HIV populations are large and accumulate high genetic diversity in a nonlinear fashion
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Large Variations in HIV-1 Viral Load Explained by Shifting-Mosaic Metapopulation DynamicsLoss and recovery of genetic diversity in adapting populations of HIVCoordinated Evolution of Influenza A Surface ProteinsClinical Determinants of HIV-1B Between-Host Evolution and their Association with Drug Resistance in Pediatric PatientsAnalysis of HIV Diversity in HIV-Infected Black Men Who Have Sex with Men (HPTN 061)Review: Influence of ART on HIV geneticsIdentifying signatures of selection in genetic time seriesA spatio-temporal assessment of simian/human immunodeficiency virus (SHIV) evolution reveals a highly dynamic process within the host.Antibody 10-1074 suppresses viremia in HIV-1-infected individualsClear: Composition of Likelihoods for Evolve and Resequence Experiments.Association between latent proviral characteristics and immune activation in antiretrovirus-treated human immunodeficiency virus type 1-infected adultsHIV-1 DNA decay dynamics in blood during more than a decade of suppressive antiretroviral therapyFifteen years later: hard and soft selection sweeps confirm a large population number for HIV in vivo.Lack of detectable HIV-1 molecular evolution during suppressive antiretroviral therapyAssessing Biases in the Evaluation of Classification Assays for HIV Infection Recency.Effect of the Latent Reservoir on the Evolution of HIV at the Within- and Between-Host Levels.Ex vivo activation of CD4+ T-cells from donors on suppressive ART can lead to sustained production of infectious HIV-1 from a subset of infected cellsProviruses with identical sequences comprise a large fraction of the replication-competent HIV reservoirPopulation genomics of intrapatient HIV-1 evolutionOrigin of Rebound Plasma HIV Includes Cells with Identical Proviruses That Are Transcriptionally Active before Stopping of Antiretroviral Therapy.The role of HIV integration in viral persistence: no more whistling past the proviral graveyard.What Integration Sites Tell Us about HIV PersistenceHIV-infected cells are frequently clonally expanded after prolonged antiretroviral therapy: implications for HIV persistence.Proliferation of latently infected CD4+ T cells carrying replication-competent HIV-1: Potential role in latent reservoir dynamics.Fifteen to twenty percent of HIV substitution mutations are associated with recombinationSingle-cell analysis of HIV-1 transcriptional activity reveals expression of proviruses in expanded clones during ART.High-Sequence Diversity and Rapid Virus Turnover Contribute to Higher Rates of Coreceptor Switching in Treatment-Experienced Subjects with HIV-1 Viremia.Allele Age Under Non-Classical Assumptions is Clarified by an Exact Computational Markov Chain Approach.Impact of Clinical Parameters in the Intrahost Evolution of HIV-1 Subtype B in Pediatric Patients: A Machine Learning ApproachPatterns and rates of viral evolution in HIV-1 subtype B infected females and males.Molecular evolution of HIV-1 integrase during the 20 years prior to the first approval of integrase inhibitors.On the effective depth of viral sequence data.HIV evolution and diversity in ART-treated patients.Effects of multiple sources of genetic drift on pathogen variation within hosts.Genetic divergence of HIV-1 B subtype in Italy over the years 2003-2016 and impact on CTL escape prevalenceDevelopment of sensitive ddPCR assays to reliably quantify the proviral DNA reservoir in all common circulating HIV subtypes and recombinant formsA majority of HIV persistence during antiretroviral therapy is due to infected cell proliferation
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HIV populations are large and accumulate high genetic diversity in a nonlinear fashion
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 15 May 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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HIV populations are large and accumulate high genetic diversity in a nonlinear fashion
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HIV populations are large and accumulate high genetic diversity in a nonlinear fashion.
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HIV populations are large and accumulate high genetic diversity in a nonlinear fashion
@en
HIV populations are large and accumulate high genetic diversity in a nonlinear fashion.
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HIV populations are large and accumulate high genetic diversity in a nonlinear fashion
@en
HIV populations are large and accumulate high genetic diversity in a nonlinear fashion.
@nl
P2093
P2860
P356
P1433
P1476
HIV populations are large and accumulate high genetic diversity in a nonlinear fashion
@en
P2093
Catherine Rehm
Daniel L Lucey
Diane Rock-Kress
Frank Maldarelli
Harvey Alter
JoAnn Mican
John M Coffin
John W Mellors
Joseph Kovacs
Julia A Metcalf
P2860
P304
10313-10323
P356
10.1128/JVI.01225-12
P407
P577
2013-05-15T00:00:00Z