The cellular ataxia telangiectasia-mutated kinase promotes epstein-barr virus lytic reactivation in response to multiple different types of lytic reactivation-inducing stimuli - Wikidata - wikimedia - TriplyDB

The cellular ataxia telangiectasia-mutated kinase promotes epstein-barr virus lytic reactivation in response to multiple different types of lytic reactivation-inducing stimuli

The cellular ataxia telangiectasia-mutated kinase promotes epstein-barr virus lytic reactivation in response to multiple different types of lytic reactivation-inducing stimuli

http://www.wikidata.org/entity/Q36414572

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Modulation of DNA damage and repair pathways by human tumour viruses Regulation of the latent-lytic switch in Epstein-Barr virus Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesis Reactive oxygen species mediate Epstein-Barr virus reactivation by N-methyl-N'-nitro-N-nitrosoguanidine Identification of Novel Small Organic Compounds with Diverse Structures for the Induction of Epstein-Barr Virus (EBV) Lytic Cycle in EBV-Positive Epithelial Malignancies Activation and repression of Epstein-Barr Virus and Kaposi's sarcoma-associated herpesvirus lytic cycles by short- and medium-chain fatty acids A locus encompassing the Epstein-Barr virus bglf4 kinase regulates expression of genes encoding viral structural proteins DNA Damage Signaling Is Induced in the Absence of Epstein-Barr Virus (EBV) Lytic DNA Replication and in Response to Expression of ZEBRA.Targeted therapy for Epstein-Barr virus-associated gastric carcinoma using low-dose gemcitabine-induced lytic activation.LMP1-Induced Sumoylation Influences the Maintenance of Epstein-Barr Virus Latency through KAP1 ATM facilitates mouse gammaherpesvirus reactivation from myeloid cells during chronic infection.Oncogenic Herpesvirus Utilizes Stress-Induced Cell Cycle Checkpoints for Efficient Lytic Replication Chloroquine triggers Epstein-Barr virus replication through phosphorylation of KAP1/TRIM28 in Burkitt lymphoma cells Murine Gammaherpesvirus 68 LANA and SOX Homologs Counteract ATM-Driven p53 Activity during Lytic Viral Replication Human papillomaviruses: research priorities for the next decade.Epstein-Barr virus induces global changes in cellular mRNA isoform usage that are important for the maintenance of latency.Potential of protein kinase inhibitors for treating herpesvirus-associated disease.Tumor Suppressor p53 Stimulates the Expression of Epstein-Barr Virus Latent Membrane Protein 1.H2AX phosphorylation and DNA damage kinase activity are dispensable for herpes simplex virus replication.Inhibition of class I histone deacetylases by romidepsin potently induces Epstein-Barr virus lytic cycle and mediates enhanced cell death with ganciclovir.Activation of DNA Damage Response Pathways during Lytic Replication of KSHV.Role of ATM in the formation of the replication compartment during lytic replication of Epstein-Barr virus in nasopharyngeal epithelial cells.EBV-related lymphomas: new approaches to treatment.Take your PIKK: tumour viruses and DNA damage response pathways.ATM supports gammaherpesvirus replication by attenuating type I interferon pathway.The Translesion Polymerase Pol η Is Required for Efficient Epstein-Barr Virus Infectivity and Is Regulated by the Viral Deubiquitinating Enzyme BPLF1.Small molecule perturbation of the CAND1-Cullin1-ubiquitin cycle stabilizes p53 and triggers Epstein-Barr virus reactivation.B Cell-Specific Expression of Ataxia-Telangiectasia Mutated Protein Kinase Promotes Chronic Gammaherpesvirus Infection.Epstein-Barr Virus Rta-Mediated Accumulation of DNA Methylation Interferes with CTCF Binding in both Host and Viral Genomes.The Rad6/18 ubiquitin complex interacts with the Epstein-Barr virus deubiquitinating enzyme, BPLF1, and contributes to virus infectivity.Epstein-Barr Virus Hijacks DNA Damage Response Transducers to Orchestrate Its Life Cycle.EBV-encoded miRNAs target ATM-mediated response in nasopharyngeal carcinoma.A Screen for Epstein-Barr Virus Proteins that Inhibit the DNA Damage Response Reveals a Novel Histone Binding Protein.Investigating genetic-and-epigenetic networks, and the cellular mechanisms occurring in Epstein-Barr virus-infected human B lymphocytes via big data mining and genome-wide two-sided NGS data identification

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P2860

The cellular ataxia telangiectasia-mutated kinase promotes epstein-barr virus lytic reactivation in response to multiple different types of lytic reactivation-inducing stimuli

http://www.wikidata.org/entity/Q36414572

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

@zh-hans

2012年学术文章

@zh-my

2012年学术文章

@zh-sg

2012年學術文章

@yue

2012年學術文章

@zh

2012年學術文章

@zh-hant

name

The cellular ataxia telangiect ...... reactivation-inducing stimuli

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The cellular ataxia telangiect ...... reactivation-inducing stimuli

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type

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The cellular ataxia telangiect ...... reactivation-inducing stimuli

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The cellular ataxia telangiect ...... reactivation-inducing stimuli

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prefLabel

The cellular ataxia telangiect ...... reactivation-inducing stimuli

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The cellular ataxia telangiect ...... reactivation-inducing stimuli

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P1433

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P1433

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P1476

The cellular ataxia telangiect ...... reactivation-inducing stimuli

@en

P2093

Elizabeth A Barlow

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Qiao Meng

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Shannon C Kenney

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Stacy R Hagemeier

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P2860

A role for ATR in the DNA damage-induced phosphorylation of p53

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage

TORC2, a coactivator of cAMP-response element-binding protein, promotes Epstein-Barr virus reactivation from latency through interaction with viral BZLF1 protein

Myc sensitizes p53-deficient cancer cells to the DNA-damaging effects of the DNA methyltransferase inhibitor decitabine

Epstein-barr virus immediate-early protein BZLF1 is SUMO-1 modified and disrupts promyelocytic leukemia bodies

Multiple roles of ATM in monitoring and maintaining DNA integrity

Targeting histone deacetylase complexes via KRAB-zinc finger proteins: the PHD and bromodomains of KAP-1 form a cooperative unit that recruits a novel isoform of the Mi-2alpha subunit of NuRD

In vivo activation of the p53 pathway by small-molecule antagonists of MDM2

DNA damage-induced phosphorylation of p53 alleviates inhibition by MDM2

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13360-13370

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scholarly article

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10.1128/JVI.01850-12

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24

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86

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2012-09-26T00:00:00Z

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231215327

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P698

23015717

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P921

Epstein–Barr virus

P932

3503132

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