PGC-1α-controlled Transcriptional Network in Skeletal Muscle Caloric restriction and exercise "mimetics'': Ready for prime time?Skeletal muscle as an endocrine organ: PGC-1α, myokines and exercise Road to exercise mimetics: targeting nuclear receptors in skeletal muscle Redox regulation of mitochondrial biogenesis Impact of exercise on muscle and nonmuscle organs Rosiglitazone-induced mitochondrial biogenesis in white adipose tissue is independent of peroxisome proliferator-activated receptor γ coactivator-1α PGC-1α is dispensable for exercise-induced mitochondrial biogenesis in skeletal muscle Exercise-training in young Drosophila melanogaster reduces age-related decline in mobility and cardiac performance Imbalanced OPA1 processing and mitochondrial fragmentation cause heart failure in mice Declining NAD(+) induces a pseudohypoxic state disrupting nuclear-mitochondrial communication during aging Inactivation of the mitochondrial carrier SLC25A25 (ATP-Mg2+/Pi transporter) reduces physical endurance and metabolic efficiency in mice Postnatal PPARdelta activation and myostatin inhibition exert distinct yet complimentary effects on the metabolic profile of obese insulin-resistant mice Exercise Intensity Modulates Glucose-Stimulated Insulin Secretion when Adjusted for Adipose, Liver and Skeletal Muscle Insulin Resistance.Adipocytokines in obesity and metabolic disease.A PGC-1α isoform induced by resistance training regulates skeletal muscle hypertrophy Disconnecting mitochondrial content from respiratory chain capacity in PGC-1-deficient skeletal muscle PGC-1alpha plays a functional role in exercise-induced mitochondrial biogenesis and angiogenesis but not fiber-type transformation in mouse skeletal muscle.Regulation of exercise-induced fiber type transformation, mitochondrial biogenesis, and angiogenesis in skeletal muscle PGC-1alpha regulation by exercise training and its influences on muscle function and insulin sensitivity.Endurance exercise rescues progeroid aging and induces systemic mitochondrial rejuvenation in mtDNA mutator mice PGC-1alpha negatively regulates hepatic FGF21 expression by modulating the heme/Rev-Erb(alpha) axis.Electric pulse stimulation of cultured murine muscle cells reproduces gene expression changes of trained mouse muscle PGC-1α regulates hepatic hepcidin expression and iron homeostasis in response to inflammation Mitochondrial dysfunction in diabetes: from molecular mechanisms to functional significance and therapeutic opportunities.Metabolic syndrome: from epidemiology to systems biology.Endothelial PGC-1α mediates vascular dysfunction in diabetes.Role of PGC-1α in exercise training- and resveratrol-induced prevention of age-associated inflammation Dual modulation of both lipid oxidation and synthesis by peroxisome proliferator-activated receptor-gamma coactivator-1alpha and -1beta in cultured myotubes.The role of mitochondria in the pathophysiology of skeletal muscle insulin resistance.Loss of Pgc-1α expression in aging mouse muscle potentiates glucose intolerance and systemic inflammation The role of PGC-1 coactivators in aging skeletal muscle and heart.Deficiency of the mitochondrial electron transport chain in muscle does not cause insulin resistance.Role of skeletal muscle mitochondrial density on exercise-stimulated lipid oxidation.PGC-1α and exercise in the control of body weight.The relationship between muscle fiber type-specific PGC-1α content and mitochondrial content varies between rodent models and humans GABP transcription factor (nuclear respiratory factor 2) is required for mitochondrial biogenesis.

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P2860

Abnormal glucose homeostasis in skeletal muscle-specific PGC-1alpha knockout mice reveals skeletal muscle-pancreatic beta cell crosstalk.

http://www.wikidata.org/entity/Q36025294

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2007 nî lūn-bûn

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Abnormal glucose homeostasis i ...... ancreatic beta cell crosstalk.

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Abnormal glucose homeostasis i ...... ancreatic beta cell crosstalk.

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Amarnath J Kurpad

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Cheol Soo Choi

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Christoph Handschin

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Dan Kawamori

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Jiang Hu

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Nicole Neubauer

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Rohit N Kulkarni

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Sheene Kim

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Sherry Chin

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Vamsi K Mootha

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P2860

Requirement for serum response factor for skeletal muscle growth and maturation revealed by tissue-specific gene deletion in mice

Erralpha and Gabpa/b specify PGC-1alpha-dependent oxidative phosphorylation gene expression that is altered in diabetic muscle

The estrogen-related receptor alpha (ERRalpha) functions in PPARgamma coactivator 1alpha (PGC-1alpha)-induced mitochondrial biogenesis

Coordinated reduction of genes of oxidative metabolism in humans with insulin resistance and diabetes: Potential role of PGC1 and NRF1

Oxidative metabolism and PGC-1beta attenuate macrophage-mediated inflammation

PGC-1alpha deficiency causes multi-system energy metabolic derangements: muscle dysfunction, abnormal weight control and hepatic steatosis

Decreased insulin-stimulated ATP synthesis and phosphate transport in muscle of insulin-resistant offspring of type 2 diabetic parents

Mechanisms controlling mitochondrial biogenesis and respiration through the thermogenic coactivator PGC-1

Activation of PPARgamma coactivator-1 through transcription factor docking

Control of hepatic gluconeogenesis through the transcriptional coactivator PGC-1

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11

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5911814

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