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Diminished prostaglandin E2 contributes to the apoptosis paradox in idiopathic pulmonary fibrosis

Diminished prostaglandin E2 contributes to the apoptosis paradox in idiopathic pulmonary fibrosis

http://www.wikidata.org/entity/Q33988131

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Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosis Fibroblasts in fibrosis: novel roles and mediators Myofibroblasts and lung fibrosis induced by carbon nanotube exposure 17β-Estradiol alters rat type-II alveolar cell recovery from high levels of ozone.Mononuclear phagocytes and airway epithelial cells: novel sources of matrix metalloproteinase-8 (MMP-8) in patients with idiopathic pulmonary fibrosis.Matrix metalloproteinase-19 is a key regulator of lung fibrosis in mice and humans Role of the lysophospholipid mediators lysophosphatidic acid and sphingosine 1-phosphate in lung fibrosis.Prostaglandin E2 inhibits α-smooth muscle actin transcription during myofibroblast differentiation via distinct mechanisms of modulation of serum response factor and myocardin-related transcription factor-A Proteases Revisited: Roles and Therapeutic Implications in Fibrosis.Targeting interleukin-13 with tralokinumab attenuates lung fibrosis and epithelial damage in a humanized SCID idiopathic pulmonary fibrosis model.Pathogenesis of idiopathic pulmonary fibrosis Lung fibroblasts from patients with idiopathic pulmonary fibrosis exhibit genome-wide differences in DNA methylation compared to fibroblasts from nonfibrotic lung Hydrogel scaffolds as in vitro models to study fibroblast activation in wound healing and disease.Lack of microsomal prostaglandin E synthase-1 reduces cardiac function following angiotensin II infusion Lung myofibroblasts are characterized by down-regulated cyclooxygenase-2 and its main metabolite, prostaglandin E2.Novel therapeutic approaches for pulmonary fibrosis.Prostaglandin E2 and the pathogenesis of pulmonary fibrosis.Inhibition of myocardin-related transcription factor/serum response factor signaling decreases lung fibrosis and promotes mesenchymal cell apoptosis.Prostaglandin Transporter (PGT/SLCO2A1) Protects the Lung from Bleomycin-Induced Fibrosis.Survivin expression induced by endothelin-1 promotes myofibroblast resistance to apoptosis.Prostaglandin E2 as an inhibitory modulator of fibrogenesis in human lung allografts Plakoglobin expression in fibroblasts and its role in idiopathic pulmonary fibrosis.Characterization of Lung Fibroblasts More than Two Decades after Mustard Gas Exposure The lysophosphatidic acid receptor LPA1 promotes epithelial cell apoptosis after lung injury Caveolin-1 deficiency protects from pulmonary fibrosis by modulating epithelial cell senescence in mice.Invasive breast cancer induces laminin-332 upregulation and integrin β4 neoexpression in myofibroblasts to confer an anoikis-resistant phenotype during tissue remodeling.Genetic partitioning of interleukin-6 signalling in mice dissociates Stat3 from Smad3-mediated lung fibrosis.Epigenetic regulation of cyclooxygenase-2 by methylation of c8orf4 in pulmonary fibrosis Targeting Inhibitor of Apoptosis Proteins Protects from Bleomycin-Induced Lung Fibrosis.Matrices of physiologic stiffness potently inactivate idiopathic pulmonary fibrosis fibroblasts.Histone modifications are responsible for decreased Fas expression and apoptosis resistance in fibrotic lung fibroblasts.Computational Modeling Predicts Simultaneous Targeting of Fibroblasts and Epithelial Cells Is Necessary for Treatment of Pulmonary Fibrosis.X-linked inhibitor of apoptosis regulates lung fibroblast resistance to Fas-mediated apoptosis.Heparin and LPS-induced COX-2 expression in airway cells: a link between its anti-inflammatory effects and GAG sulfation.Skewed Lung CCR4 to CCR6 CD4+ T Cell Ratio in Idiopathic Pulmonary Fibrosis Is Associated with Pulmonary Function.Mouse Lung Fibroblast Resistance to Fas-Mediated Apoptosis Is Dependent on the Baculoviral Inhibitor of Apoptosis Protein 4 and the Cellular FLICE-Inhibitory Protein.Update in diffuse parenchymal lung disease 2010.Myofibroblast differentiation and survival in fibrotic disease.Targeting defective Toll-like receptor-3 function and idiopathic pulmonary fibrosis.Regulation and Relevance of Myofibroblast Responses in Idiopathic Pulmonary Fibrosis.

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Diminished prostaglandin E2 contributes to the apoptosis paradox in idiopathic pulmonary fibrosis

http://www.wikidata.org/entity/Q33988131

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Diminished prostaglandin E2 co ...... idiopathic pulmonary fibrosis

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Diminished prostaglandin E2 co ...... idiopathic pulmonary fibrosis

@en

type

label

Diminished prostaglandin E2 co ...... idiopathic pulmonary fibrosis

@ast

Diminished prostaglandin E2 co ...... idiopathic pulmonary fibrosis

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prefLabel

Diminished prostaglandin E2 co ...... idiopathic pulmonary fibrosis

@ast

Diminished prostaglandin E2 co ...... idiopathic pulmonary fibrosis

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RCCM.200905-0674OC

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American Journal of Respiratory and Critical Care Medicine

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Diminished prostaglandin E2 co ...... idiopathic pulmonary fibrosis

@en

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Andrew G Nicholson

http://www.w3.org/2001/XMLSchema#string

Andrew J Thorley

http://www.w3.org/2001/XMLSchema#string

Geoffrey J Laurent

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Iona C Evans

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Paul F Mercer

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Rachel C Chambers

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Robin J McAnulty

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Stephen E Bottoms

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Teresa D Tetley

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Toby M Maher

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P2860

Phosphatidylinositol 3-kinase/Akt positively regulates fas (CD95)-mediated apoptosis in epidermal Cl41 cells

Altered expression of membrane-bound and soluble CD95/Fas contributes to the resistance of fibrotic lung fibroblasts to FasL induced apoptosis

Quantification of collagen and proteoglycan deposition in a murine model of airway remodelling

Idiopathic pulmonary fibrosis: prevailing and evolving hypotheses about its pathogenesis and implications for therapy

Cyclooxygenase-2 deficiency results in a loss of the anti-proliferative response to transforming growth factor-beta in human fibrotic lung fibroblasts and promotes bleomycin-induced pulmonary fibrosis in mice

Prostaglandin E2 reduces radiation-induced epithelial apoptosis through a mechanism involving AKT activation and bax translocation

Alveolar epithelial cell mesenchymal transition develops in vivo during pulmonary fibrosis and is regulated by the extracellular matrix

Attenuation of bleomycin-induced pneumopathy in mice by a caspase inhibitor.

Prostaglandin E(2) induces fibroblast apoptosis by modulating multiple survival pathways.

Evidence of type II pneumocyte apoptosis in the pathogenesis of idiopathic pulmonary fibrosis (IFP)/usual interstitial pneumonia (UIP).

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73-82

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10.1164/RCCM.200905-0674OC

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1

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2010-03-04T00:00:00Z

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20203246

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pulmonary fibrosis

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2902759

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