Naturally occurring missense mutation in the polymerase gene terminating hepatitis B virus replication.
about
Hepatitis B: the virus and diseaseHepatitis B virus reverse transcriptase: diverse functions as classical and emerging targets for antiviral interventionMutation profiling of the hepatitis B virus strains circulating in North Indian populationMolecular characteristics of occult hepatitis B virus from blood donors in southeast China.Naturally occurring mutations define a novel function of the hepatitis B virus core promoter in core protein expression.The human immunodeficiency virus type 1 gag gene encodes an internal ribosome entry site.Targeted transfection and expression of hepatitis B viral DNA in human hepatoma cells.Detection of pre-C and core region mutants of hepatitis B virus in chronic hepatitis B virus carriers.p53-mediated cellular response to DNA damage in cells with replicative hepatitis B virusAcute hepatitis in rats expressing human hepatitis B virus transgenes.Hepatitis B virus x protein induces perinuclear mitochondrial clustering in microtubule- and Dynein-dependent manners.A novel method for efficient amplification of whole hepatitis B virus genomes permits rapid functional analysis and reveals deletion mutants in immunosuppressed patientsThe small envelope protein is required for secretion of a naturally occurring hepatitis B virus mutant with pre-S1 deleted.Occult hepatitis B virus infection in patients with chronic hepatitis C treated with antiviral therapy.Enhanced replication of a hepatitis B virus mutant associated with an epidemic of fulminant hepatitis.Hepatitis B virus X protein is not central to the viral life cycle in vitro.trans-Complementation of HBV rtM204I mutant replication by HBV wild-type polymerase.Two core promotor mutations identified in a hepatitis B virus strain associated with fulminant hepatitis result in enhanced viral replication.Mutations associated with occult hepatitis B virus infection result in decreased surface antigen expression in vitroA single amino acid in the reverse transcriptase domain of hepatitis B virus affects virus replication efficiencyIdentification of two separable modules in the duck hepatitis B virus core protein.Intracellular retention of surface protein by a hepatitis B virus mutant that releases virion particles.Selected mutations of the duck hepatitis B virus P gene RNase H domain affect both RNA packaging and priming of minus-strand DNA synthesis.Naturally occurring point mutation in the C terminus of the polymerase gene prevents duck hepatitis B virus RNA packaging.An RNAi-based high-throughput screening assay to identify small molecule inhibitors of hepatitis B virus replication.Mapping of Functional Subdomains in the Terminal Protein Domain of Hepatitis B Virus Polymerase.Hepadnaviral assembly is initiated by polymerase binding to the encapsidation signal in the viral RNA genomeOccult Hepatitis B Virus Infection and Associated Genotypes among HBsAg-negative Subjects in Burkina Faso.
P2860
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P2860
Naturally occurring missense mutation in the polymerase gene terminating hepatitis B virus replication.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Naturally occurring missense m ...... hepatitis B virus replication.
@ast
Naturally occurring missense m ...... hepatitis B virus replication.
@en
type
label
Naturally occurring missense m ...... hepatitis B virus replication.
@ast
Naturally occurring missense m ...... hepatitis B virus replication.
@en
prefLabel
Naturally occurring missense m ...... hepatitis B virus replication.
@ast
Naturally occurring missense m ...... hepatitis B virus replication.
@en
P2093
P2860
P1433
P1476
Naturally occurring missense m ...... hepatitis B virus replication.
@en
P2093
F von Weizsäcker
P2860
P304
P407
P577
1991-04-01T00:00:00Z