Transcriptional upregulation of α2δ-1 elevates arterial smooth muscle cell voltage-dependent Ca2+ channel surface expression and cerebrovascular constriction in genetic hypertension.
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Enhanced parenchymal arteriole tone and astrocyte signaling protect neurovascular coupling mediated parenchymal arteriole vasodilation in the spontaneously hypertensive rat.Expression of microRNAs is essential for arterial myogenic tone and pressure-induced activation of the PI3-kinase/Akt pathway.Expression of Calcium Channel Subunit Variants in Small Mesenteric Arteries of WKY and SHRIdentification of Glycosylation Sites Essential for Surface Expression of the CaVα2δ1 Subunit and Modulation of the Cardiac CaV1.2 Channel Activity.An elevation in physical coupling of type 1 inositol 1,4,5-trisphosphate (IP3) receptors to transient receptor potential 3 (TRPC3) channels constricts mesenteric arteries in genetic hypertensionT-type Ca²⁺ channels in cerebral arteries: approaches, hypotheses, and speculation.Proteolytic cleavage of the hydrophobic domain in the CaVα2δ1 subunit improves assembly and activity of cardiac CaV1.2 channels.Angiotensin II reduces the surface abundance of KV 1.5 channels in arterial myocytes to stimulate vasoconstriction.Calcium Channels in Vascular Smooth Muscle.Smooth Muscle Ion Channels and Regulation of Vascular Tone in Resistance Arteries and Arterioles.Intravascular pressure enhances the abundance of functional Kv1.5 channels at the surface of arterial smooth muscle cells.Negatively charged residues in the first extracellular loop of the L-type CaV1.2 channel anchor the interaction with the CaVα2δ1 auxiliary subunit.Adding accessories for hypertension: α2δ-1 subunits upregulate CaV1.2 channels in arterial myocytes in a model of genetic hypertension.Down-regulation of CaV1.2 channels during hypertension: how fewer CaV1.2 channels allow more Ca(2+) into hypertensive arterial smooth muscle.Alternative Splicing of L-type CaV1.2 Calcium Channels: Implications in Cardiovascular Diseases.Membrane depolarization activates BK channels through ROCK-mediated β1 subunit surface trafficking to limit vasoconstriction.Pregabalin and heart failure: A population-based study.Impaired P2X signalling pathways in renal microvascular myocytes in genetic hypertension.The voltage-dependent L-type Ca2+ (CaV1.2) channel C-terminus fragment is a bi-modal vasodilator.Mutations in voltage-gated L-type calcium channel: implications in cardiac arrhythmia
P2860
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P2860
Transcriptional upregulation of α2δ-1 elevates arterial smooth muscle cell voltage-dependent Ca2+ channel surface expression and cerebrovascular constriction in genetic hypertension.
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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name
Transcriptional upregulation o ...... ction in genetic hypertension.
@en
Transcriptional upregulation o ...... ction in genetic hypertension.
@nl
type
label
Transcriptional upregulation o ...... ction in genetic hypertension.
@en
Transcriptional upregulation o ...... ction in genetic hypertension.
@nl
prefLabel
Transcriptional upregulation o ...... ction in genetic hypertension.
@en
Transcriptional upregulation o ...... ction in genetic hypertension.
@nl
P2093
P2860
P1433
P1476
Transcriptional upregulation o ...... ction in genetic hypertension.
@en
P2093
Candice Thomas-Gatewood
Damodaran Narayanan
John P Bannister
Jonathan H Jaggar
Judith Pachuau
Patrik Luzny
Simon Bulley
P2860
P304
P356
10.1161/HYPERTENSIONAHA.112.199661
P407
P577
2012-09-04T00:00:00Z