T cell expression of MyD88 is required for resistance to Toxoplasma gondii. - Wikidata - wikimedia - TriplyDB

T cell expression of MyD88 is required for resistance to Toxoplasma gondii.

T cell expression of MyD88 is required for resistance to Toxoplasma gondii.

http://www.wikidata.org/entity/Q36499083

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Recombinant Toxoplasma gondii phosphoglycerate mutase 2 confers protective immunity against toxoplasmosis in BALB/c mice Interferon-gamma- and perforin-mediated immune responses for resistance against Toxoplasma gondii in the brain Communication between Toxoplasma gondii and its host: impact on parasite growth, development, immune evasion, and virulence.MyD88 plays a critical T cell-intrinsic role in supporting CD8 T cell expansion during acute lymphocytic choriomeningitis virus infection.Critical role of IRF-5 in the development of T helper 1 responses to Leishmania donovani infection.Protective Toxoplasma gondii-specific T-cell responses require T-cell-specific expression of protein kinase C-theta.Educating CD4 T cells with vaccine adjuvants: lessons from lipopolysaccharide.When Toll-like receptor and T-cell receptor signals collide: a mechanism for enhanced CD8 T-cell effector function.Critical coordination of innate immune defense against Toxoplasma gondii by dendritic cells responding via their Toll-like receptors MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resol Vaccine-induced protection against 3 systemic mycoses endemic to North America requires Th17 cells in mice.Innate responses to Toxoplasma gondii in mice and humans Inhibition of IRAK1/4 sensitizes T cell acute lymphoblastic leukemia to chemotherapies.Toxoplasma gondii effectors are master regulators of the inflammatory response.Intrinsic MyD88-Akt1-mTOR Signaling Coordinates Disparate Tc17 and Tc1 Responses during Vaccine Immunity against Fungal Pneumonia.Toxoplasma on the brain: understanding host-pathogen interactions in chronic CNS infection.TLR9 and MyD88 are crucial for the development of protective immunity to malaria.Vaccination with Toxoplasma gondii calcium-dependent protein kinase 6 and rhoptry protein 18 encapsulated in poly(lactide-co-glycolide) microspheres induces long-term protective immunity in mice.MyD88 Shapes Vaccine Immunity by Extrinsically Regulating Survival of CD4+ T Cells during the Contraction Phase T Cell Production of IFNγ in Response to TLR7/IL-12 Stimulates Optimal B Cell Responses to Viruses.Effects of Toll-like receptor signals in T-cell neoplasms.Immune-mediated regression of established B16F10 melanoma by intratumoral injection of attenuated Toxoplasma gondii protects against rechallenge.MyD88 is essential to sustain mTOR activation necessary to promote T helper 17 cell proliferation by linking IL-1 and IL-23 signaling Plasticity within the αβ⁺CD4⁺ T-cell lineage: when, how and what for?TLR agonists: our best frenemy in cancer immunotherapy TLR-independent neutrophil-derived IFN-γ is important for host resistance to intracellular pathogens.Caspase-11 Modulates Inflammation and Attenuates Toxoplasma gondii Pathogenesis Antigen-free adjuvant assists late effector CD4 T cells to transit to memory in lymphopenic hosts.TLR adaptor MyD88 is essential for pathogen control during oral toxoplasma gondii infection but not adaptive immunity induced by a vaccine strain of the parasite.MyD88 intrinsically regulates CD4 T-cell responses Inflammasome-derived IL-1β regulates the production of GM-CSF by CD4(+) T cells and γδ T cells An ENU-induced splicing mutation reveals a role for Unc93b1 in early immune cell activation following influenza A H1N1 infection.On the hunt for helminths: innate immune cells in the recognition and response to helminth parasites.CD4+ T cell expression of MyD88 is essential for normal resolution of Chlamydia muridarum genital tract infection.Functional aspects of Toll-like receptor/MyD88 signalling during protozoan infection: focus on Toxoplasma gondii.Toll-like receptor initiated host defense against Toxoplasma gondii.Dual role for inflammasome sensors NLRP1 and NLRP3 in murine resistance to Toxoplasma gondii.Complex immune cell interplay in the gamma interferon response during Toxoplasma gondii infection Do you see what I see: Recognition of protozoan parasites by Toll-like receptors.Dendritic cell specific targeting of MyD88 signalling pathways in vivo.

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P2860

T cell expression of MyD88 is required for resistance to Toxoplasma gondii.

http://www.wikidata.org/entity/Q36499083

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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2008年學術文章

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name

T cell expression of MyD88 is required for resistance to Toxoplasma gondii.

@ast

T cell expression of MyD88 is required for resistance to Toxoplasma gondii.

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type

label

T cell expression of MyD88 is required for resistance to Toxoplasma gondii.

@ast

T cell expression of MyD88 is required for resistance to Toxoplasma gondii.

@en

prefLabel

T cell expression of MyD88 is required for resistance to Toxoplasma gondii.

@ast

T cell expression of MyD88 is required for resistance to Toxoplasma gondii.

@en

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PNAS.0706663105

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Proceedings of the National Academy of Sciences of the United States of America

P1476

T cell expression of MyD88 is required for resistance to Toxoplasma gondii.

@en

P2093

Adeeb H Rahman

http://www.w3.org/2001/XMLSchema#string

Andrew E Gelman

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Christopher A Hunter

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David F LaRosa

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Devon K Taylor

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Jason S Stumhofer

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Laurence A Turka

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P2860

Toll-like receptor 8-mediated reversal of CD4+ regulatory T cell function

Toll-like receptor signalling

Evidence for licensing of IFN-gamma-induced IFN regulatory factor 1 transcription factor by MyD88 in Toll-like receptor-dependent gene induction program

Interleukin (IL)-1F6, IL-1F8, and IL-1F9 signal through IL-1Rrp2 and IL-1RAcP to activate the pathway leading to NF-kappaB and MAPKs

The interleukin-1 receptor family

Integral role of IRF-5 in the gene induction programme activated by Toll-like receptors

Toll-like receptor control of the adaptive immune responses

The family of five: TIR-domain-containing adaptors in Toll-like receptor signalling

The adaptor molecule MyD88 activates PI-3 kinase signaling in CD4+ T cells and enables CpG oligodeoxynucleotide-mediated costimulation.

The CD40/CD40 ligand interaction is required for resistance to toxoplasmic encephalitis.

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3855-3860

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scholarly article

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10.1073/PNAS.0706663105

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10

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105

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2008-02-28T00:00:00Z

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5543354

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18308927

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Toxoplasma gondii

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2268781

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