Pathogenesis of the slow disease of the central nervous system associated with WM 1504 E virus. I. Relationship of strain susceptibility and replication to disease.
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Retrovirus-induced murine motor neuron disease: mapping the determinant of spongiform degeneration within the envelope gene.Murine neurotropic retrovirus spongiform polioencephalomyelopathy: acceleration of disease by virus inoculum concentration.Protection against murine leukemia virus-induced spongiform myeloencephalopathy in mice overexpressing Bcl-2 but not in mice deficient for interleukin-6, inducible nitric oxide synthetase, ICE, Fas, Fas ligand, or TNF-R1 genesRheumatoid arthritis--a virus disease?Transmission in NFS/N mice of the heritable spongiform encephalopathy associated with the gray tremor mutationRetrovirus-induced spongiform neurodegeneration is mediated by unique central nervous system viral targeting and expression of env aloneOligodendrocytes are a major target of the toxicity of spongiogenic murine retroviruses.Disparate regions of envelope protein regulate syncytium formation versus spongiform encephalopathy in neurological disease induced by murine leukemia virus TR.Murine retroviral neurovirulence correlates with an enhanced ability ofvirus to infect selectively, replicate in, and activate resident microglial cellsInduction of focal spongiform neurodegeneration in developmentally resistant mice by implantation of murine retrovirus-infected microglia.Noninflammatory spongiform polioencephalomyelopathy caused by a neurotropic temperature-sensitive mutant of Moloney murine leukemia virus TB.Characterization of a neurologic disease induced by a polytropic murine retrovirus: evidence for differential targeting of ecotropic and polytropic viruses in the brainImmunopathology of natural and experimental lymphomas induced by wild mouse leukemia virus.Late virus replication events in microglia are required for neurovirulent retrovirus-induced spongiform neurodegeneration: evidence from neural progenitor-derived chimeric mouse brainsThe prion protein gene is dispensable for the development of spongiform myeloencephalopathy induced by the neurovirulent Cas-Br-E murine leukemia virusRetrovirus antigens in brains of mice with scrapie- and murine leukemia virus-induced spongiform encephalopathy.Murine retrovirus-induced spongiform encephalopathy: disease expression is dependent on postnatal development of the central nervous system.Age-dependent resistance to murine retrovirus-induced spongiform neurodegeneration results from central nervous system-specific restriction of virus replicationEcotropic Murine Leukemia Virus Infection of Glial Progenitors Interferes with Oligodendrocyte Differentiation: Implications for Neurovirulence.Murine retrovirus-induced spongiform encephalomyelopathy: host and viral factors which determine the length of the incubation periodSubstitution of the U3 long terminal repeat region of the neurotropic Cas-Br-E retrovirus affects its disease-inducing potentialMonoclonal antibodies specific for wild mouse neurotropic retrovirus: detection of comparable levels of virus replication in mouse strains susceptible and resistant to paralytic disease.Characterization of a progressive neurodegenerative disease induced by a temperature-sensitive Moloney murine leukemia virus infection.Role of immunity in age-related resistance to paralysis after murine leukemia virus infection.Host genetic determinants of neurological disease induced by Cas-Br-M murine leukemia virus.ts1, a Paralytogenic mutant of Moloney murine leukemia virus TB, has an enhanced ability to replicate in the central nervous system and primary nerve cell cultureMolecular cloning of infectious viral DNA from ecotropic neurotropic wild mouse retrovirusCellular localization of human immunodeficiency virus infection within the brains of acquired immune deficiency syndrome patients.Retrovirus-induced spongiform myeloencephalopathy in mice: regional distribution of infected target cells and neuronal loss occurring in the absence of viral expression in neurons.Retrovirus-induced spongiform encephalopathy: the 3'-end long terminal repeat-containing viral sequences influence the incidence of the disease and the specificity of the neurological syndrome.Transmissible spongiform encephalopathy in the gray tremor mutant mouse.Isolation of paralysis-inducing murine leukemia viruses from Friend virus passaged in ratsBiochemical and immunological characterization of murine leukemia viruses that are paralysis-inducing in rats.Identification of the infected target cell type in spongiform myeloencephalopathy induced by the neurotropic Cas-Br-E murine leukemia virus.Physical mapping of the paralysis-inducing determinant of a wild mouse ecotropic neurotropic retrovirus.
P2860
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P2860
Pathogenesis of the slow disease of the central nervous system associated with WM 1504 E virus. I. Relationship of strain susceptibility and replication to disease.
description
1977 nî lūn-bûn
@nan
1977年の論文
@ja
1977年論文
@yue
1977年論文
@zh-hant
1977年論文
@zh-hk
1977年論文
@zh-mo
1977年論文
@zh-tw
1977年论文
@wuu
1977年论文
@zh
1977年论文
@zh-cn
name
Pathogenesis of the slow disea ...... ty and replication to disease.
@ast
Pathogenesis of the slow disea ...... ty and replication to disease.
@en
type
label
Pathogenesis of the slow disea ...... ty and replication to disease.
@ast
Pathogenesis of the slow disea ...... ty and replication to disease.
@en
prefLabel
Pathogenesis of the slow disea ...... ty and replication to disease.
@ast
Pathogenesis of the slow disea ...... ty and replication to disease.
@en
P2093
P2860
P1476
Pathogenesis of the slow disea ...... ty and replication to disease.
@en
P2093
P2860
P304
P407
P577
1977-07-01T00:00:00Z