Organ-specific attenuation of murine hepatitis virus strain A59 by replacement of catalytic residues in the putative viral cyclic phosphodiesterase ns2.
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Coronavirus reverse genetic systems: infectious clones and repliconsStructure of the C-terminal domain of nsp4 from feline coronavirusViral phosphodiesterases that antagonize double-stranded RNA signaling to RNase L by degrading 2-5AAccessory protein 5a is a major antagonist of the antiviral action of interferon against murine coronavirus.Genetic determinants of mouse hepatitis virus strain 1 pneumovirulence.Murine AKAP7 has a 2',5'-phosphodiesterase domain that can complement an inactive murine coronavirus ns2 gene.Cell-type-specific type I interferon antagonism influences organ tropism of murine coronavirusThe nsp1, nsp13, and M proteins contribute to the hepatotropism of murine coronavirus JHM.WU.Activation of RNase L by Murine Coronavirus in Myeloid Cells Is Dependent on Basal Oas Gene Expression and Independent of Virus-Induced InterferonA review of genetic methods and models for analysis of coronavirus-induced severe pneumonitis.Middle East Respiratory Syndrome Coronavirus NS4b Protein Inhibits Host RNase L Activation.Cell-type-specific activation of the oligoadenylate synthetase-RNase L pathway by a murine coronavirus.Homologous 2',5'-phosphodiesterases from disparate RNA viruses antagonize antiviral innate immunity.Biochemical characterization of arterivirus nonstructural protein 11 reveals the nidovirus-wide conservation of a replicative endoribonuclease.Antagonism of RNase L Is Required for Murine Coronavirus Replication in Kupffer Cells and Liver Sinusoidal Endothelial Cells but Not in Hepatocytes.Murine coronavirus neuropathogenesis: determinants of virulence.Lineage A Betacoronavirus NS2 Proteins and the Homologous Torovirus Berne pp1a Carboxy-Terminal Domain Are Phosphodiesterases That Antagonize Activation of RNase L.Murine Coronavirus Cell Type Dependent Interaction with the Type I Interferon ResponseConstruction and genetic analysis of murine hepatitis virus strain A59 Nsp16 temperature sensitive mutant and the revertant virus.Coronavirus nonstructural protein 15 mediates evasion of dsRNA sensors and limits apoptosis in macrophages.Murine coronavirus delays expression of a subset of interferon-stimulated genesAntagonism of the interferon-induced OAS-RNase L pathway by murine coronavirus ns2 protein is required for virus replication and liver pathology.An "Old" protein with a new story: Coronavirus endoribonuclease is important for evading host antiviral defenses.
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Organ-specific attenuation of murine hepatitis virus strain A59 by replacement of catalytic residues in the putative viral cyclic phosphodiesterase ns2.
description
article científic
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article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 28 January 2009
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Organ-specific attenuation of ...... cyclic phosphodiesterase ns2.
@en
Organ-specific attenuation of ...... cyclic phosphodiesterase ns2.
@nl
type
label
Organ-specific attenuation of ...... cyclic phosphodiesterase ns2.
@en
Organ-specific attenuation of ...... cyclic phosphodiesterase ns2.
@nl
prefLabel
Organ-specific attenuation of ...... cyclic phosphodiesterase ns2.
@en
Organ-specific attenuation of ...... cyclic phosphodiesterase ns2.
@nl
P2093
P2860
P50
P356
P1433
P1476
Organ-specific attenuation of ...... cyclic phosphodiesterase ns2.
@en
P2093
Guohui Chang
Helen Stokes
Jessica K Roth-Cross
Ming Ming Chua
P2860
P304
P356
10.1128/JVI.02203-08
P407
P577
2009-01-28T00:00:00Z