Comparative analysis of lesion development and intraspinal inflammation in four strains of mice following spinal contusion injury.
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Characterization of early and terminal complement proteins associated with polymorphonuclear leukocytes in vitro and in vivo after spinal cord injuryQuantitative analysis of cellular inflammation after traumatic spinal cord injury: evidence for a multiphasic inflammatory response in the acute to chronic environmentDeficiency in complement C1q improves histological and functional locomotor outcome after spinal cord injuryTranslational spinal cord injury research: preclinical guidelines and challengesHematogenous macrophage depletion reduces the fibrotic scar and increases axonal growth after spinal cord injuryAnalysis of host-mediated repair mechanisms after human CNS-stem cell transplantation for spinal cord injury: correlation of engraftment with recoveryADAM8 is selectively up-regulated in endothelial cells and is associated with angiogenesis after spinal cord injury in adult miceMinimum information about a spinal cord injury experiment: a proposed reporting standard for spinal cord injury experimentsSyndromics: a bioinformatics approach for neurotrauma researchIdentification of two distinct macrophage subsets with divergent effects causing either neurotoxicity or regeneration in the injured mouse spinal cordIdentifying the Long-Term Role of Inducible Nitric Oxide Synthase after Contusive Spinal Cord Injury Using a Transgenic Mouse Model.B cells produce pathogenic antibodies and impair recovery after spinal cord injury in mice.Age decreases macrophage IL-10 expression: Implications for functional recovery and tissue repair in spinal cord injuryDominant inheritance of retinal ganglion cell resistance to optic nerve crush in mice.Evaluating regional blood spinal cord barrier dysfunction following spinal cord injury using longitudinal dynamic contrast-enhanced MRI.Differential response of C57BL/6J mouse and DBA/2J mouse to optic nerve crushNeutralizing endogenous VEGF following traumatic spinal cord injury modulates microvascular plasticity but not tissue sparing or functional recovery.The complement cascade: Yin-Yang in neuroinflammation--neuro-protection and -degeneration.Age is a determinant of leukocyte infiltration and loss of cortical volume after traumatic brain injury.Comparison of immunopathology and locomotor recovery in C57BL/6, BUB/BnJ, and NOD-SCID mice after contusion spinal cord injuryRescuing vasculature with intravenous angiopoietin-1 and alpha v beta 3 integrin peptide is protective after spinal cord injury.IL-4 signaling drives a unique arginase+/IL-1β+ microglia phenotype and recruits macrophages to the inflammatory CNS: consequences of age-related deficits in IL-4Rα after traumatic spinal cord injury.Regional heterogeneity in astrocyte responses following contusive spinal cord injury in miceExtracellular matrix regulation of inflammation in the healthy and injured spinal cordThe paradox of chronic neuroinflammation, systemic immune suppression, autoimmunity after traumatic chronic spinal cord injuryOptic nerve crush induces spatial and temporal gene expression patterns in retina and optic nerve of BALB/cJ mice.Self-assembling peptide amphiphile promotes plasticity of serotonergic fibers following spinal cord injury.Central but not systemic administration of XPro1595 is therapeutic following moderate spinal cord injury in miceDifferential gene expression in the EphA4 knockout spinal cord and analysis of the inflammatory response following spinal cord injuryProgranulin expression is upregulated after spinal contusion in mice.BMPR1a and BMPR1b signaling exert opposing effects on gliosis after spinal cord injuryQuantitative evaluation of 3D mouse behaviors and motor function in the open-field after spinal cord injury using markerless motion tracking.Cytosolic phospholipase A2 protein as a novel therapeutic target for spinal cord injuryMacrophages in spinal cord injury: phenotypic and functional change from exposure to myelin debris.Deficient CX3CR1 signaling promotes recovery after mouse spinal cord injury by limiting the recruitment and activation of Ly6Clo/iNOS+ macrophages.Anti-inflammatory treatments during the chronic phase of spinal cord injury improve locomotor function in adult mice.Matrix metalloproteinase-9 and stromal cell-derived factor-1 act synergistically to support migration of blood-borne monocytes into the injured spinal cordFibronectin Matrix Assembly after Spinal Cord InjuryTraumatic spinal cord injury in mice with human immune systemsCNS injury, glial scars, and inflammation: Inhibitory extracellular matrices and regeneration failure
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Comparative analysis of lesion development and intraspinal inflammation in four strains of mice following spinal contusion injury.
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on February 2006
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Comparative analysis of lesion ...... owing spinal contusion injury.
@en
Comparative analysis of lesion ...... owing spinal contusion injury.
@nl
type
label
Comparative analysis of lesion ...... owing spinal contusion injury.
@en
Comparative analysis of lesion ...... owing spinal contusion injury.
@nl
prefLabel
Comparative analysis of lesion ...... owing spinal contusion injury.
@en
Comparative analysis of lesion ...... owing spinal contusion injury.
@nl
P2093
P2860
P356
P1476
Comparative analysis of lesion ...... owing spinal contusion injury.
@en
P2093
Kristina A Kigerl
Phillip G Popovich
Violeta M McGaughy
P2860
P304
P356
10.1002/CNE.20827
P407
P577
2006-02-01T00:00:00Z