APOBEC3G cytosine deamination hotspots are defined by both sequence context and single-stranded DNA secondary structure.
about
Multiple APOBEC3 restriction factors for HIV-1 and one Vif to rule them allMechanisms of viral mutationAPOBEC3G inhibits HIV-1 RNA elongation by inactivating the viral trans-activation response element.Association of a germline copy number polymorphism of APOBEC3A and APOBEC3B with burden of putative APOBEC-dependent mutations in breast cancerAPOBEC3A cytidine deaminase induces RNA editing in monocytes and macrophagesPotent Cell-Intrinsic Immune Responses in Dendritic Cells Facilitate HIV-1-Specific T Cell Immunity in HIV-1 Elite Controllers.HIV-1 and HIV-2 exhibit similar mutation frequencies and spectra in the absence of G-to-A hypermutation.Minimal Contribution of APOBEC3-Induced G-to-A Hypermutation to HIV-1 Recombination and Genetic Variation.The topography of mutational processes in breast cancer genomes.Structural basis for targeted DNA cytosine deamination and mutagenesis by APOBEC3A and APOBEC3B.Mechanisms underlying mutational signatures in human cancers.APOBECs and virus restriction.APOBEC Enzymes as Targets for Virus and Cancer Therapy.Biochemical Regulatory Features of Activation-Induced Cytidine Deaminase Remain Conserved from Lampreys to Humans.Viral subversion of APOBEC3s: Lessons for anti-tumor immunity and tumor immunotherapy.Replacement of feline foamy virus bet by feline immunodeficiency virus vif yields replicative virus with novel vaccine candidate potential.Asymmetric Modification of Hepatitis B Virus (HBV) Genomes by an Endogenous Cytidine Deaminase inside HBV Cores Informs a Model of Reverse Transcription.Substrate sequence selectivity of APOBEC3A implicates intra-DNA interactions.The Neuropeptides Vasoactive Intestinal Peptide and Pituitary Adenylate Cyclase-Activating Polypeptide Control HIV-1 Infection in Macrophages Through Activation of Protein Kinases A and C.Alternative DNA secondary structure formation affects RNA polymerase II promoter-proximal pausing in human.Modeling the Embrace of a Mutator: APOBEC Selection of Nucleic Acid LigandsStrategy of Human Cytomegalovirus To Escape Interferon Beta-Induced APOBEC3G Editing Activity
P2860
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P2860
APOBEC3G cytosine deamination hotspots are defined by both sequence context and single-stranded DNA secondary structure.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
APOBEC3G cytosine deamination ...... anded DNA secondary structure.
@en
type
label
APOBEC3G cytosine deamination ...... anded DNA secondary structure.
@en
prefLabel
APOBEC3G cytosine deamination ...... anded DNA secondary structure.
@en
P2093
P2860
P356
P1476
APOBEC3G cytosine deamination ...... anded DNA secondary structure.
@en
P2093
Colleen M Holtz
Holly A Sadler
Louis M Mansky
P2860
P304
P356
10.1093/NAR/GKT246
P407
P577
2013-04-24T00:00:00Z