Cardiomyocyte-restricted knockout of STAT3 results in higher sensitivity to inflammation, cardiac fibrosis, and heart failure with advanced age.
about
Altered expression and activation of signal transducers and activators of transcription (STATs) in hepatitis C virus infection: in vivo and in vitro studiesCytoprotection by the modulation of mitochondrial electron transport chain: the emerging role of mitochondrial STAT3Embryonic caffeine exposure acts via A1 adenosine receptors to alter adult cardiac function and DNA methylation in miceSTAT3 deletion sensitizes cells to oxidative stressEnhancing or eliminating signals for cell survival to treat disease.Gene expression and biological processes influenced by deletion of Stat3 in pulmonary type II epithelial cells.Granulocyte colony-stimulating factor protects cardiac mitochondria in the early phase of cardiac injury.Gene expression profiling of dilated cardiomyopathy in older male EP4 knockout mice.Regulation and function of signal transducer and activator of transcription 3.Overexpression of Nrdp1 in the heart exacerbates doxorubicin-induced cardiac dysfunction in miceRescue of cardiac leptin receptors in db/db mice prevents myocardial triglyceride accumulationP53 inhibition exacerbates late-stage anthracycline cardiotoxicity.Sca-1+ cardiac stem cells mediate acute cardioprotection via paracrine factor SDF-1 following myocardial ischemia/reperfusionDeciphering STAT3 signaling in the heart: plasticity and vascular inflammation.Activation of host tissue trophic factors through JAK-STAT3 signaling: a mechanism of mesenchymal stem cell-mediated cardiac repair.Blockade of Janus kinase-2 signaling ameliorates mouse liver damage due to ischemia and reperfusionMaternal obesity induces fibrosis in fetal myocardium of sheepSTAT subtype specificity and ischemic preconditioning in mice: is STAT-3 enough?Activation of the aryl hydrocarbon receptor by doxorubicin mediates cytoprotective effects in the heartAdult aortic valve interstitial cells have greater responses to toll-like receptor 4 stimulation.Network analysis of inflammatory genes and their transcriptional regulators in coronary artery disease.Mitochondrial-targeted Signal transducer and activator of transcription 3 (STAT3) protects against ischemia-induced changes in the electron transport chain and the generation of reactive oxygen speciesThe sexual dimorphism associated with pulmonary hypertension corresponds to a fibrotic phenotypeIL-11 Attenuates Liver Ischemia/Reperfusion Injury (IRI) through STAT3 Signaling Pathway in Mice.Mesenchymal stem cell therapy for doxorubicin cardiomyopathy: hopes and fearsFL3, a Synthetic Flavagline and Ligand of Prohibitins, Protects Cardiomyocytes via STAT3 from Doxorubicin ToxicityEpigenetic modifiers reduce inflammation and modulate macrophage phenotype during endotoxemia-induced acute lung injury.Nemo-Like Kinase (NLK) Is a Pathological Signaling Effector in the Mouse Heart.New frontiers in heart hypertrophy during pregnancy.Rapamycin protects against myocardial ischemia-reperfusion injury through JAK2-STAT3 signaling pathway.Critical Roles of STAT3 in β-Adrenergic Functions in the HeartHerbal Supplement Ameliorates Cardiac Hypertrophy in Rats with CCl(4)-Induced Liver CirrhosisCardiomyocyte-specific deletion of leptin receptors causes lethal heart failure in Cre-recombinase-mediated cardiotoxicity.Role of gp130-mediated signalling pathways in the heart and its impact on potential therapeutic aspectsMyocardial Hypertrophic Remodeling and Impaired Left Ventricular Function in Mice with a Cardiac-Specific Deletion of Janus Kinase 2.Leptin affects endocardial cushion formation by modulating EMT and migration via Akt signaling cascades.Ataxia telangiectasia mutated in cardiac fibroblasts regulates doxorubicin-induced cardiotoxicity.Crosstalk between signaling pathways of adrenoreceptors and signal transducers and activators of transcription 3 (STAT3) in heart.STAT3 activation in pressure-overloaded feline myocardium: role for integrins and the tyrosine kinase BMXα(1A)-adrenergic receptor differentially regulates STAT3 phosphorylation through PKCϵ and PKCδ in myocytes.
P2860
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P2860
Cardiomyocyte-restricted knockout of STAT3 results in higher sensitivity to inflammation, cardiac fibrosis, and heart failure with advanced age.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Cardiomyocyte-restricted knock ...... art failure with advanced age.
@ast
Cardiomyocyte-restricted knock ...... art failure with advanced age.
@en
type
label
Cardiomyocyte-restricted knock ...... art failure with advanced age.
@ast
Cardiomyocyte-restricted knock ...... art failure with advanced age.
@en
prefLabel
Cardiomyocyte-restricted knock ...... art failure with advanced age.
@ast
Cardiomyocyte-restricted knock ...... art failure with advanced age.
@en
P2093
P2860
P356
P1476
Cardiomyocyte-restricted knock ...... art failure with advanced age.
@en
P2093
April Kalinowski
Gui-Xuan Chai
Jörg J Jacoby
Kerry S Russell
Michael Schneider
Mu-Gen Liu
Samuel S-M Zhang
P2860
P304
12929-12934
P356
10.1073/PNAS.2134694100
P407
P577
2003-10-17T00:00:00Z