Caveolin-1 deficiency protects from pulmonary fibrosis by modulating epithelial cell senescence in mice. - Wikidata - wikimedia - TriplyDB

Caveolin-1 deficiency protects from pulmonary fibrosis by modulating epithelial cell senescence in mice.

Caveolin-1 deficiency protects from pulmonary fibrosis by modulating epithelial cell senescence in mice.

http://www.wikidata.org/entity/Q36110540

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Redox mechanisms in age-related lung fibrosis Rupatadine protects against pulmonary fibrosis by attenuating PAF-mediated senescence in rodents Mitochondria in the spotlight of aging and idiopathic pulmonary fibrosis Cellular senescence in normal and premature lung aging.Airway hyperresponsiveness is associated with airway remodeling but not inflammation in aging Cav1-/- mice.Regulation and involvement of matrix metalloproteinases in vascular diseases Effect of P2X7 receptor knockout on AQP-5 expression of type I alveolar epithelial cells.miR-199a-5p Is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1 Central role of cellular senescence in TSLP-induced airway remodeling in asthma.Simvastatin impairs the induction of pulmonary fibrosis caused by a western style diet: a preliminary study.Senescent Cells Contribute to the Physiological Remodeling of Aged Lungs Differences in forward angular light scattering distributions between M1 and M2 macrophages.Regulation of lung injury and fibrosis by p53-mediated changes in urokinase and plasminogen activator inhibitor-1.Modulation of bacterial pathogenesis by oppressive aging factors: insights into host-pneumococcal interaction strategies Role of type II pneumocyte senescence in radiation-induced lung fibrosis Pro-apoptotic Noxa is involved in ablative focal irradiation-induced lung injury The role of caveolae in the pathophysiology of lung diseases.Senescent cells: SASPected drivers of age-related pathologies.Role of cellular senescence and NOX4-mediated oxidative stress in systemic sclerosis pathogenesis.Senolytic drugs target alveolar epithelial cell function and attenuate experimental lung fibrosis ex vivo.Mitochondrial dysfunction in inflammatory responses and cellular senescence: pathogenesis and pharmacological targets for chronic lung diseases.Caveolin-1 in the Pathogenesis of Diabetic Nephropathy: Potential Therapeutic Target?IL1- and TGFβ-Nox4 signaling, oxidative stress and DNA damage response are shared features of replicative, oncogene-induced, and drug-induced paracrine 'bystander senescence'.miR-34a Inhibits Lung Fibrosis by Inducing Lung Fibroblast Senescence.Epithelial cell senescence: an adaptive response to pre-carcinogenic stresses?Senescent cells: an emerging target for diseases of ageing.Is There a Potential Therapeutic Role for Caveolin-1 in Fibrosis?Role of caveolin-1 expression in the pathogenesis of pulmonary edema in ventilator-induced lung injury.Potential contribution of alveolar epithelial type I cells to pulmonary fibrosis.Effects and mechanisms of pirfenidone, prednisone and acetylcysteine on pulmonary fibrosis in rat idiopathic pulmonary fibrosis models.Caveolin-1 deficiency protects against mesangial matrix expansion in a mouse model of type 1 diabetic nephropathy.

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P2860

Caveolin-1 deficiency protects from pulmonary fibrosis by modulating epithelial cell senescence in mice.

http://www.wikidata.org/entity/Q36110540

description

2012 nî lūn-bûn

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American Journal of Respiratory Cell and Molecular Biology

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Caveolin-1 deficiency protects ...... elial cell senescence in mice.

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Christopher Brampton

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Claude Jourdan Le Saux

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Michael Kasper

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Pooja Shivshankar

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Shelley Miyasato

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Victor J Thannickal

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P2860

Caveolin-1 regulates transforming growth factor (TGF)-beta/SMAD signaling through an interaction with the TGF-beta type I receptor

Tenascins: regulation and putative functions during pathological stress

Idiopathic pulmonary fibrosis: prevailing and evolving hypotheses about its pathogenesis and implications for therapy

Loss of caveolae, vascular dysfunction, and pulmonary defects in caveolin-1 gene-disrupted mice

Telomerase mutations in families with idiopathic pulmonary fibrosis

The senescence-associated secretory phenotype: the dark side of tumor suppression

Caveolin-1, cellular senescence and pulmonary emphysema.

Use of senescence-accelerated mouse model in bleomycin-induced lung injury suggests that bone marrow-derived cells can alter the outcome of lung injury in aged mice.

Inflammatory networks during cellular senescence: causes and consequences.

Diminished prostaglandin E2 contributes to the apoptosis paradox in idiopathic pulmonary fibrosis

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pulmonary fibrosis

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