Cross talk of signals between EGFR and IL-6R through JAK2/STAT3 mediate epithelial-mesenchymal transition in ovarian carcinomas.
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BLT2 is a pro-tumorigenic mediator during cancer progression and a therapeutic target for anti-cancer drug developmentSTAT3: A Novel Molecular Mediator of Resistance to ChemoradiotherapyMyeloid-derived suppressor cells in the tumor microenvironment: expect the unexpectedEpidermal growth factor protects squamous cell carcinoma against cisplatin-induced cytotoxicity through increased interleukin-1β expressionThe Mu opioid receptor promotes opioid and growth factor-induced proliferation, migration and Epithelial Mesenchymal Transition (EMT) in human lung cancerInduction of epithelial-mesenchymal transition (EMT) in breast cancer cells is calcium signal dependentVoting-based cancer module identification by combining topological and data-driven propertiesTemporal-logic analysis of microglial phenotypic conversion with exposure to amyloid-β.Adipose Stromal Cells from Visceral and Subcutaneous Fat Facilitate Migration of Ovarian Cancer Cells via IL-6/JAK2/STAT3 PathwaySTAT3 and epithelial-mesenchymal transitions in carcinomasInhibition of the JAK2/STAT3 pathway in ovarian cancer results in the loss of cancer stem cell-like characteristics and a reduced tumor burdenStat3-positive tumor cells contribute to vessels neoformation in primary central nervous system lymphomaEMT and inflammation: inseparable actors of cancer progression.Epithelial-to-Mesenchymal Transition in the Female Reproductive Tract: From Normal Functioning to Disease Pathology.IL-6 stabilizes Twist and enhances tumor cell motility in head and neck cancer cells through activation of casein kinase 2.Activated networking of platelet activating factor receptor and FAK/STAT1 induces malignant potential in BRCA1-mutant at-risk ovarian epithelium.Neuronal transcription factor Brn-3a(l) is over expressed in high-grade ovarian carcinomas and tumor cells from ascites of patients with advanced-stage ovarian cancerJAK2/STAT3 targeted therapy suppresses tumor invasion via disruption of the EGFRvIII/JAK2/STAT3 axis and associated focal adhesion in EGFRvIII-expressing glioblastoma.Copy number variation analysis of matched ovarian primary tumors and peritoneal metastasisDiindolylmethane suppresses ovarian cancer growth and potentiates the effect of cisplatin in tumor mouse model by targeting signal transducer and activator of transcription 3 (STAT3).EGF-induced EMT and invasiveness in serous borderline ovarian tumor cells: a possible step in the transition to low-grade serous carcinoma cells?Efficient inhibition of intraperitoneal ovarian cancer growth in nude mice by liposomal delivery of short hairpin RNA against STAT3.Function and regulation of MTA1 and MTA3 in malignancies of the female reproductive system.Lymphocyte deficiency limits Epstein-Barr virus latent membrane protein 1 induced chronic inflammation and carcinogenic pathology in vivoCrosstalk of Oncogenic Signaling Pathways during Epithelial-Mesenchymal Transition.Immune biomarkers of anti-EGFR monoclonal antibody therapyJanus kinase 2 polymorphisms are associated with risk in patients with gastric cancer in a Chinese population.Activation of JAK2/STAT3 signaling by osteopontin promotes tumor growth in human breast cancer cells.IL-6 promotes prostate tumorigenesis and progression through autocrine cross-activation of IGF-IR.Interleukin 6 receptor is an independent prognostic factor and a potential therapeutic target of ovarian cancer.Ovarian epithelial-stromal interactions: role of interleukins 1 and 6.Metformin inhibits the IL-6-induced epithelial-mesenchymal transition and lung adenocarcinoma growth and metastasis.Cell death induced by the Jak2 inhibitor, G6, correlates with cleavage of vimentin filaments.Induction of murine macrophage M2 polarization by cigarette smoke extract via the JAK2/STAT3 pathway.Targeted Blockade of JAK/STAT3 Signaling Inhibits Ovarian Carcinoma Growth.Sorafenib inhibits growth and metastasis of hepatocellular carcinoma by blocking STAT3MyD88-Dependent Signaling Decreases the Antitumor Efficacy of Epidermal Growth Factor Receptor Inhibition in Head and Neck Cancer Cells.STAT1-mediated down-regulation of Bcl-2 expression is involved in IFN-γ/TNF-α-induced apoptosis in NIT-1 cells.Screening for characteristic microRNAs between pre-invasive and invasive stages of cervical cancer.Roles of STAT3 and ZEB1 proteins in E-cadherin down-regulation and human colorectal cancer epithelial-mesenchymal transition
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Cross talk of signals between EGFR and IL-6R through JAK2/STAT3 mediate epithelial-mesenchymal transition in ovarian carcinomas.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 16 December 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Cross talk of signals between ...... nsition in ovarian carcinomas.
@en
Cross talk of signals between ...... nsition in ovarian carcinomas.
@nl
type
label
Cross talk of signals between ...... nsition in ovarian carcinomas.
@en
Cross talk of signals between ...... nsition in ovarian carcinomas.
@nl
prefLabel
Cross talk of signals between ...... nsition in ovarian carcinomas.
@en
Cross talk of signals between ...... nsition in ovarian carcinomas.
@nl
P2093
P2860
P50
P356
P1476
Cross talk of signals between ...... ansition in ovarian carcinomas
@en
P2093
P2860
P2888
P304
P356
10.1038/SJ.BJC.6604794
P407
P577
2008-12-16T00:00:00Z