High Glucose-Repressed CITED2 Expression Through miR-200b Triggers the Unfolded Protein Response and Endoplasmic Reticulum Stress
about
High glucose suppresses embryonic stem cell differentiation into cardiomyocytes : High glucose inhibits ES cell cardiogenesisMiR-17 Downregulation by High Glucose Stabilizes Thioredoxin-Interacting Protein and Removes Thioredoxin Inhibition on ASK1 Leading to Apoptosis.High glucose-induced oxidative stress represses sirtuin deacetylase expression and increases histone acetylation leading to neural tube defectsSuperoxide dismutase 2 overexpression alleviates maternal diabetes-induced neural tube defects, restores mitochondrial function and suppresses cellular stress in diabetic embryopathy.Type 2 diabetes mellitus induces congenital heart defects in murine embryos by increasing oxidative stress, endoplasmic reticulum stress, and apoptosis.The green tea polyphenol EGCG alleviates maternal diabetes-induced neural tube defects by inhibiting DNA hypermethylationmicroRNA expression profiling and functional annotation analysis of their targets modulated by oxidative stress during embryonic heart development in diabetic mice.High glucose suppresses embryonic stem cell differentiation into neural lineage cellsThe Nrf2 Activator Vinylsulfone Reduces High Glucose-Induced Neural Tube Defects by Suppressing Cellular Stress and Apoptosis.The current status and future of cardiac stem/progenitor cell therapy for congenital heart defects from diabetic pregnancy.Endoplasmic reticulum stress stimulates the release of extracellular vesicles carrying danger-associated molecular pattern (DAMP) molecules.Endoplasmic Reticulum Stress-Induced CHOP Inhibits PGC-1α and Causes Mitochondrial Dysfunction in Diabetic Embryopathy.Cellular stress and apoptosis contribute to the pathogenesis of autism spectrum disorder.
P2860
Q30831503-7A954057-8EB2-4CEE-92DF-93FF4EC2D6F1Q36794190-922AFCE4-2FD0-4BE7-A4CB-554D7BD6B9EAQ36813699-DA36141D-83CE-4C7E-B13C-5BA1C874146DQ37017347-2EDB8E93-6D84-40D4-B7F4-E8545CC84287Q37603119-F0CFA712-A5AC-4D9B-9610-69525CEDD30AQ37610042-4C375F3B-084F-432D-A721-8D335FA1138BQ37620057-3A17F5B2-8833-4558-BA79-B20C2259320CQ38789070-0B5BD2E4-B137-49A5-A56F-348F42010A39Q40070862-028D187E-A6C0-4272-804D-4A633BA69209Q46302230-9887930E-E1E0-4293-BCDC-CD23D097119BQ50199385-7796F9A4-5EFC-4B96-BBCB-9EC7ED3549EEQ51015129-28B9C8D1-7092-48EA-A5CE-8060F31A643FQ54109826-731F2A26-3EE8-4D1A-B128-C3B74A9E00E4
P2860
High Glucose-Repressed CITED2 Expression Through miR-200b Triggers the Unfolded Protein Response and Endoplasmic Reticulum Stress
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年学术文章
@wuu
2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
@zh
2015年學術文章
@zh-hant
name
High Glucose-Repressed CITED2 ...... d Endoplasmic Reticulum Stress
@ast
High Glucose-Repressed CITED2 ...... d Endoplasmic Reticulum Stress
@en
type
label
High Glucose-Repressed CITED2 ...... d Endoplasmic Reticulum Stress
@ast
High Glucose-Repressed CITED2 ...... d Endoplasmic Reticulum Stress
@en
prefLabel
High Glucose-Repressed CITED2 ...... d Endoplasmic Reticulum Stress
@ast
High Glucose-Repressed CITED2 ...... d Endoplasmic Reticulum Stress
@en
P2093
P2860
P356
P1433
P1476
High Glucose-Repressed CITED2 ...... d Endoplasmic Reticulum Stress
@en
P2093
Jian-Ying Wang
Jingwen Yu
Peixin Yang
Shengyun Fang
P2860
P304
P356
10.2337/DB15-0108
P407
P50
P577
2015-10-08T00:00:00Z