Type I interferon signaling exacerbates Chlamydia muridarum genital infection in a murine model.
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Immunomodulatory functions of type I interferonsDifferential effects of type I and II interferons on myeloid cells and resistance to intracellular bacterial infectionsInterferon-ε protects the female reproductive tract from viral and bacterial infectionInduction of interferon-stimulated genes by Chlamydia pneumoniae in fibroblasts is mediated by intracellular nucleotide-sensing receptors.Genetic variation in Chlamydia trachomatis and their hosts: impact on disease severity and tissue tropismGetting "Inside" Type I IFNs: Type I IFNs in Intracellular Bacterial InfectionsDown regulation of macrophage IFNGR1 exacerbates systemic L. monocytogenes infectionStimulator of IFN gene is critical for induction of IFN-beta during Chlamydia muridarum infectionType I IFN signaling constrains IL-17A/F secretion by gammadelta T cells during bacterial infections.MyD88 and STING signaling pathways are required for IRF3-mediated IFN-β induction in response to Brucella abortus infectionUnraveling the convoluted biological roles of type I interferons in infection and immunity: a way forward for therapeutics and vaccine design.Type I IFN receptor regulates neutrophil functions and innate immunity to Leishmania parasites.Type I interferons in bacterial infections: taming of myeloid cells and possible implications for autoimmunity.Antagonistic crosstalk between type I and II interferons and increased host susceptibility to bacterial infectionsThe DNA sensor, cyclic GMP-AMP synthase, is essential for induction of IFN-β during Chlamydia trachomatis infection.Type I interferons increase host susceptibility to Trypanosoma cruzi infection.Interferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infectionCD4 T cell depletion at the cervix during HIV infection is associated with accumulation of terminally differentiated T cells.Identifying a role for Toll-like receptor 3 in the innate immune response to Chlamydia muridarum infection in murine oviduct epithelial cells.Chlamydial Pre-Infection Protects from Subsequent Herpes Simplex Virus-2 Challenge in a Murine Vaginal Super-Infection Model.NOD1 in contrast to NOD2 functional polymorphism influence Chlamydia trachomatis infection and the risk of tubal factor infertility.Transcriptional Profiling in Experimental Visceral Leishmaniasis Reveals a Broad Splenic Inflammatory Environment that Conditions Macrophages toward a Disease-Promoting Phenotype.Role of STAT1 in Chlamydia-Induced Type-1 Interferon Production in Oviduct Epithelial Cells.Detection of a microbial metabolite by STING regulates inflammasome activation in response to Chlamydia trachomatis infection.Chlamydia pneumoniae impairs the innate immune response in infected epithelial cells by targeting TRAF3STING-dependent recognition of cyclic di-AMP mediates type I interferon responses during Chlamydia trachomatis infection.Strategies Used by Bacteria to Grow in MacrophagesT cell responses in the absence of IFN-gamma exacerbate uterine infection with Chlamydia trachomatis.Chlamydia muridarum-specific CD4 T-cell clones recognize infected reproductive tract epithelial cells in an interferon-dependent fashion.B cells are essential for moderating the inflammatory response and controlling bacterial multiplication in a mouse model of vaccination against Chlamydophila abortus infectionLeading a sheltered life: intracellular pathogens and maintenance of vacuolar compartments.MicroRNAs Modulate Pathogenesis Resulting from Chlamydial Infection in MiceA Protective Vaccine against Chlamydia Genital Infection Using Vault Nanoparticles without an Added Adjuvant.Induction of type I interferons by bacteria.Towards a Chlamydia trachomatis vaccine: how close are we?Sensing the enemy, containing the threat: cell-autonomous immunity to Chlamydia trachomatis.Immunopathogenesis of Chlamydial Infections.Induction and function of IFNβ during viral and bacterial infection.Type I interferons in regulation of mucosal immunity.Type I IFN exhaustion is a host defence protecting against secondary bacterial infections.
P2860
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P2860
Type I interferon signaling exacerbates Chlamydia muridarum genital infection in a murine model.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Type I interferon signaling ex ...... l infection in a murine model.
@en
type
label
Type I interferon signaling ex ...... l infection in a murine model.
@en
prefLabel
Type I interferon signaling ex ...... l infection in a murine model.
@en
P2093
P2860
P356
P1476
Type I interferon signaling ex ...... l infection in a murine model.
@en
P2093
Anna M Goodwin
Charles W Andrews
Daniel Prantner
James D Sikes
Shanmugam Nagarajan
Toni Darville
Uma M Nagarajan
P2860
P304
P356
10.1128/IAI.00629-08
P407
P577
2008-07-28T00:00:00Z