Chronic inflammatory pain leads to increased blood-brain barrier permeability and tight junction protein alterations.
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Physiology and pathophysiology of the blood-brain barrier: P-glycoprotein and occludin trafficking as therapeutic targets to optimize central nervous system drug deliveryPeptides at the blood brain barrier: Knowing me knowing youInduction of blood brain barrier tight junction protein alterations by CD8 T cells.α1-Acid glycoprotein induced effects in rat brain microvessel endothelial cells.Acute and chronic deficits in the urinary bladder after spinal contusion injury in the adult ratMouse models of neurological disorders: a view from the blood-brain barrier.CD8 T cell-initiated vascular endothelial growth factor expression promotes central nervous system vascular permeability under neuroinflammatory conditions.The role of the blood-brain barrier in the development and treatment of migraine and other pain disorders.Activation of C6 glioblastoma cell ceruloplasmin expression by neighboring human brain endothelia-derived interleukins in an in vitro blood-brain barrier model system.Protein kinase C activation modulates reversible increase in cortical blood-brain barrier permeability and tight junction protein expression during hypoxia and posthypoxic reoxygenation.Neuregulin1-β decreases IL-1β-induced neutrophil adhesion to human brain microvascular endothelial cellsBlood-brain barrier disruption and oxidative stress in guinea pig after systemic exposure to modified cell-free hemoglobin.Targeting the neurovascular unit for treatment of neurological disorders.Pharmacological attenuation of chronic alcoholic pancreatitis induced hypersensitivity in rats.Central histamine H3 receptor signaling negatively regulates susceptibility to autoimmune inflammatory disease of the CNS.Targeting blood-brain barrier changes during inflammatory pain: an opportunity for optimizing CNS drug delivery.Dendritic cell transmigration through brain microvessel endothelium is regulated by MIP-1alpha chemokine and matrix metalloproteinases.Tight junction protein expression and barrier properties of immortalized mouse brain microvessel endothelial cells.Activation of peroxisome proliferator-activated receptor gamma (PPARgamma) suppresses Rho GTPases in human brain microvascular endothelial cells and inhibits adhesion and transendothelial migration of HIV-1 infected monocytesCD8 T cell-initiated blood-brain barrier disruption is independent of neutrophil supportSufficient virus-neutralizing antibody in the central nerve system improves the survival of rabid rats.STAT1 signaling modulates HIV-1-induced inflammatory responses and leukocyte transmigration across the blood-brain barrier.Triggers and effectors of oxidative stress at blood-brain barrier level: relevance for brain ageing and neurodegenerationNeurovascular unit in chronic pain.Nociceptive inhibition prevents inflammatory pain induced changes in the blood-brain barrier.Occludin oligomeric assembly at tight junctions of the blood-brain barrier is disrupted by peripheral inflammatory hyperalgesia.MyD88-deficient mice exhibit decreased parasite-induced immune responses but reduced disease severity in a murine model of neurocysticercosis.Biphasic cytoarchitecture and functional changes in the BBB induced by chronic inflammatory painTransforming growth factor-beta signaling alters substrate permeability and tight junction protein expression at the blood-brain barrier during inflammatory painNonpharmacological Interventions in Targeting Pain-Related Brain PlasticityTight junction in blood-brain barrier: an overview of structure, regulation, and regulator substances.Protein Interactions at Endothelial Junctions and Signaling Mechanisms Regulating Endothelial Permeability.Hypoxic Stress and Inflammatory Pain Disrupt Blood-Brain Barrier Tight Junctions: Implications for Drug Delivery to the Central Nervous SystemDifferential effects of hydrocortisone and TNFalpha on tight junction proteins in an in vitro model of the human blood-brain barrier.Occludin regulates glucose uptake and ATP production in pericytes by influencing AMP-activated protein kinase activity.Serum Neuroinflammatory Disease-Induced Central Nervous System Proteins Predict Clinical Onset of Experimental Autoimmune Encephalomyelitis.Regulation of Toll-like receptors in the choroid plexus in the immature brain after systemic inflammatory stimuli.Astroglial integrins in the development and regulation of neurovascular unitsExperimental obstructive jaundice alters claudin-4 expression in intestinal mucosa: effect of bombesin and neurotensin.Amyloid-beta-induced occludin down-regulation and increased permeability in human brain endothelial cells is mediated by MAPK activation.
P2860
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P2860
Chronic inflammatory pain leads to increased blood-brain barrier permeability and tight junction protein alterations.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Chronic inflammatory pain lead ...... junction protein alterations.
@ast
Chronic inflammatory pain lead ...... junction protein alterations.
@en
type
label
Chronic inflammatory pain lead ...... junction protein alterations.
@ast
Chronic inflammatory pain lead ...... junction protein alterations.
@en
prefLabel
Chronic inflammatory pain lead ...... junction protein alterations.
@ast
Chronic inflammatory pain lead ...... junction protein alterations.
@en
P2093
P2860
P921
P1476
Chronic inflammatory pain lead ...... junction protein alterations.
@en
P2093
Jason D Huber
Richard D Egleton
Tracy A Brooks
P2860
P304
P356
10.1152/AJPHEART.01288.2004
P577
2005-03-25T00:00:00Z