A genome-wide RNAi screen identifies core components of the G₂-M DNA damage checkpoint.
about
High-dose irradiation induces cell cycle arrest, apoptosis, and developmental defects during Drosophila oogenesisA functional screen reveals an extensive layer of transcriptional and splicing control underlying RAS/MAPK signaling in DrosophilaA genomic approach to identify hybrid incompatibility genesThe histone demethylase dKDM5/LID interacts with the SIN3 histone deacetylase complex and shares functional similarities with SIN3.SUMOylation of ATRIP potentiates DNA damage signaling by boosting multiple protein interactions in the ATR pathwayRole of checkpoint kinase 1 (Chk1) in the mechanisms of resistance to histone deacetylase inhibitorsSafeguarding genetic information in Drosophila.A Genetic Screen for Dominant Enhancers of the Cell-Cycle Regulator α-Endosulfine Identifies Matrimony as a Strong Functional Interactor in Drosophila.A genetic interaction map of cell cycle regulators.Direct Binding to Replication Protein A (RPA)-coated Single-stranded DNA Allows Recruitment of the ATR Activator TopBP1 to Sites of DNA Damage.NEK8 regulates DNA damage-induced RAD51 foci formation and replication fork protectionA small RNA response at DNA ends in DrosophilaAn essential cell cycle regulation gene causes hybrid inviability in DrosophilaRoles for the Histone Modifying and Exchange Complex NuA4 in Cell Cycle Progression in Drosophila melanogaster.Computational approaches for analyzing information flow in biological networks.Unraveling DNA damage response-signaling networks through systems approaches.Distinct mechanisms of transcriptional pausing orchestrated by GAGA factor and M1BP, a novel transcription factor.Computational approaches for the identification of cancer genes and pathwaysEvaluation of cytotoxicity and DNA damage response with analysis of intracellular ATM signaling pathwaysDNA Replication Control During Drosophila Development: Insights into the Onset of S Phase, Replication Initiation, and Fork Progression.Initiation of Drosophila chorion gene amplification requires Claspin and mus101, whereas Claspin, but not mus101, plays a major role during elongation.Systematic epistatic mapping of cellular processes.Proliferation of Double-Strand Break-Resistant Polyploid Cells Requires Drosophila FANCD2.Replication fork progression during re-replication requires the DNA damage checkpoint and double-strand break repair.GFZF, a glutathione S-transferase protein implicated in cell cycle regulation and hybrid inviability, is a transcriptional co-activator.Drosophila splicing factor SF2 knock-down mutant shows altered cell-cycle in vivo.
P2860
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P2860
A genome-wide RNAi screen identifies core components of the G₂-M DNA damage checkpoint.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年學術文章
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name
A genome-wide RNAi screen identifies core components of the G₂-M DNA damage checkpoint.
@ast
A genome-wide RNAi screen identifies core components of the G₂-M DNA damage checkpoint.
@en
type
label
A genome-wide RNAi screen identifies core components of the G₂-M DNA damage checkpoint.
@ast
A genome-wide RNAi screen identifies core components of the G₂-M DNA damage checkpoint.
@en
prefLabel
A genome-wide RNAi screen identifies core components of the G₂-M DNA damage checkpoint.
@ast
A genome-wide RNAi screen identifies core components of the G₂-M DNA damage checkpoint.
@en
P2860
P1433
P1476
A genome-wide RNAi screen identifies core components of the G₂-M DNA damage checkpoint.
@en
P2860
P356
10.1126/SCISIGNAL.2001350
P577
2011-01-04T00:00:00Z