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Vascular calcium channels and high blood pressure: pathophysiology and therapeutic implications.

Vascular calcium channels and high blood pressure: pathophysiology and therapeutic implications.

http://www.wikidata.org/entity/Q36374384

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Main ion channels and receptors associated with visceral hypersensitivity in irritable bowel syndrome Ion channel remodeling in vascular smooth muscle during hypertension: Implications for novel therapeutic approaches Inhibition of ENaC by endothelin-1 A genetic screen for dihydropyridine (DHP)-resistant worms reveals new residues required for DHP-blockage of mammalian calcium channels Heart of the matter: coronary dysfunction in metabolic syndrome.Failure of Bay K 8644 to induce RhoA kinase-dependent calcium sensitization in rabbit blood vessels.Eugenol dilates rat cerebral arteries by inhibiting smooth muscle cell voltage-dependent calcium channels.Ca(V)1.2 channel N-terminal splice variants modulate functional surface expression in resistance size artery smooth muscle cells Antihypertension Induced by Tanshinone IIA Isolated from the Roots of Salvia miltiorrhiza.Chronic exercise normalizes changes in Cav 1.2 and KCa 1.1 channels in mesenteric arteries from spontaneously hypertensive rats.The COP9 signalosome and vascular function: intriguing possibilities?Regulation of myogenic tone and structure of parenchymal arterioles by hypertension and the mineralocorticoid receptor.Increased arterial smooth muscle Ca2+ signaling, vasoconstriction, and myogenic reactivity in Milan hypertensive rats Contribution of transient and sustained calcium influx, and sensitization to depolarization-induced contractions of the intact mouse aorta A novel Ca(V)1.2 N terminus expressed in smooth muscle cells of resistance size arteries modifies channel regulation by auxiliary subunits.Local regulation of L-type Ca²⁺ channel sparklets in arterial smooth muscle An elevation in physical coupling of type 1 inositol 1,4,5-trisphosphate (IP3) receptors to transient receptor potential 3 (TRPC3) channels constricts mesenteric arteries in genetic hypertension Calcium dynamics and signaling in vascular regulation: computational models.Disrupting calcium channel expression to lower blood pressure: new targeting of a well-known channel Rab25 influences functional Cav1.2 channel surface expression in arterial smooth muscle cells.The β3 subunit contributes to vascular calcium channel upregulation and hypertension in angiotensin II-infused C57BL/6 mice.Computational methods of studying the binding of toxins from venomous animals to biological ion channels: theory and applications.Alternative splicing of Cav1.2 channel exons in smooth muscle cells of resistance-size arteries generates currents with unique electrophysiological properties.Vasodilator efficacy of nitric oxide depends on mechanisms of intracellular calcium mobilization in mouse aortic smooth muscle cells.Molecular basis for zinc transporter 1 action as an endogenous inhibitor of L-type calcium channels.Increased constrictor tone induced by ouabain treatment in rats.L-type calcium channels and psychiatric disorders: A brief review.1,4-dihydropyridines: the multiple personalities of a blockbuster drug family.Delivery of ion channel genes to treat cardiovascular diseases.Nonsteroidal anti-inflammatory drugs inhibit vascular smooth muscle cell proliferation by enabling the Ca2+-dependent inactivation of calcium release-activated calcium/orai channels normally prevented by mitochondria.Sustained Contraction in Vascular Smooth Muscle by Activation of L-type Ca2+ Channels Does Not Involve Ca2+ Sensitization or Caldesmon Transcriptional upregulation of α2δ-1 elevates arterial smooth muscle cell voltage-dependent Ca2+ channel surface expression and cerebrovascular constriction in genetic hypertension.17β-estradiol reduces Cav 1.2 channel abundance and attenuates Ca2+ -dependent contractions in coronary arteries Smooth muscle cell alpha2delta-1 subunits are essential for vasoregulation by CaV1.2 channels.Down-regulation of CaV1.2 channels during hypertension: how fewer CaV1.2 channels allow more Ca(2+) into hypertensive arterial smooth muscle.Increased sensitivity to diltiazem hypotensive effect in an experimental model of high-renin hypertension.Vasodilator and antihypertensive effects of a novel N-acylhydrazone derivative mediated by the inhibition of L-type Ca²⁺ channels.Calcium-dependent and calcium-sensitizing pathways in the mature and immature ductus arteriosus.P/Q-type and T-type voltage-gated calcium channels are involved in the contraction of mammary and brain blood vessels from hypertensive patients.The voltage-dependent L-type Ca2+ (CaV1.2) channel C-terminus fragment is a bi-modal vasodilator.

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P2860

Vascular calcium channels and high blood pressure: pathophysiology and therapeutic implications.

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Vascular Pharmacology

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Philip T Palade

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P2860

Molecular determinants of inactivation within the I-II linker of alpha1E (CaV2.3) calcium channels.

Regulation of voltage-gated calcium channel activity by the Rem and Rad GTPases

Ca2+-induced inhibition of the cardiac Ca2+ channel depends on calmodulin

Absence of the beta subunit (cchb1) of the skeletal muscle dihydropyridine receptor alters expression of the alpha 1 subunit and eliminates excitation-contraction coupling

Functional regulation of L-type calcium channels via protein kinase A-mediated phosphorylation of the beta(2) subunit

Structure and regulation of voltage-gated Ca2+ channels

Molecular heterogeneity of calcium channel beta-subunits in canine and human heart: evidence for differential subcellular localization

Structural basis of the alpha1-beta subunit interaction of voltage-gated Ca2+ channels

New short splice variants of the human cardiac Cavbeta2 subunit: redefining the major functional motifs implemented in modulation of the Cav1.2 channel

A family of gamma-like calcium channel subunits

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pathophysiology

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4917380

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