about
Identification and preliminary characterization of protein-cysteine farnesyltransferaseLoss of the amino-terminal helix-loop-helix domain of the vav proto-oncogene activates its transforming potentialKRAS mutant allele-specific imbalance in lung adenocarcinomaMolecular cloning of the mouse grb2 gene: differential interaction of the Grb2 adaptor protein with epidermal growth factor and nerve growth factor receptorsRestricted 12p amplification and RAS mutation in human germ cell tumors of the adult testis.Genetic analysis of the K-rev-1 transformation-suppressor gene.Glutamine deprivation induces abortive s-phase rescued by deoxyribonucleotides in k-ras transformed fibroblasts.Malignant transformation by ras and other oncogenes produces common alterations in inositol phospholipid signaling pathwaysGenetic analysis of the Kirsten-ras-revertant 1 gene: potentiation of its tumor suppressor activity by specific point mutations.Identification of a provirally activated c-Ha-ras oncogene in an avian nephroblastoma via a novel procedure: cDNA cloning of a chimaeric viral-host transcript.Ras in cancer and developmental diseasesIncreased expression of the putative growth factor receptor p185HER2 causes transformation and tumorigenesis of NIH 3T3 cells.Next generation sequencing analysis of platinum refractory advanced germ cell tumor sensitive to Sunitinib (Sutent®) a VEGFR2/PDGFRβ/c-kit/ FLT3/RET/CSF1R inhibitor in a phase II trialMechanisms of cell-cycle arrest in Spitz nevi with constitutive activation of the MAP-kinase pathwayOncogenic K-Ras decouples glucose and glutamine metabolism to support cancer cell growth.Mutations and copy number increase of HRAS in Spitz nevi with distinctive histopathological featuresTranscriptional activation of cKi-ras proto-oncogene resulting from retroviral promoter insertionProtein Kinase A Activation Promotes Cancer Cell Resistance to Glucose Starvation and AnoikisK-Ras Activation Induces Differential Sensitivity to Sulfur Amino Acid Limitation and Deprivation and to Oxidative and Anti-Oxidative Stress in Mouse FibroblastsDominant transformation by mutated human ras genes in vitro requires more than 100 times higher expression than is observed in cancersc-ras-Ha gene expression is regulated by insulin or insulinlike growth factor and by epidermal growth factor in murine fibroblasts.Expression of H-ras correlates with metastatic potential: evidence for direct regulation of the metastatic phenotype in 10T1/2 and NIH 3T3 cellsInterferon-induced revertants of ras-transformed cells: resistance to transformation by specific oncogenes and retransformation by 5-azacytidine.Structural and functional characterization of the promoter region of the mouse c-Ki-ras gene.Synergism of v-myc and v-Ha-ras in the in vitro neoplastic progression of murine lymphoid cellsActivation of oncogenicity of the c-rel proto-oncogene.Oncogene amplification during tumorigenesis of established rat fibroblasts reversibly transformed by activated human ras oncogenesDissection of the mouse N-ras gene upstream regulatory sequences and identification of the promoter and a negative regulatory element.Effects of mutant human Ki-ras(G12C) gene dosage on murine lung tumorigenesis and signaling to its downstream effectorsGlucose starvation induces cell death in K-ras-transformed cells by interfering with the hexosamine biosynthesis pathway and activating the unfolded protein response.Molecular analysis of ras oncogenes in CIN III and in stage I and II invasive squamous cell carcinoma of the uterine cervix.A novel method, digital genome scanning detects KRAS gene amplification in gastric cancers: involvement of overexpressed wild-type KRAS in downstream signaling and cancer cell growthIdentification of an activated c-Ki-ras oncogene in rat liver tumors induced by aflatoxin B1.Divergent in vitro/in vivo responses to drug treatments of highly aggressive NIH-Ras cancer cells: a PET imaging and metabolomics-mass-spectrometry study.An S1 nuclease-sensitive homopurine/homopyrimidine domain in the c-Ki-ras promoter interacts with a nuclear factor.Oncogenic proteins new targets for chemotherapeutic agents against cancer.An overview of tumor biology.HRAS, EGFR, MET, and RON Genes Are Recurrently Activated by Provirus Insertion in Liver Tumors Induced by the Retrovirus Myeloblastosis-Associated Virus 2.Targeting the KRAS variant for treatment of non-small cell lung cancer: potential therapeutic applications.H-ras distribution and signaling in plasma membrane microdomains are regulated by acylation and deacylation events.
P2860
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P2860
description
1985 nî lūn-bûn
@nan
1985年の論文
@ja
1985年論文
@yue
1985年論文
@zh-hant
1985年論文
@zh-hk
1985年論文
@zh-mo
1985年論文
@zh-tw
1985年论文
@wuu
1985年论文
@zh
1985年论文
@zh-cn
name
ras gene Amplification and malignant transformation.
@en
type
label
ras gene Amplification and malignant transformation.
@en
prefLabel
ras gene Amplification and malignant transformation.
@en
P2093
P2860
P356
P1476
ras gene Amplification and malignant transformation.
@en
P2093
P2860
P304
P356
10.1128/MCB.5.10.2836
P407
P577
1985-10-01T00:00:00Z