Metabolism and expression of RNA polymerase II transcripts in influenza virus-infected cells
about
Influenza A virus NS1 protein targets poly(A)-binding protein II of the cellular 3'-end processing machinery.Influenza B virus NS1 protein inhibits conjugation of the interferon (IFN)-induced ubiquitin-like ISG15 proteinTranslational regulation by the interferon-induced double-stranded-RNA-activated 68-kDa protein kinaseFunctional association between viral and cellular transcription during influenza virus infection.Gene expression signature-based screening identifies new broadly effective influenza a antiviralsInfluenza virus NS1 protein inhibits pre-mRNA splicing and blocks mRNA nucleocytoplasmic transportInefficient control of host gene expression by the 2009 pandemic H1N1 influenza A virus NS1 proteinRole of host immune response and viral load in the differential outcome of pandemic H1N1 (2009) influenza virus infection in Indian patientsRNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cellsGlobal impact of influenza virus on cellular pathways is mediated by both replication-dependent and -independent events.Protein synthesis shut-off induced by influenza virus infection is independent of PKR activityTranslational control of viral gene expression in eukaryotesEvasion of innate and adaptive immune responses by influenza A virus.Global mRNA degradation during lytic gammaherpesvirus infection contributes to establishment of viral latency.Cytopathogenesis and inhibition of host gene expression by RNA viruses.Human influenza viruses activate an interferon-independent transcription of cellular antiviral genes: outcome with influenza A virus is unique.Selective translation of eukaryotic mRNAs: functional molecular analysis of GRSF-1, a positive regulator of influenza virus protein synthesis.Individual expression of influenza virus PA protein induces degradation of coexpressed proteins.Influenza virus NS1 protein enhances the rate of translation initiation of viral mRNAs.H5N1 virus causes significant perturbations in host proteome very early in influenza virus-infected primary human monocyte-derived macrophages.Cyclic AMP regulates potassium channel expression in C6 glioma by destabilizing Kv1.1 mRNAThe influenza virus NS1 protein is a poly(A)-binding protein that inhibits nuclear export of mRNAs containing poly(A)Modification of eukaryotic initiation factor 4F during infection by influenza virus.TRIM22 inhibits influenza A virus infection by targeting the viral nucleoprotein for degradation.Effect of mutations and deletions in a bicistronic mRNA on the synthesis of influenza B virus NB and NA glycoproteins.Inhibition of influenza viral mRNA synthesis in cells expressing the interferon-induced Mx gene product.Effect of human alpha A interferon on influenza virus replication in MDBK cells.Efficient transcription, not translation, is dependent on adenovirus tripartite leader sequences at late times of infection.Cellular mRNA translation is blocked at both initiation and elongation after infection by influenza virus or adenovirusInfluenza virus regulates protein synthesis during infection by repressing autophosphorylation and activity of the cellular 68,000-Mr protein kinase.Influenza virus gene expression: control mechanisms at early and late times of infection and nuclear-cytoplasmic transport of virus-specific RNAsTranslational control by influenza virus: suppression of the kinase that phosphorylates the alpha subunit of initiation factor eIF-2 and selective translation of influenza viral mRNAsInhibition of p38 mitogen-activated protein kinase impairs influenza virus-induced primary and secondary host gene responses and protects mice from lethal H5N1 infection.Influenza virus mRNA translation revisited: is the eIF4E cap-binding factor required for viral mRNA translation?CHD6, a cellular repressor of influenza virus replication, is degraded in human alveolar epithelial cells and mice lungs during infection.Eukaryotic translation initiation factor 4GI is a cellular target for NS1 protein, a translational activator of influenza virus.Interferon induction and/or production and its suppression by influenza A viruses.Highly pathogenic avian influenza viruses inhibit effective immune responses of human blood-derived macrophages.Thogoto and Dhori virus replication is blocked by inhibitors of cellular polymerase II activity but does not cause shutoff of host cell protein synthesis.Comprehending a Killer: The Akt/mTOR Signaling Pathways Are Temporally High-Jacked by the Highly Pathogenic 1918 Influenza Virus.
P2860
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P2860
Metabolism and expression of RNA polymerase II transcripts in influenza virus-infected cells
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年論文
@yue
1984年論文
@zh-hant
1984年論文
@zh-hk
1984年論文
@zh-mo
1984年論文
@zh-tw
1984年论文
@wuu
1984年论文
@zh
1984年论文
@zh-cn
name
Metabolism and expression of R ...... influenza virus-infected cells
@en
type
label
Metabolism and expression of R ...... influenza virus-infected cells
@en
prefLabel
Metabolism and expression of R ...... influenza virus-infected cells
@en
P2860
P356
P1476
Metabolism and expression of R ...... influenza virus-infected cells
@en
P2093
P2860
P304
P356
10.1128/MCB.4.10.2198
P407
P577
1984-10-01T00:00:00Z