Balancing co-stimulation and inhibition with BTLA and HVEM. - Wikidata - wikimedia - TriplyDB

Balancing co-stimulation and inhibition with BTLA and HVEM.

Balancing co-stimulation and inhibition with BTLA and HVEM.

http://www.wikidata.org/entity/Q36577555

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Specific lymphocyte subsets predict response to adoptive cell therapy using expanded autologous tumor-infiltrating lymphocytes in metastatic melanoma patients BTLA mediates inhibition of human tumor-specific CD8+ T cells that can be partially reversed by vaccination Biomarkers for Sepsis: What Is and What Might Be?Autoimmune therapies targeting costimulation and emerging trends in multivalent therapeutics Secreted and transmembrane 1A is a novel co-stimulatory ligand.Activation-induced accumulation of B and T lymphocyte attenuator at the immunological synapse in CD4+ T cells.In situ patrolling of regulatory T cells is essential for protecting autoimmune exocrinopathy.Functional interaction between herpes simplex virus type 2 gD and HVEM transiently dampens local chemokine production after murine mucosal infection T cell intrinsic heterodimeric complexes between HVEM and BTLA determine receptivity to the surrounding microenvironment.B and T lymphocyte attenuator tempers early infection immunity Polymorphic variants of LIGHT (TNF superfamily-14) alter receptor avidity and bioavailability.Herpes simplex virus glycoprotein D interferes with binding of herpesvirus entry mediator to its ligands through downregulation and direct competition BTLA exhibits immune memory for αβ T cells in patients with active pulmonary tuberculosis LIGHT/TNFSR14 can regulate hepatic lipase expression by hepatocytes independent of T cells and Kupffer cells.Soluble PD-1 rescues the proliferative response of simian immunodeficiency virus-specific CD4 and CD8 T cells during chronic infection.Dendritic cell programming by cytomegalovirus stunts naive T cell responses via the PD-L1/PD-1 pathway.A functional polymorphism in B and T lymphocyte attenuator is associated with susceptibility to rheumatoid arthritis.Dichotomous regulation of GVHD through bidirectional functions of the BTLA-HVEM pathway.Mixing Signals: Molecular Turn Ons and Turn Offs for Innate γδ T-Cells.Gene polymorphisms of novel immunotolerant molecule BTLA: distribution of alleles, genotypes and haplotypes in Polish Caucasian population.HVEM gene polymorphisms are associated with sporadic breast cancer in Chinese women.CD8 T cell memory to a viral pathogen requires trans cosignaling between HVEM and BTLA.Cell surface signaling molecules in the control of immune responses: a tide model.Tumor necrosis factor superfamily in innate immunity and inflammation.CD161 DEFINES EFFECTOR T CELLS THAT EXPRESS LIGHT AND RESPOND TO TL1A-DR3 SIGNALING.CD8(+) T cells specific for tumor antigens can be rendered dysfunctional by the tumor microenvironment through upregulation of the inhibitory receptors BTLA and PD-1.Challenges and future perspectives of T cell immunotherapy in cancer Inflammatory Pathways in Parkinson's Disease; A BNE Microarray Study.Herpes virus entry mediator licenses Listeria infection induced immunopathology through control of type I interferon.The role of coinhibitory signaling pathways in transplantation and tolerance BTLA marks a less-differentiated tumor-infiltrating lymphocyte subset in melanoma with enhanced survival properties.Recombinant adeno-associated virus vectors induce functionally impaired transgene product-specific CD8+ T cells in mice.Alternative entry receptors for herpes simplex virus and their roles in disease LIGHT (TNFSF14) Increases the Survival and Proliferation of Human Bone Marrow-Derived Mesenchymal Stem Cells Herpesvirus entry mediator on radiation-resistant cell lineages promotes ocular herpes simplex virus 1 pathogenesis in an entry-independent manner.Immunoregulation by tumor necrosis factor superfamily member LIGHT T-cell inhibitors: a bench-to-bedside review.The canonical and unconventional ligands of the herpesvirus entry mediator Human cytomegalovirus encoded homologs of cytokines, chemokines and their receptors: roles in immunomodulation.Nonalloreactive T cells prevent donor lymphocyte infusion-induced graft-versus-host disease by controlling microbial stimuli.

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P2860

Balancing co-stimulation and inhibition with BTLA and HVEM.

http://www.wikidata.org/entity/Q36577555

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

@zh-mo

2006年論文

@zh-tw

2006年论文

@wuu

2006年论文

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2006年论文

@zh-cn

name

Balancing co-stimulation and inhibition with BTLA and HVEM.

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Balancing co-stimulation and inhibition with BTLA and HVEM.

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Balancing co-stimulation and inhibition with BTLA and HVEM.

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Balancing co-stimulation and inhibition with BTLA and HVEM.

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prefLabel

Balancing co-stimulation and inhibition with BTLA and HVEM.

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Balancing co-stimulation and inhibition with BTLA and HVEM.

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Nature Reviews Immunology

P1476

Balancing co-stimulation and inhibition with BTLA and HVEM.

@en

P2093

Christopher A Nelson

http://www.w3.org/2001/XMLSchema#string

John R Sedý

http://www.w3.org/2001/XMLSchema#string

Kenneth M Murphy

http://www.w3.org/2001/XMLSchema#string

P2860

Reciprocal expression of the TNF family receptor herpes virus entry mediator and its ligand LIGHT on activated T cells: LIGHT down-regulates its own receptor

Constitutive expression of LIGHT on T cells leads to lymphocyte activation, inflammation, and tissue destruction

Inhibition of CTLA-4 function by the regulatory subunit of serine/threonine phosphatase 2A

A TNF family member LIGHT transduces costimulatory signals into human T cells

SHP-1 and SHP-2 associate with immunoreceptor tyrosine-based switch motif of programmed death 1 upon primary human T cell stimulation, but only receptor ligation prevents T cell activation.

Regulation of T lymphocyte metabolism.

The TNF superfamily members LIGHT and CD154 (CD40 ligand) costimulate induction of dendritic cell maturation and elicit specific CTL activity.

CTLA-4 suppresses proximal TCR signaling in resting human CD4(+) T cells by inhibiting ZAP-70 Tyr(319) phosphorylation: a potential role for tyrosine phosphatases.

Cutting edge: selective impairment of CD8+ T cell function in mice lacking the TNF superfamily member LIGHT.

B and T lymphocyte attenuator exhibits structural and expression polymorphisms and is highly Induced in anergic CD4+ T cells.

P304

671-681

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scholarly article

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10.1038/NRI1917

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9

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6

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2006-09-01T00:00:00Z

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6855224

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16932752

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