Effects of Clostridium difficile toxins given intragastrically to animals.
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Clostridium difficile toxins: mechanism of action and role in disease.Toxins A and B from Clostridium difficile differ with respect to enzymatic potencies, cellular substrate specificities, and surface binding to cultured cellsThe low molecular mass GTP-binding protein Rho is affected by toxin A from Clostridium difficileClostridium difficile: its disease and toxinsToxin B is essential for virulence of Clostridium difficilePurification and characterization of toxin B from Clostridium difficileEffects of Clostridium difficile toxins A and B in rabbit small and large intestine in vivo and on cultured cells in vitro.Clostridium difficile Toxins A and B: Insights into Pathogenic Properties and Extraintestinal EffectsStructure-Function Analysis of Inositol Hexakisphosphate-induced Autoprocessing in Clostridium difficile Toxin AStructural Basis for Substrate Recognition in the Enzymatic Component of ADP-ribosyltransferase Toxin CDTa from Clostridium difficileStructural Determinants of Clostridium difficile Toxin A Glucosyltransferase ActivityAsymptomatic Clostridium difficile colonization: epidemiology and clinical implicationsThe binary toxin CDT enhances Clostridium difficile virulence by suppressing protective colonic eosinophilia.Glucosylation Drives the Innate Inflammatory Response to Clostridium difficile Toxin AHigh temporal resolution of glucosyltransferase dependent and independent effects of Clostridium difficile toxins across multiple cell types.In vivo physiological and transcriptional profiling reveals host responses to Clostridium difficile toxin A and toxin BImmune responses to Clostridium difficile infectionEffects of adenosine A₂A receptor activation and alanyl-glutamine in Clostridium difficile toxin-induced ileitis in rabbits and cecitis in mice.Enteric bacterial toxins: mechanisms of action and linkage to intestinal secretion.Clostridium difficile toxin A induces the release of neutrophil chemotactic factors from rat peritoneal macrophages: role of interleukin-1beta, tumor necrosis factor alpha, and leukotrienes.Identification of Clostridium difficile toxin B cardiotoxicity using a zebrafish embryo model of intoxication.Expression of recombinant Clostridium difficile toxin A and B in Bacillus megateriumPiglet models of acute or chronic Clostridium difficile illness.Toxin gene analysis of a variant strain of Clostridium difficile that causes human clinical disease.Purification and characterization of alpha-toxin produced by Clostridium novyi type AIdentification of toxin A-negative, toxin B-positive Clostridium difficile by PCR.Clostridium difficile toxin B differentially affects GPCR-stimulated Ca2+ responses in macrophages: independent roles for Rho and PLA2.Immunogenicity of a Salmonella typhimurium aroA aroD vaccine expressing a nontoxic domain of Clostridium difficile toxin A.Characterization of a toxin A-negative, toxin B-positive strain of Clostridium difficile responsible for a nosocomial outbreak of Clostridium difficile-associated diarrhea.Serum antitoxin antibodies mediate systemic and mucosal protection from Clostridium difficile disease in hamsters.Local and systemic neutralizing antibody responses induced by intranasal immunization with the nontoxic binding domain of toxin A from Clostridium difficile.The role of toxin A and toxin B in Clostridium difficile-associated disease: Past and present perspectives.Role of FliC and FliD flagellar proteins of Clostridium difficile in adherence and gut colonization.The role of the humoral immune response to Clostridium difficile toxins A and B in susceptibility to C. difficile infection: a case-control study.Clostridium difficile toxin A-induced microvascular dysfunction. Role of histamine.Clostridium difficile toxin B is more potent than toxin A in damaging human colonic epithelium in vitroEvolution of the caecal epithelial barrier during Clostridium difficile infection in the mouse.Using phenotype microarrays to determine culture conditions that induce or repress toxin production by Clostridium difficile and other microorganisms.The roles of host and pathogen factors and the innate immune response in the pathogenesis of Clostridium difficile infection.Immunization with Bacillus spores expressing toxin A peptide repeats protects against infection with Clostridium difficile strains producing toxins A and B
P2860
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P2860
Effects of Clostridium difficile toxins given intragastrically to animals.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on February 1985
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Effects of Clostridium difficile toxins given intragastrically to animals.
@en
Effects of Clostridium difficile toxins given intragastrically to animals.
@nl
type
label
Effects of Clostridium difficile toxins given intragastrically to animals.
@en
Effects of Clostridium difficile toxins given intragastrically to animals.
@nl
prefLabel
Effects of Clostridium difficile toxins given intragastrically to animals.
@en
Effects of Clostridium difficile toxins given intragastrically to animals.
@nl
P2093
P2860
P1476
Effects of Clostridium difficile toxins given intragastrically to animals.
@en
P2093
D K MacDonald
D M Lyerly
T D Wilkins
P2860
P304
P407
P577
1985-02-01T00:00:00Z