Advances on the understanding of the origins of synaptic pathology in AD. - Wikidata - wikimedia - TriplyDB

Advances on the understanding of the origins of synaptic pathology in AD.

Advances on the understanding of the origins of synaptic pathology in AD.

http://www.wikidata.org/entity/Q37108156

about

A new target for amyloid beta toxicity validated by standard and high-throughput electrophysiology Deleterious effects of amyloid beta oligomers acting as an extracellular scaffold for mGluR5 The Aβ oligomer hypothesis for synapse failure and memory loss in Alzheimer's disease.Role of the Transforming-Growth-Factor-β1 Gene in Late-Onset Alzheimer's Disease: Implications for the Treatment Insulin receptor dysfunction impairs cellular clearance of neurotoxic oligomeric a{beta}Aβ oligomer-induced synapse degeneration in Alzheimer's disease.Role of amyloid β protein receptors in mediating synaptic plasticity.Synaptophysin depletion and intraneuronal Aβ in organotypic hippocampal slice cultures from huAPP transgenic mice.Endoplasmic reticulum stress occurs downstream of GluN2B subunit of N-methyl-d-aspartate receptor in mature hippocampal cultures treated with amyloid-β oligomers.

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Advances on the understanding of the origins of synaptic pathology in AD.

http://www.wikidata.org/entity/Q37108156

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on December 2007

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Advances on the understanding of the origins of synaptic pathology in AD.

@en

Advances on the understanding of the origins of synaptic pathology in AD.

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type

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Advances on the understanding of the origins of synaptic pathology in AD.

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Advances on the understanding of the origins of synaptic pathology in AD.

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prefLabel

Advances on the understanding of the origins of synaptic pathology in AD.

@en

Advances on the understanding of the origins of synaptic pathology in AD.

@nl

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Current Genomics

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Advances on the understanding of the origins of synaptic pathology in AD.

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Pascale Nathalie Lacor

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A Oligomer-Induced Aberrations in Synapse Composition, Shape, and Density Provide a Molecular Basis for Loss of Connectivity in Alzheimer's Disease

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Alzheimer Disease in the US Population

beta-Amyloid(1-42) binds to alpha7 nicotinic acetylcholine receptor with high affinity. Implications for Alzheimer's disease pathology

Drebrin, a dendritic spine protein, is manifold decreased in brains of patients with Alzheimer's disease and Down syndrome

Caspase cleavage of tau: linking amyloid and neurofibrillary tangles in Alzheimer's disease

Dendritic spines elongate after stimulation of group 1 metabotropic glutamate receptors in cultured hippocampal neurons.

beta-Amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease

Amyloid plaque core protein in Alzheimer disease and Down syndrome

Pathways towards and away from Alzheimer's disease

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scholarly article

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