Derepression of pathological cardiac genes by members of the CaM kinase superfamily.
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Protein kinase C-related kinase targets nuclear localization signals in a subset of class IIa histone deacetylasesHistone deacetylase signaling in cardioprotectionEmerging roles for histone deacetylases in pulmonary hypertension and right ventricular remodeling (2013 Grover Conference series)Acetyl-lysine erasers and readers in the control of pulmonary hypertension and right ventricular hypertrophyAKAP-Lbc mobilizes a cardiac hypertrophy signaling pathwayPhospholipase Cε hydrolyzes perinuclear phosphatidylinositol 4-phosphate to regulate cardiac hypertrophyHistone deacetylase 5 acquires calcium/calmodulin-dependent kinase II responsiveness by oligomerization with histone deacetylase 4Potent and selective disruption of protein kinase D functionality by a benzoxoloazepinolone.CaM kinase II in colonic smooth muscle contributes to dysmotility in murine DSS-colitis.HDAC6 contributes to pathological responses of heart and skeletal muscle to chronic angiotensin-II signaling.Urotensin II-induced signaling involved in proliferation of vascular smooth muscle cellsProtein kinase D as a potential new target for cancer therapy.Novel function of cardiac protein kinase D1 as a dynamic regulator of Ca2+ sensitivity of contractionSmall heat shock protein 20 (Hsp20) facilitates nuclear import of protein kinase D 1 (PKD1) during cardiac hypertrophyHistone deacetylase 5 interacts with Krüppel-like factor 2 and inhibits its transcriptional activity in endothelium.Dysfunctional ryanodine receptor and cardiac hypertrophy: role of signaling moleculesRegulation of gastrointestinal motility by Ca2+/calmodulin-stimulated protein kinase II.Spatiotemporally distinct protein kinase D activation in adult cardiomyocytes in response to phenylephrine and endothelinSelective repression of MEF2 activity by PKA-dependent proteolysis of HDAC4.Promiscuous actions of small molecule inhibitors of the protein kinase D-class IIa HDAC axis in striated muscleProtein kinase D3 is a pivotal activator of pathological cardiac hypertrophy by selectively increasing the expression of hypertrophic transcription factors.Non-sirtuin histone deacetylases in the control of cardiac agingDeletion of CaMKK2 from the liver lowers blood glucose and improves whole-body glucose tolerance in the mouseCalcium/calmodulin-dependent protein kinase kinase 2: roles in signaling and pathophysiologyOxidant and redox signaling in vascular oxygen sensing: implications for systemic and pulmonary hypertensionUncovering G protein-coupled receptor kinase-5 as a histone deacetylase kinase in the nucleus of cardiomyocytes.Class IIa histone deacetylases: regulating the regulators.Is CaMKII a link between inflammation and hypertrophy in heart?The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload.A class of their own: exploring the nondeacetylase roles of class IIa HDACs in cardiovascular diseaseHistone Deacetylase Inhibitor Phenylbutyrate Exaggerates Heart Failure in Pressure Overloaded Mice independently of HDAC inhibition.Phosphodiesterase 5 inhibition blocks pressure overload-induced cardiac hypertrophy independent of the calcineurin pathway.Targeting histone deacetylases for heart failure.Class IIa HDACs: from important roles in differentiation to possible implications in tumourigenesis.Physiological functions of protein kinase D in vivo.Integrated mechanisms of CaMKII-dependent ventricular remodeling.Regulation of cardiac myocyte cell death and differentiation by myocardin.Aurora B-dependent regulation of class IIa histone deacetylases by mitotic nuclear localization signal phosphorylation.Inducible cardiomyocyte-specific deletion of CaM kinase II protects from pressure overload-induced heart failure.Ca2+-calmodulin-dependent protein kinase II represses cardiac transcription of the L-type calcium channel alpha(1C)-subunit gene (Cacna1c) by DREAM translocation.
P2860
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P2860
Derepression of pathological cardiac genes by members of the CaM kinase superfamily.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Derepression of pathological cardiac genes by members of the CaM kinase superfamily.
@ast
Derepression of pathological cardiac genes by members of the CaM kinase superfamily.
@en
type
label
Derepression of pathological cardiac genes by members of the CaM kinase superfamily.
@ast
Derepression of pathological cardiac genes by members of the CaM kinase superfamily.
@en
prefLabel
Derepression of pathological cardiac genes by members of the CaM kinase superfamily.
@ast
Derepression of pathological cardiac genes by members of the CaM kinase superfamily.
@en
P2860
P1476
Derepression of pathological cardiac genes by members of the CaM kinase superfamily.
@en
P2093
Timothy A McKinsey
P2860
P304
P356
10.1016/J.CARDIORES.2006.11.036
P577
2006-12-05T00:00:00Z