Requirement for endocytic antigen processing and influence of invariant chain and H-2M deficiencies in CNS autoimmunity
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The crystal structure of myelin oligodendrocyte glycoprotein, a key autoantigen in multiple sclerosisHuman leukocyte Antigen-DM polymorphisms in autoimmune diseasesCD24 controls expansion and persistence of autoreactive T cells in the central nervous system during experimental autoimmune encephalomyelitisPathogenesis of brain and spinal cord atrophy in multiple sclerosis.Apolipoprotein E mediation of neuro-inflammation in a murine model of multiple sclerosis.Immunodominant T cell determinants of aquaporin-4, the autoantigen associated with neuromyelitis optica.A switch in pathogenic mechanism in myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitis in IFN-γ-inducible lysosomal thiol reductase-free mice.Developmental maturation of innate immune cell function correlates with susceptibility to central nervous system autoimmunity.Immunodominant T-cell epitopes of MOG reside in its transmembrane and cytoplasmic domains in EAE.Traumatic brain injury causes selective, CD74-dependent peripheral lymphocyte activation that exacerbates neurodegeneration.Limited sufficiency of antigen presentation by dendritic cells in models of central nervous system autoimmunity.Myelin oligodendrocyte glycoprotein-specific T and B cells cooperate to induce a Devic-like disease in mice.Nogo-receptor 1 deficiency has no influence on immune cell repertoire or function during experimental autoimmune encephalomyelitis.B cell antigen presentation is sufficient to drive neuroinflammation in an animal model of multiple sclerosis.A Single Amino Acid Substitution Prevents Recognition of a Dominant Human Aquaporin-4 Determinant in the Context of HLA-DRB1*03:01 by a Murine TCR.B-cell activation influences T-cell polarization and outcome of anti-CD20 B-cell depletion in central nervous system autoimmunityAquaporin 4-specific T cells in neuromyelitis optica exhibit a Th17 bias and recognize Clostridium ABC transporterIL-12/IL-23p40 Is Highly Expressed in Secondary Lymphoid Organs and the CNS during All Stages of EAE, but Its Deletion Does Not Affect Disease Perpetuation.Low dose zymosan ameliorates both chronic and relapsing experimental autoimmune encephalomyelitis.Critical role of activation induced cytidine deaminase in experimental autoimmune encephalomyelitis.The neonatal CNS is not conducive for encephalitogenic Th1 T cells and B cells during experimental autoimmune encephalomyelitisVitamin D3 Suppresses Class II Invariant Chain Peptide Expression on Activated B-Lymphocytes: A Plausible Mechanism for Downregulation of Acute Inflammatory Conditions.Suppression of autoimmune retinal disease by lovastatin does not require Th2 cytokine induction.Exposing the Specific Roles of the Invariant Chain Isoforms in Shaping the MHC Class II Peptidome.MHC class II-dependent B cell APC function is required for induction of CNS autoimmunity independent of myelin-specific antibodies.The immunopathophysiology of multiple sclerosis.The role of myelin oligodendrocyte glycoprotein in autoimmune demyelination: a target for multiple sclerosis therapy?Immune surveillance of the central nervous system in multiple sclerosis--relevance for therapy and experimental models.Does MOG Ig-positive AQP4-seronegative opticospinal inflammatory disease justify a diagnosis of NMO spectrum disorder?Tregs restrain dendritic cell autophagy to ameliorate autoimmunity.Virus-encoded ectopic CD74 enhances poxvirus vaccine efficacy.Cutting edge: Conditional MHC class II expression reveals a limited role for B cell antigen presentation in primary and secondary CD4 T cell responses.TLR-mediated B cell activation results in ectopic CLIP expression that promotes B cell-dependent inflammation.MHCII is required for α-synuclein-induced activation of microglia, CD4 T cell proliferation, and dopaminergic neurodegeneration.The protease inhibitor, Bowman-Birk Inhibitor, suppresses experimental autoimmune encephalomyelitis: a potential oral therapy for multiple sclerosis.
P2860
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P2860
Requirement for endocytic antigen processing and influence of invariant chain and H-2M deficiencies in CNS autoimmunity
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Requirement for endocytic anti ...... ficiencies in CNS autoimmunity
@ast
Requirement for endocytic anti ...... ficiencies in CNS autoimmunity
@en
type
label
Requirement for endocytic anti ...... ficiencies in CNS autoimmunity
@ast
Requirement for endocytic anti ...... ficiencies in CNS autoimmunity
@en
prefLabel
Requirement for endocytic anti ...... ficiencies in CNS autoimmunity
@ast
Requirement for endocytic anti ...... ficiencies in CNS autoimmunity
@en
P2093
P2860
P356
P1476
Requirement for endocytic anti ...... ficiencies in CNS autoimmunity
@en
P2093
A J Slavin
E K Bikoff
H L Weiner
J C Patarroyo
S S Zamvil
P2860
P304
P356
10.1172/JCI13360
P407
P577
2001-10-01T00:00:00Z