Transcriptional activation of the Epstein-Barr virus latency C promoter after 5-azacytidine treatment: evidence that demethylation at a single CpG site is crucial.
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A role for SKIP in EBNA2 activation of CBF1-repressed promotersNovel Insights into Insect-Microbe Interactions-Role of Epigenomics and Small RNAsRole of EBNA-3 Family Proteins in EBV Associated B-cell LymphomagenesisSite-selective in vivo targeting of cytosine-5 DNA methylation by zinc-finger proteinsSite-specific methylation of the promoter alters deoxyribonucleic acid-protein interactions and prevents follicle-stimulating hormone receptor gene transcription.The site specific demethylation in the 5'-regulatory area of NMDA receptor 2B subunit gene associated with CIE-induced up-regulation of transcription.Transcriptional activation signals found in the Epstein-Barr virus (EBV) latency C promoter are conserved in the latency C promoter sequences from baboon and Rhesus monkey EBV-like lymphocryptoviruses (cercopithicine herpesviruses 12 and 15).CTCF prevents the epigenetic drift of EBV latency promoter QpA phase I dose-finding study of 5-azacytidine in combination with sodium phenylbutyrate in patients with refractory solid tumorsThe human ARF cell cycle regulatory gene promoter is a CpG island which can be silenced by DNA methylation and down-regulated by wild-type p53.The Epstein-Barr virus major latent promoter Qp is constitutively active, hypomethylated, and methylation sensitiveProtein-DNA binding and CpG methylation at nucleotide resolution of latency-associated promoters Qp, Cp, and LMP1p of Epstein-Barr virusInduction of Epstein-Barr virus kinases to sensitize tumor cells to nucleoside analoguesRNA polymerase II stalling promotes nucleosome occlusion and pTEFb recruitment to drive immortalization by Epstein-Barr virus.The importance of epigenetic alterations in the development of epstein-barr virus-related lymphomas.Activation of latent Kaposi's sarcoma-associated herpesvirus by demethylation of the promoter of the lytic transactivator.The role of promoter methylation in Epstein-Barr virus (EBV) microRNA expression in EBV-infected B cell lines.trans-Repression of protein expression dependent on the Epstein-Barr virus promoter Wp during latency.Epstein-Barr virus in gastric adenocarcinomas: association with ethnicity and CDKN2A promoter methylationMicroRNA 34c gene down-regulation via DNA methylation promotes self-renewal and epithelial-mesenchymal transition in breast tumor-initiating cells.Methylation status of the Epstein-Barr virus major latent promoter C in iatrogenic B cell lymphoproliferative disease. Application of PCR-based analysis.Interferon-independent and -induced regulation of Epstein-Barr virus EBNA-1 gene transcription in Burkitt lymphoma.Hypermethylation of the tumor suppressor gene PRDM1/Blimp-1 supports a pathogenetic role in EBV-positive Burkitt lymphoma.Host-cell-determined methylation of specific Epstein-Barr virus promoters regulates the choice between distinct viral latency programsDaxx represses RelB target promoters via DNA methyltransferase recruitment and DNA hypermethylationA model system to study genomic imprinting of human genes.Identification of Epstein-Barr virus (EBV) nuclear antigen 2 (EBNA2) target proteins by proteome analysis: activation of EBNA2 in conditionally immortalized B cells reflects early events after infection of primary B cells by EBV.Regulation and dysregulation of Epstein-Barr virus latency: implications for the development of autoimmune diseases.Epigenetic regulation of EBV and KSHV latencyCharacterization of the CBF2 binding site within the Epstein-Barr virus latency C promoter and its role in modulating EBNA2-mediated transactivation.Restricted TET2 Expression in Germinal Center Type B Cells Promotes Stringent Epstein-Barr Virus Latency.5' long terminal repeat (LTR)-selective methylation of latently infected HIV-1 provirus that is demethylated by reactivation signals.Effects of chronic variable stress on cognition and Bace1 expression among wild-type miceContributions of CTCF and DNA methyltransferases DNMT1 and DNMT3B to Epstein-Barr virus restricted latency.Specific methylation patterns in two control regions of Epstein-Barr virus latency: the LMP-1-coding upstream regulatory region and an origin of DNA replication (oriP).Methylation of transcription factor binding sites in the Epstein-Barr virus latent cycle promoter Wp coincides with promoter down-regulation during virus-induced B-cell transformation.Epstein-barr virus (EBV) nuclear protein 2-induced disruption of EBV latency in the Burkitt's lymphoma cell line Akata: analysis by tetracycline-regulated expression.Differential methylation of Epstein-Barr virus latency promoters facilitates viral persistence in healthy seropositive individuals.Promoter analysis of mouse Scn3a gene and regulation of the promoter activity by GC box and CpG methylation.Phosphorylation/dephosphorylation of the repressor MDBP-2-H1 selectively affects the level of transcription from a methylated promoter in vitro.
P2860
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P2860
Transcriptional activation of the Epstein-Barr virus latency C promoter after 5-azacytidine treatment: evidence that demethylation at a single CpG site is crucial.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Transcriptional activation of ...... a single CpG site is crucial.
@ast
Transcriptional activation of ...... a single CpG site is crucial.
@en
type
label
Transcriptional activation of ...... a single CpG site is crucial.
@ast
Transcriptional activation of ...... a single CpG site is crucial.
@en
prefLabel
Transcriptional activation of ...... a single CpG site is crucial.
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Transcriptional activation of ...... a single CpG site is crucial.
@en
P2093
P2860
P356
P1476
Transcriptional activation of ...... t a single CpG site is crucial
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P2093
P2860
P304
P356
10.1128/MCB.15.11.6150
P407
P577
1995-11-01T00:00:00Z