Mechanisms of urinary K+ and H+ excretion: primary structure and functional expression of a novel H,K-ATPase
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Nongastric H-K-ATPase in rodent prostate: lobe-specific expression and apical localizationCloning and characterization of the entire cDNA encoded by ATP1AL1--a member of the human Na,K/H,K-ATPase gene familyStimulation of protein kinase C pathway mediates endocytosis of human nongastric H+-K+-ATPase, ATP1AL1.Pharmacological profiles of the murine gastric and colonic H,K-ATPases.Functional expression and segmental localization of rat colonic K-adenosine triphosphataseElectrogenic sulfate/chloride exchange in Xenopus oocytes mediated by murine AE1 E699Q.Airway acidification initiates host defense abnormalities in cystic fibrosis mice.Does the colonic H,K-ATPase also act as an Na,K-ATPase?Interaction of alpha- and beta-subunits in native H-K-ATPase and cultured cells transfected with H-K-ATPase beta-subunit.Mutational analysis of putative SCH 28080 binding sites of the gastric H+,K+-ATPase.Proton and potassium transport by H+/K(+)-ATPases.Deceleration of the E1P-E2P transition and ion transport by mutation of potentially salt bridge-forming residues Lys-791 and Glu-820 in gastric H+/K+-ATPase.A transcription activator with restricted tissue distribution regulates cell-specific expression of alpha1(XI) collagen.Bufo marinus bladder H-K-ATPase carries out electroneutral ion transport.Electrogenicity of Na,K- and H,K-ATPase activity and presence of a positively charged amino acid in the fifth transmembrane segment.Functional significance of E2 state stabilization by specific alpha/beta-subunit interactions of Na,K- and H,K-ATPase.Amino acids Val115-Ile126 of rat gastric H(+)-K(+)-ATPase confer high affinity for Sch-28080 to Na(+)-K(+)-ATPase.Functional expression of the colonic H+,K+-ATPase alpha-subunit. Pharmacologic properties and assembly with X+,K+-ATPase beta-subunits.The rat distal colon P-ATPase alpha subunit encodes a ouabain-sensitive H+, K+-ATPase.Human ATP1AL1 gene encodes a ouabain-sensitive H-K-ATPase.Conversion of the low affinity ouabain-binding site of non-gastric H,K-ATPase into a high affinity binding site by substitution of only five amino acids.ATP1AL1, a member of the non-gastric H,K-ATPase family, functions as a sodium pump.Proton Pump Inhibitors and Dementia: Physiopathological Mechanisms and Clinical Consequences.
P2860
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P2860
Mechanisms of urinary K+ and H+ excretion: primary structure and functional expression of a novel H,K-ATPase
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Mechanisms of urinary K+ and H ...... pression of a novel H,K-ATPase
@ast
Mechanisms of urinary K+ and H ...... pression of a novel H,K-ATPase
@en
type
label
Mechanisms of urinary K+ and H ...... pression of a novel H,K-ATPase
@ast
Mechanisms of urinary K+ and H ...... pression of a novel H,K-ATPase
@en
prefLabel
Mechanisms of urinary K+ and H ...... pression of a novel H,K-ATPase
@ast
Mechanisms of urinary K+ and H ...... pression of a novel H,K-ATPase
@en
P2093
P2860
P356
P1476
Mechanisms of urinary K+ and H ...... pression of a novel H,K-ATPase
@en
P2093
B C Rossier
J D Horisberger
P2860
P304
P356
10.1083/JCB.123.6.1421
P407
P433
P577
1993-12-01T00:00:00Z