Hypothalamic malonyl-CoA as a mediator of feeding behavior. - Wikidata - wikimedia - TriplyDB

Hypothalamic malonyl-CoA as a mediator of feeding behavior.

Hypothalamic malonyl-CoA as a mediator of feeding behavior.

http://www.wikidata.org/entity/Q36689525

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Endocannabinoid activation at hepatic CB1 receptors stimulates fatty acid synthesis and contributes to diet-induced obesity Effect of food deprivation and hormones of glucose homeostasis on the acetyl CoA carboxylase activity in mouse brain: a potential role of acc in the regulation of energy balance.Hypothalamic AMPK as a Regulator of Energy Homeostasis Regulation of lipid stores and metabolism by lipophagy Hypothalamic malonyl-CoA and the control of food intake Regulation of peripheral metabolism by substrate partitioning in the brain.Brain Regulation of Energy Metabolism Olanzapine-induced hyperphagia and weight gain associate with orexigenic hypothalamic neuropeptide signaling without concomitant AMPK phosphorylation Malonyl-CoA decarboxylase is present in the cytosolic, mitochondrial and peroxisomal compartments of rat hepatocytes Characterization of the inactivation of rat fatty acid synthase by C75: inhibition of partial reactions and protection by substrates The Role of Circulating Amino Acids in the Hypothalamic Regulation of Liver Glucose Metabolism.The short-chain fatty acid acetate reduces appetite via a central homeostatic mechanism.Effects of chronic social defeat stress on peripheral leptin and its hypothalamic actions Overexpression of glucose-6-phosphate dehydrogenase is associated with lipid dysregulation and insulin resistance in obesity Effect of centrally administered C75, a fatty acid synthase inhibitor, on ghrelin secretion and its downstream effects The CPT1C 5'UTR contains a repressing upstream open reading frame that is regulated by cellular energy availability and AMPK A ketone ester diet increases brain malonyl-CoA and Uncoupling proteins 4 and 5 while decreasing food intake in the normal Wistar Rat.Inhibition of hypothalamic fatty acid synthase triggers rapid activation of fatty acid oxidation in skeletal muscle.Hypothalamic AMPK and fatty acid metabolism mediate thyroid regulation of energy balance.α-Mangostin induces apoptosis and suppresses differentiation of 3T3-L1 cells via inhibiting fatty acid synthase.Hypothalamic Angptl4/Fiaf is a novel regulator of food intake and body weight.AMP kinase and malonyl-CoA: targets for therapy of the metabolic syndrome.Selective reversible inhibition of liver carnitine palmitoyl-transferase 1 by teglicar reduces gluconeogenesis and improves glucose homeostasis.Berberine improves lipid dysregulation in obesity by controlling central and peripheral AMPK activity.Oleic acid and octanoic acid sensing capacity in rainbow trout Oncorhynchus mykiss is direct in hypothalamus and Brockmann bodies Leptin activates hypothalamic acetyl-CoA carboxylase to inhibit food intake.Estrogen regulates energy metabolic pathway and upstream adenosine 5'-monophosphate-activated protein kinase and phosphatase enzyme expression in dorsal vagal complex metabolosensory neurons during glucostasis and hypoglycemia.Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin-deficient rats.The brain-specific carnitine palmitoyltransferase-1c regulates energy homeostasis.Important roles of brain-specific carnitine palmitoyltransferase and ceramide metabolism in leptin hypothalamic control of feeding Pregnancy induces resistance to the anorectic effect of hypothalamic malonyl-CoA and the thermogenic effect of hypothalamic AMPK inhibition in female rats.Hypothalamic malonyl-CoA triggers mitochondrial biogenesis and oxidative gene expression in skeletal muscle: Role of PGC-1alpha.Long-term increased carnitine palmitoyltransferase 1A expression in ventromedial hypotalamus causes hyperphagia and alters the hypothalamic lipidomic profile A novel inhibitor of fatty acid synthase shows activity against HER2+ breast cancer xenografts and is active in anti-HER2 drug-resistant cell lines.Brain fatty acid synthase activates PPARalpha to maintain energy homeostasis.AMPK activation with glabridin ameliorates adiposity and lipid dysregulation in obesity.Hypothalamic sensing of fatty acids.Central regulation of energy balance: inputs, outputs and leptin resistance.Transcriptomes of parents identify parenting strategies and sexual conflict in a subsocial beetle.Treating the metabolic syndrome: acetyl-CoA carboxylase inhibition.

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P2860

Hypothalamic malonyl-CoA as a mediator of feeding behavior.

http://www.wikidata.org/entity/Q36689525

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2003 nî lūn-bûn

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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2003年论文

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2003年论文

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Hypothalamic malonyl-CoA as a mediator of feeding behavior.

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Hypothalamic malonyl-CoA as a mediator of feeding behavior.

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M Daniel Lane

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Seung Hun Cha

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Shigeru Chohnan

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Zhiyuan Hu

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P2860

Inhibition of hypothalamic carnitine palmitoyltransferase-1 decreases food intake and glucose production

Central nervous system control of food intake

The mitochondrial carnitine palmitoyltransferase system. From concept to molecular analysis

Effect of a fatty acid synthase inhibitor on food intake and expression of hypothalamic neuropeptides.

Differential effects of a centrally acting fatty acid synthase inhibitor in lean and obese mice.

Hypothalamic neuropeptide mechanisms for regulating energy balance: from rodent models to human obesity.

The hypothalamus and the control of energy homeostasis: different circuits, different purposes.

Effect of the anorectic fatty acid synthase inhibitor C75 on neuronal activity in the hypothalamus and brainstem.

Synthesis and antitumor activity of an inhibitor of fatty acid synthase.

Cerulenin mimics effects of leptin on metabolic rate, food intake, and body weight independent of the melanocortin system, but unlike leptin, cerulenin fails to block neuroendocrine effects of fasting.

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