Lipopolysaccharide-induced interleukin-6 production is controlled by glycogen synthase kinase-3 and STAT3 in the brain.
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Early activation of STAT3 regulates reactive astrogliosis induced by diverse forms of neurotoxicityGlycogen synthase kinase 3: a point of convergence for the host inflammatory responseNeuronal seipin knockout facilitates Aβ-induced neuroinflammation and neurotoxicity via reduction of PPARγ in hippocampus of mouseB cells responses and cytokine production are regulated by their immune microenvironmentEffects of lithium on lipopolysaccharide-induced inflammation in rat primary glia cellsGenetic and pharmacological evidence for schizophrenia-related Disc1 interaction with GSK-3.Glycogen synthase kinase-3 in the etiology and treatment of mood disorders.Is glycogen synthase kinase-3 a central modulator in mood regulation?Selective deletion of forebrain glycogen synthase kinase 3β reveals a central role in serotonin-sensitive anxiety and social behaviourInhibition of GSK3β-mediated BACE1 expression reduces Alzheimer-associated phenotypes.Beneficial effects of mood stabilizers lithium, valproate and lamotrigine in experimental stroke models.Innate and adaptive immune responses regulated by glycogen synthase kinase-3 (GSK3).A new avenue for lithium: intervention in traumatic brain injury.Heteroarylketones inhibit astroglial interleukin-6 expression via a STAT3/NF-κB signaling pathwayLithium suppresses astrogliogenesis by neural stem and progenitor cells by inhibiting STAT3 pathway independently of glycogen synthase kinase 3 beta.Glycogen synthase kinase-3 regulates inflammatory tolerance in astrocytes.Evidence of reactive astrocytes but not peripheral immune system activation in a mouse model of Fragile X syndromeFever and the thermal regulation of immunity: the immune system feels the heat.Hypoxia antagonizes glucose deprivation on interleukin 6 expression in an Akt dependent, but HIF-1/2α independent manner.Inflammation and lithium: clues to mechanisms contributing to suicide-linked traits.Signaling pathways underlying the rapid antidepressant actions of ketamineSignaling pathways underlying the pathophysiology and treatment of depression: novel mechanisms for rapid-acting agents.Lithium Decreases Glial Fibrillary Acidic Protein in a Mouse Model of Alexander Disease.GSK-3β-induced Tau pathology drives hippocampal neuronal cell death in Huntington's disease: involvement of astrocyte-neuron interactionsLow molecular weight heparin prevents lipopolysaccharide induced-hippocampus-dependent cognitive impairments in mice.The role of glycogen synthase kinase-3 signaling in neurodevelopment and fragile X syndromeChronic lithium feeding reduces upregulated brain arachidonic acid metabolism in HIV-1 transgenic rat.GSK3β inhibition protects the immature brain from hypoxic-ischaemic insult via reduced STAT3 signalling.Retina-on-a-chip: a microfluidic platform for point access signaling studiesVagal nerve stimulation blocks interleukin 6-dependent synaptic hyperexcitability induced by lipopolysaccharide-induced acute stress in the rodent prefrontal cortex.Stress-induced neuroinflammation is mediated by GSK3-dependent TLR4 signaling that promotes susceptibility to depression-like behavior.Regulatory role of GSK-3 β on NF- κ B, nitric oxide, and TNF- α in group A streptococcal infection.STAT3-mediated coincidence detection regulates noncanonical immediate early gene induction.Angiotensin II causes imbalance between pro- and anti-inflammatory cytokines by modulating GSK-3β in neuronal culture.Lung inflammation induces IL-1β expression in hypoglossal neurons in rat brainstem.Lithium chloride promotes host resistance against Pseudomonas aeruginosa keratitis.Activation of Autophagy Pathway Suppresses the Expression of iNOS, IL6 and Cell Death of LPS-Stimulated Microglia Cells.Inhibition of glycogen synthase kinase 3β activity with lithium prevents and attenuates paclitaxel-induced neuropathic pain.Glycogen synthase kinase-3β: a mediator of inflammation in Alzheimer's disease?Role of glycogen synthase kinase-3 beta in the inflammatory response caused by bacterial pathogens
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Lipopolysaccharide-induced interleukin-6 production is controlled by glycogen synthase kinase-3 and STAT3 in the brain.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 11 March 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Lipopolysaccharide-induced int ...... nase-3 and STAT3 in the brain.
@en
Lipopolysaccharide-induced int ...... nase-3 and STAT3 in the brain.
@nl
type
label
Lipopolysaccharide-induced int ...... nase-3 and STAT3 in the brain.
@en
Lipopolysaccharide-induced int ...... nase-3 and STAT3 in the brain.
@nl
prefLabel
Lipopolysaccharide-induced int ...... nase-3 and STAT3 in the brain.
@en
Lipopolysaccharide-induced int ...... nase-3 and STAT3 in the brain.
@nl
P2860
P356
P1476
Lipopolysaccharide-induced int ...... nase-3 and STAT3 in the brain.
@en
P2093
Eléonore Beurel
Richard S Jope
P2860
P2888
P356
10.1186/1742-2094-6-9
P577
2009-03-11T00:00:00Z
P5875
P6179
1032573469