SH3GL2 is frequently deleted in non-small cell lung cancer and downregulates tumor growth by modulating EGFR signaling. - Wikidata - wikimedia - TriplyDB

SH3GL2 is frequently deleted in non-small cell lung cancer and downregulates tumor growth by modulating EGFR signaling.

SH3GL2 is frequently deleted in non-small cell lung cancer and downregulates tumor growth by modulating EGFR signaling.

http://www.wikidata.org/entity/Q36953349

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Loss of SH3GL2 promotes the migration and invasion behaviours of glioblastoma cells through activating the STAT3/MMP2 signalling.Context-dependent function of the deubiquitinating enzyme USP9X in pancreatic ductal adenocarcinoma Overexpression of EGFR in head and neck squamous cell carcinoma is associated with inactivation of SH3GL2 and CDC25A genes.MiR-330-mediated regulation of SH3GL2 expression enhances malignant behaviors of glioblastoma stem cells by activating ERK and PI3K/AKT signaling pathways Integrated Analysis of DNA Methylation and mRNA Expression Profiles Data to Identify Key Genes in Lung Adenocarcinoma.Loss of Sh3gl2/endophilin A1 is a common event in urothelial carcinoma that promotes malignant behavior.Uncovering Driver DNA Methylation Events in Nonsmoking Early Stage Lung Adenocarcinoma.Identification of genes and critical control proteins associated with inflammatory breast cancer using network controllability.Decreased expression of USP9X is associated with poor prognosis in Chinese pancreatic ductal adenocarcinoma patients.

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P2860

SH3GL2 is frequently deleted in non-small cell lung cancer and downregulates tumor growth by modulating EGFR signaling.

http://www.wikidata.org/entity/Q36953349

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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name

SH3GL2 is frequently deleted i ...... by modulating EGFR signaling.

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type

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SH3GL2 is frequently deleted i ...... by modulating EGFR signaling.

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SH3GL2 is frequently deleted i ...... by modulating EGFR signaling.

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Journal of Molecular Medicine

P1476

SH3GL2 is frequently deleted i ...... by modulating EGFR signaling.

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P2093

Adi F Gazdar

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Chunbo Shao

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Conover Talbot

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David Sidransky

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Jin Jen

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Jin Sung Jang

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Joanne E Yi

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Nitai D Mukhopadhyay

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Paul B Fisher

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Ping Yang

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P2860

CIN85 participates in Cbl-b-mediated down-regulation of receptor tyrosine kinases

High-resolution genomic profiles of human lung cancer

Characterizing the cancer genome in lung adenocarcinoma.

cbl-b inhibits epidermal growth factor receptor signaling

Cbl-CIN85-endophilin complex mediates ligand-induced downregulation of EGF receptors

Lung cancer in never smokers--a different disease

Ubiquitin ligase activity and tyrosine phosphorylation underlie suppression of growth factor signaling by c-Cbl/Sli-1

Recurrent genomic gains in preinvasive lesions as a biomarker of risk for lung cancer

Following mitochondrial footprints through a long mucosal path to lung cancer.

Potential predictive markers of chemotherapy resistance in stage III ovarian serous carcinomas.

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381-393

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10.1007/S00109-012-0955-3

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3

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91

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Mohammad Obaidul Hoque

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2012-09-12T00:00:00Z

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22968441

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3691869

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