Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants. - Wikidata - wikimedia - TriplyDB

Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants.

Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants.

http://www.wikidata.org/entity/Q36142180

about

An Epstein-Barr virus anti-apoptotic protein constitutively expressed in transformed cells and implicated in burkitt lymphomagenesis: the Wp/BHRF1 link Revisiting G3BP1 as a RasGAP binding protein: sensitization of tumor cells to chemotherapy by the RasGAP 317-326 sequence does not involve G3BP1 Proteasome inhibitors evoke latent tumor suppression programs in pro-B MLL leukemias through MLL-AF4 PI3K-dependent phosphorylation of Fbw7 modulates substrate degradation and activity Domain-specific c-Myc ubiquitylation controls c-Myc transcriptional and apoptotic activity Myc protein is stabilized by suppression of a novel E3 ligase complex in cancer cells Oncogenic transformation in the absence of Xrcc4 targets peripheral B cells that have undergone editing and switching The Bcl-2 apoptotic switch in cancer development and therapy Targeted deletion reveals essential and overlapping functions of the miR-17 through 92 family of miRNA clusters miR-19 is a key oncogenic component of mir-17-92 Epstein-Barr virus and Burkitt lymphoma Regulation of Bim in Health and Disease MYC and the art of microRNA maintenance A phosphodiesterase 4B-dependent interplay between tumor cells and the microenvironment regulates angiogenesis in B-cell lymphoma.A role for E2F activities in determining the fate of Myc-induced lymphomagenesis A System to Study Aneuploidy In Vivo Putting the pieces together: How is the mitochondrial pathway of apoptosis regulated in cancer and chemotherapy?A SUMOylation-dependent transcriptional subprogram is required for Myc-driven tumorigenesis p53 status dictates responses of B lymphomas to monotherapy with proteasome inhibitors Inhibition of mTOR-kinase destabilizes MYCN and is a potential therapy for MYCN-dependent tumors Autophagy inhibition enhances therapy-induced apoptosis in a Myc-induced model of lymphoma Molecular requirements for transformation of fallopian tube epithelial cells into serous carcinoma.A look to future directions in gene therapy research for monogenic diseases.CpG island methylation in a mouse model of lymphoma is driven by the genetic configuration of tumor cells.Immortalization of human neural stem cells with the c-myc mutant T58A.Epstein-barr virus latency in B cells leads to epigenetic repression and CpG methylation of the tumour suppressor gene Bim Phosphorylation by Cdk2 is required for Myc to repress Ras-induced senescence in cotransformation.Nucleophosmin suppresses oncogene-induced apoptosis and senescence and enhances oncogenic cooperation in cells with genomic instability.MYC and the control of apoptosis.Burkitt lymphoma in Brazil is characterized by geographically distinct clinicopathologic features.MYC Deregulation in Primary Human Cancers.Loss of p73 promotes dissemination of Myc-induced B cell lymphomas in mice Mechanistic insight into Myc stabilization in breast cancer involving aberrant Axin1 expression Cancer gene discovery in hepatocellular carcinoma Developments in Burkitt's lymphoma: novel cooperations in oncogenic MYC signaling.Therapeutic potential of a synthetic lethal interaction between the MYC proto-oncogene and inhibition of aurora-B kinase.Promotion of direct reprogramming by transformation-deficient Myc.Repression of BIM mediates survival signaling by MYC and AKT in high-risk T-cell acute lymphoblastic leukemia.Cell and gene therapy for severe heart failure patients: the time and place for Pim-1 kinase.MYC cofactors: molecular switches controlling diverse biological outcomes.

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P2860

Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants.

http://www.wikidata.org/entity/Q36142180

description

2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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2005年论文

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2005年论文

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name

Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants.

@ast

Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants.

@en

type

label

Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants.

@ast

Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants.

@en

prefLabel

Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants.

@ast

Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants.

@en

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Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants.

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Andreas Herbst

http://www.w3.org/2001/XMLSchema#string

Anka Bric

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John L Cleveland

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Jonas A Nilsson

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Michael T Hemann

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Scott W Lowe

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P2860

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

Bim: a novel member of the Bcl-2 family that promotes apoptosis

c-Myc augments gamma irradiation-induced apoptosis by suppressing Bcl-XL

Tumor suppression by Ink4a-Arf: progress and puzzles

Induction of apoptosis in fibroblasts by c-myc protein

Destruction of Myc by ubiquitin-mediated proteolysis: cancer-associated and transforming mutations stabilize Myc.

c-Myc target genes involved in cell growth, apoptosis, and metabolism

The c-Myc transactivation domain is a direct modulator of apoptotic versus proliferative signals.

Dissecting p53 tumor suppressor functions in vivo.

The life cycle of C-myc: from synthesis to degradation.

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7052

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436

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Carlos Cordon-Cardo

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1032103889

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16094360

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4599579

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