TNF-α triggers rapid membrane insertion of Ca(2+) permeable AMPA receptors into adult motor neurons and enhances their susceptibility to slow excitotoxic injury.
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Tumor necrosis factor alpha: a link between neuroinflammation and excitotoxicityDifferent occupations associated with amyotrophic lateral sclerosis: is diesel exhaust the link?Cytokine profiling in patients with VCP-associated disease.Differential distribution of PI3K isoforms in spinal cord and dorsal root ganglia: potential roles in acute inflammatory painPeripheral noxious stimulation reduces withdrawal threshold to mechanical stimuli after spinal cord injury: role of tumor necrosis factor alpha and apoptosis.AMPA Receptor Phosphorylation and Synaptic Colocalization on Motor Neurons Drive Maladaptive Plasticity below Complete Spinal Cord Injury.Does brain inflammation mediate pathological outcomes in epilepsy?Managing inflammation after spinal cord injury through manipulation of macrophage functionInactivity-induced respiratory plasticity: protecting the drive to breathe in disorders that reduce respiratory neural activityCytokine cascades induced by mechanical trauma injury alter voltage-gated sodium channel activity in intact cortical neurons.Calcium dysregulation and neuroinflammation: discrete and integrated mechanisms for age-related synaptic dysfunction.Organotypic Spinal Cord Culture: a Proper Platform for the Functional Screening.Inflammation-induced GluA1 trafficking and membrane insertion of Ca2+ permeable AMPA receptors in dorsal horn neurons is dependent on spinal tumor necrosis factor, PI3 kinase and protein kinase A.What Is Being Trained? How Divergent Forms of Plasticity Compete To Shape Locomotor Recovery after Spinal Cord Injury.Neurobiological Effects of Morphine after Spinal Cord Injury.An unexpected effect of TNF-α on F508del-CFTR maturation and function.Amyotrophic Lateral Sclerosis, a Multisystem Pathology: Insights into the Role of TNFα.Atorvastatin prevents ATP-driven invasiveness via P2X7 and EHBP1 signaling in PTEN-expressing prostate cancer cells.Spinal TNF is necessary for inactivity-induced phrenic motor facilitation.Disruption of calcitonin gene-related peptide signaling accelerates muscle denervation and dampens cytotoxic neuroinflammation in SOD1 mutant mice.
P2860
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P2860
TNF-α triggers rapid membrane insertion of Ca(2+) permeable AMPA receptors into adult motor neurons and enhances their susceptibility to slow excitotoxic injury.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
TNF-α triggers rapid membrane ...... ty to slow excitotoxic injury.
@ast
TNF-α triggers rapid membrane ...... ty to slow excitotoxic injury.
@en
type
label
TNF-α triggers rapid membrane ...... ty to slow excitotoxic injury.
@ast
TNF-α triggers rapid membrane ...... ty to slow excitotoxic injury.
@en
prefLabel
TNF-α triggers rapid membrane ...... ty to slow excitotoxic injury.
@ast
TNF-α triggers rapid membrane ...... ty to slow excitotoxic injury.
@en
P2093
P2860
P1476
TNF-α triggers rapid membrane ...... ty to slow excitotoxic injury.
@en
P2093
Cheng-I Hsu
Hong Z Yin
John H Weiss
Linda S Sorkin
Shyam D Rao
Stephen Yu
P2860
P304
P356
10.1016/J.EXPNEUROL.2012.08.004
P407
P577
2012-08-19T00:00:00Z