Monocyte chemoattractant protein 1-dependent leukocytic infiltrates are responsible for autoimmune disease in MRL-Fas(lpr) mice. - Wikidata - wikimedia - TriplyDB

Monocyte chemoattractant protein 1-dependent leukocytic infiltrates are responsible for autoimmune disease in MRL-Fas(lpr) mice.

Monocyte chemoattractant protein 1-dependent leukocytic infiltrates are responsible for autoimmune disease in MRL-Fas(lpr) mice.

http://www.wikidata.org/entity/Q36375621

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Enhanced expression of interferon-inducible protein 10 associated with Th1 profiles of chemokine receptor in autoimmune pulmonary inflammation of MRL/lpr mice Chemokines and Chemokine Receptors in the Development of Lupus Nephritis Mechanisms of Kidney Injury in Lupus Nephritis - the Role of Anti-dsDNA Antibodies Lupus nephritis: the evolving role of novel therapeutics The renal mononuclear phagocytic system The Link between Autoimmunity and Lymphoma: Does NOTCH Signaling Play a Contributing Role?Wound healing in MIP-1alpha(-/-) and MCP-1(-/-) mice Mechanisms of tissue injury in lupus nephritis Defects in the peripheral taste structure and function in the MRL/lpr mouse model of autoimmune disease Absence of monocyte chemoattractant protein 1 in mice leads to decreased local macrophage recruitment and antigen-specific T helper cell type 1 immune response in experimental autoimmune encephalomyelitis Anti-alpha8 integrin immunoliposomes in glomeruli of lupus-susceptible mice: a novel system for delivery of therapeutic agents to the renal glomerulus in systemic lupus erythematosus.Human lupus autoantibody-DNA complexes activate DCs through cooperation of CD32 and TLR9.Recent advances in the understanding of renal inflammation and fibrosis in lupus nephritis.Type I interferons produced by resident renal cells may promote end-organ disease in autoantibody-mediated glomerulonephritis.Conditional up-regulation of MHC class I in skeletal muscle leads to self-sustaining autoimmune myositis and myositis-specific autoantibodies Genetics of systemic autoimmunity in mouse models of lupus.Immune enhancement by novel vaccine adjuvants in autoimmune-prone NZB/W F1 mice: relative efficacy and safety.Targeted tumor necrosis factor receptor I preligand assembly domain improves skin lesions in MRL/lpr mice.Lupus serum IgG induces skin inflammation through the TNFR1 signaling pathway Etiopathology of chronic tubular, glomerular and renovascular nephropathies: clinical implications Fli-1 controls transcription from the MCP-1 gene promoter, which may provide a novel mechanism for chemokine and cytokine activation IL-15, a survival factor for kidney epithelial cells, counteracts apoptosis and inflammation during nephritis.Lowering anti-dsDNA antibodies--what's new?A small-molecule macrophage migration inhibitory factor antagonist protects against glomerulonephritis in lupus-prone NZB/NZW F1 and MRL/lpr mice.Conditional ablation of macrophages halts progression of crescentic glomerulonephritis.Treatment of murine lupus with cDNA encoding IFN-gammaR/Fc.Alterations in leucocyte trafficking in lupus-prone mice: an examination of the MRL/faslpr mouse.Distinct roles of CSF-1 isoforms in lupus nephritis N-Acetyl-Seryl-Aspartyl-Lysyl-Proline: mechanisms of renal protection in mouse model of systemic lupus erythematosus HMGB1 Promotes Systemic Lupus Erythematosus by Enhancing Macrophage Inflammatory Response.Tumor-specific T cells signal tumor destruction via the lymphotoxin beta receptor Biomarkers for kidney involvement in pediatric lupus.Identifying chemokines as therapeutic targets in renal disease: lessons from antagonist studies and knockout mice.Current paradigms about chemokines as therapeutic targets.Aberrant macrophages mediate defective kidney repair that triggers nephritis in lupus-susceptible mice Beneficial effect of Bupleurum polysaccharides on autoimmune-prone MRL-lpr mice.The Pathogenesis and Therapeutic Implications of Tubulointerstitial Inflammation in Human Lupus Nephritis Mesangial cells of lupus-prone mice are sensitive to chemokine production Fli-1 transcription factor affects glomerulonephritis development by regulating expression of monocyte chemoattractant protein-1 in endothelial cells in the kidney.Treatment targets in systemic lupus erythematosus: biology and clinical perspective.

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Monocyte chemoattractant protein 1-dependent leukocytic infiltrates are responsible for autoimmune disease in MRL-Fas(lpr) mice.

http://www.wikidata.org/entity/Q36375621

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1999 nî lūn-bûn

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Monocyte chemoattractant prote ...... disease in MRL-Fas(lpr) mice.

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A Schwarting

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B J Rollins

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G H Tesch

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V R Kelley

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P2860

Distinct expression and function of the novel mouse chemokine monocyte chemotactic protein-5 in lung allergic inflammation

Monocyte chemoattractant protein 1 acts as a T-lymphocyte chemoattractant

RANTES and monocyte chemoattractant protein-1 (MCP-1) play an important role in the inflammatory phase of crescentic nephritis, but only MCP-1 is involved in crescent formation and interstitial fibrosis

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

IgG3 cryoglobulins in autoimmune MRL-lpr/lpr mice: immunopathogenesis, therapeutic approaches and relevance to similar human diseases.

Monocyte chemoattractant protein-1 promotes macrophage-mediated tubular injury, but not glomerular injury, in nephrotoxic serum nephritis

Production of monocyte chemoattractant protein-1 and macrophage inflammatory protein-1 alpha by inflammatory granuloma fibroblasts.

Autoimmune disease in mice due to integration of an endogenous retrovirus in an apoptosis gene.

An antagonist of monocyte chemoattractant protein 1 (MCP-1) inhibits arthritis in the MRL-lpr mouse model

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10601356

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