Transmembrane signaling by an insulin receptor lacking a cytoplasmic beta-subunit domain.

Item

http://www.wikidata.org/entity/Q36295758

description

1993 nî lūn-bûn

@nan

1993年の論文

@ja

1993年学术文章

@wuu

1993年学术文章

@zh-cn

1993年学术文章

@zh-hans

1993年学术文章

@zh-my

1993年学术文章

@zh-sg

1993年學術文章

@yue

1993年學術文章

@zh

1993年學術文章

@zh-hant

name

Transmembrane signaling by an insulin receptor lacking a cytoplasmic beta-subunit domain.

@ast

Transmembrane signaling by an insulin receptor lacking a cytoplasmic beta-subunit domain.

@en

type

Item

label

Transmembrane signaling by an insulin receptor lacking a cytoplasmic beta-subunit domain.

@ast

Transmembrane signaling by an insulin receptor lacking a cytoplasmic beta-subunit domain.

@en

prefLabel

Transmembrane signaling by an insulin receptor lacking a cytoplasmic beta-subunit domain.

@ast

Transmembrane signaling by an insulin receptor lacking a cytoplasmic beta-subunit domain.

@en

P1476

Transmembrane signaling by an insulin receptor lacking a cytoplasmic beta-subunit domain.

@en

P2093

C M Anderson

http://www.w3.org/2001/XMLSchema#string

J Kusari

http://www.w3.org/2001/XMLSchema#string

T Sasaoka

http://www.w3.org/2001/XMLSchema#string

W J Langlois

http://www.w3.org/2001/XMLSchema#string

Y Takata

http://www.w3.org/2001/XMLSchema#string

P2860

DNA sequencing with chain-terminating inhibitors

Replacement of insulin receptor tyrosine residues 1162 and 1163 compromises insulin-stimulated kinase activity and uptake of 2-deoxyglucose

Structure of the insulin receptor substrate IRS-1 defines a unique signal transduction protein

Human insulin receptor and its relationship to the tyrosine kinase family of oncogenes

Replacement of insulin receptor tyrosine residues 1162 and 1163 does not alter the mitogenic effect of the hormone.

Detection of an alteration in the insulin-receptor gene in a patient with insulin resistance, acanthosis nigricans, and the polycystic ovary syndrome (type A insulin resistance).

Replacement of lysine residue 1030 in the putative ATP-binding region of the insulin receptor abolishes insulin- and antibody-stimulated glucose uptake and receptor kinase activity.

Insulin-induced surface redistribution regulates internalization of the insulin receptor and requires its autophosphorylation.

Transdominant inhibition of tyrosine kinase activity in mutant insulin/insulin-like growth factor I hybrid receptors.

Insulin and growth factor effects on c-fos expression in normal and protein kinase C-deficient 3T3-L1 fibroblasts and adipocytes.

P304

4379-4383

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P31

scholarly article

P356

10.1073/PNAS.90.10.4379

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Jerrold M. Olefsky

P577

1993-05-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

14693595

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P698

8506276

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P921

transmembrane protein

P932

46514

http://www.w3.org/2001/XMLSchema#string