Modulation of transcription parameters in glucocorticoid receptor-mediated repression. - Wikidata - wikimedia - TriplyDB

Modulation of transcription parameters in glucocorticoid receptor-mediated repression.

Modulation of transcription parameters in glucocorticoid receptor-mediated repression.

http://www.wikidata.org/entity/Q37143831

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Org 214007-0: a novel non-steroidal selective glucocorticoid receptor modulator with full anti-inflammatory properties and improved therapeutic index Disruption of Ttll5/stamp gene (tubulin tyrosine ligase-like protein 5/SRC-1 and TIF2-associated modulatory protein gene) in male mice causes sperm malformation and infertility STAMP alters the growth of transformed and ovarian cancer cells.Crosstalk in inflammation: the interplay of glucocorticoid receptor-based mechanisms and kinases and phosphatases Research resource: modulators of glucocorticoid receptor activity identified by a new high-throughput screening assay.A theoretical framework for gene induction and experimental comparisons.Impact of glucocorticoid receptor density on ligand-independent dimerization, cooperative ligand-binding and basal priming of transactivation: a cell culture model.What goes on behind closed doors: physiological versus pharmacological steroid hormone actions.Kinetically-defined component actions in gene repression Separate regions of glucocorticoid receptor, coactivator TIF2, and comodulator STAMP modify different parameters of glucocorticoid-mediated gene induction.The hypersensitive glucocorticoid response specifically regulates period 1 and expression of circadian genes SRC-2-mediated coactivation of anti-tumorigenic target genes suppresses MYC-induced liver cancer.Interindividual Variability in Stress Susceptibility: A Role for Epigenetic Mechanisms in PTSD.A conserved protein motif is required for full modulatory activity of negative elongation factor subunits NELF-A and NELF-B in modifying glucocorticoid receptor-regulated gene induction properties.The use of mode of action information in risk assessment: quantitative key events/dose-response framework for modeling the dose-response for key events.N-acetylcysteine protects pancreatic islet against glucocorticoid toxicity.An Approach to Greater Specificity for Glucocorticoids.

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Modulation of transcription parameters in glucocorticoid receptor-mediated repression.

http://www.wikidata.org/entity/Q37143831

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 21 May 2008

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Modulation of transcription parameters in glucocorticoid receptor-mediated repression.

@en

Modulation of transcription parameters in glucocorticoid receptor-mediated repression.

@nl

type

label

Modulation of transcription parameters in glucocorticoid receptor-mediated repression.

@en

Modulation of transcription parameters in glucocorticoid receptor-mediated repression.

@nl

prefLabel

Modulation of transcription parameters in glucocorticoid receptor-mediated repression.

@en

Modulation of transcription parameters in glucocorticoid receptor-mediated repression.

@nl

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J.MCE.2008.05.008

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Molecular and Cellular Endocrinology

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Modulation of transcription parameters in glucocorticoid receptor-mediated repression

@en

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Benjamin L Kagan

http://www.w3.org/2001/XMLSchema#string

S Stoney Simons

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Yong-Guang Tao

http://www.w3.org/2001/XMLSchema#string

Yuangzheng He

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59-69

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scholarly article

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10.1016/J.MCE.2008.05.008

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1-2

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295

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2662735

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