Inflammation, the complement system and the diseases of aging.
about
Systemic LPS causes chronic neuroinflammation and progressive neurodegenerationCD4 T cells in immunity and immunotherapy of Alzheimer's diseaseThe effects of age on pain sensitivity: preclinical studiesAge-dependent retinal iron accumulation and degeneration in hepcidin knockout miceCirculating Autoantibodies in Age-Related Macular Degeneration Recognize Human Macular Tissue Antigens Implicated in Autophagy, Immunomodulation, and Protection from Oxidative Stress and ApoptosisCellular and molecular mechanisms of immunomodulation in the brain through environmental enrichment.Neuroinflammation in Parkinson's disease: its role in neuronal death and implications for therapeutic interventionEffects of age on thermal sensitivity in the rat.Retinal ultrastructure of murine models of dry age-related macular degeneration (AMD).Ubiquitin/proteasome pathway impairment in neurodegeneration: therapeutic implicationsThe oral iron chelator deferiprone protects against iron overload-induced retinal degenerationComplement 3 and factor h in human cerebrospinal fluid in Parkinson's disease, Alzheimer's disease, and multiple-system atrophyP2Y receptors in Alzheimer's disease.Brain progranulin expression in GRN-associated frontotemporal lobar degenerationAlzheimer risk associated with a copy number variation in the complement receptor 1 increasing C3b/C4b binding sites.Regulator of G-protein signaling-10 negatively regulates NF-κB in microglia and neuroprotects dopaminergic neurons in hemiparkinsonian rats.Systemic administration of the iron chelator deferiprone protects against light-induced photoreceptor degeneration in the mouse retinaInduction of microglial toll-like receptor 4 by prothrombin kringle-2: a potential pathogenic mechanism in Parkinson's diseaseActivation of MyD88-dependent TLR1/2 signaling by misfolded α-synuclein, a protein linked to neurodegenerative disorders.Neuronal RING finger protein 11 (RNF11) regulates canonical NF-κB signaling.Effect of complement CR1 on brain amyloid burden during aging and its modification by APOE genotypeSquamosamide derivative FLZ protects dopaminergic neurons against inflammation-mediated neurodegeneration through the inhibition of NADPH oxidase activity.Antibodies against small heat-shock proteins in Alzheimer's disease as a part of natural human immune repertoire or activation of humoral response?Proteomics of Alzheimer's disease: understanding mechanisms and seeking biomarkers.Age-related macular degeneration and the immune response: implications for therapy.Tumor necrosis factor-alpha potentiates intraneuronal Ca2+ signaling via regulation of the inositol 1,4,5-trisphosphate receptor.A journey with Tony Hugli up the inflammatory cascade towards the auto-digestion hypothesisRole of complement cascade in abdominal aortic aneurysmsImmunological outcomes of exercise in older adultsChronic neuron-specific tumor necrosis factor-alpha expression enhances the local inflammatory environment ultimately leading to neuronal death in 3xTg-AD miceNormal aging involves modulation of specific inflammatory markers in the rat retina and choroid.The Oral Iron Chelator Deferiprone Protects Against Retinal Degeneration Induced through Diverse Mechanisms.Regulator of G-protein signaling 10 promotes dopaminergic neuron survival via regulation of the microglial inflammatory responseProteasome inactivation promotes p38 mitogen-activated protein kinase-dependent phosphatidylinositol 3-kinase activation and increases interleukin-8 production in retinal pigment epithelial cells.Vitamin B-6 intake is inversely related to, and the requirement is affected by, inflammation status.Inflammation and the pathogenesis of age-related macular degeneration.Neuronal nicotinic receptors as novel targets for inflammation and neuroprotection: mechanistic considerations and clinical relevance.Loss of P2Y₂ nucleotide receptors enhances early pathology in the TgCRND8 mouse model of Alzheimer's disease.Microglial activation and chronic neurodegeneration.Genetic analysis of pathways to Parkinson disease.
P2860
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P2860
Inflammation, the complement system and the diseases of aging.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Inflammation, the complement system and the diseases of aging.
@ast
Inflammation, the complement system and the diseases of aging.
@en
type
label
Inflammation, the complement system and the diseases of aging.
@ast
Inflammation, the complement system and the diseases of aging.
@en
prefLabel
Inflammation, the complement system and the diseases of aging.
@ast
Inflammation, the complement system and the diseases of aging.
@en
P2093
P1476
Inflammation, the complement system and the diseases of aging.
@en
P2093
Andis Klegeris
Edith G McGeer
Patrick L McGeer
P356
10.1016/J.NEUROBIOLAGING.2005.08.008
P478
26 Suppl 1
P577
2005-09-29T00:00:00Z